Hypertrophic cardiomyopathy in

Age-related HCM MYBPC3 alternative splicing chaperone-mediated autophagy nonsense-mediated decay ubiquitin-proteosome system

Journal

The journal of cardiovascular aging
ISSN: 2768-5993
Titre abrégé: J Cardiovasc Aging
Pays: United States
ID NLM: 101778645

Informations de publication

Date de publication:
Jan 2024
Historique:
pmc-release: 01 01 2025
medline: 26 2 2024
pubmed: 26 2 2024
entrez: 26 2 2024
Statut: ppublish

Résumé

Hypertrophic cardiomyopathy (HCM) is characterized by abnormal thickening of the myocardium, leading to arrhythmias, heart failure, and elevated risk of sudden cardiac death, particularly among the young. This inherited disease is predominantly caused by mutations in sarcomeric genes, among which those in the cardiac myosin binding protein-C3 (

Identifiants

pubmed: 38406555
doi: 10.20517/jca.2023.29
pmc: PMC10883298
pii:
doi:

Types de publication

Journal Article

Langues

eng

Déclaration de conflit d'intérêts

Conflict of interest Sadayappan S provides consulting and collaborative research studies to the Leducq Foundation (CUREPLAN), Red Saree Inc., Greater Cincinnati Tamil Sangam, Affinia Therapeutics Inc., and Cosmogene Skincare Private Limited, but such work is unrelated to the content of this article. Dr. Ananthamohan K and Dr. Stelzer JE have no conflicts of interest to disclose.

Auteurs

Kalyani Ananthamohan (K)

Department of Internal Medicine, Division of Cardiovascular Health and Disease, University of Cincinnati, Cincinnati, OH 45267, USA.

Julian E Stelzer (JE)

Department of Physiology and Biophysics, School of Medicine, Case Western Reserve University, Cleveland, OH 45267, USA.

Sakthivel Sadayappan (S)

Department of Internal Medicine, Division of Cardiovascular Health and Disease, University of Cincinnati, Cincinnati, OH 45267, USA.

Classifications MeSH