Apolipoprotein A-IV concentrations and cancer in a large cohort of chronic kidney disease patients: results from the GCKD study.


Journal

BMC cancer
ISSN: 1471-2407
Titre abrégé: BMC Cancer
Pays: England
ID NLM: 100967800

Informations de publication

Date de publication:
07 Mar 2024
Historique:
received: 24 03 2023
accepted: 26 02 2024
medline: 11 3 2024
pubmed: 8 3 2024
entrez: 8 3 2024
Statut: epublish

Résumé

Chronic kidney disease (CKD) is highly connected to inflammation and oxidative stress. Both favour the development of cancer in CKD patients. Serum apolipoprotein A-IV (apoA-IV) concentrations are influenced by kidney function and are an early marker of kidney impairment. Besides others, it has antioxidant and anti-inflammatory properties. Proteomic studies and small case-control studies identified low apoA-IV as a biomarker for various forms of cancer; however, prospective studies are lacking. We therefore investigated whether serum apoA-IV is associated with cancer in the German Chronic Kidney Disease (GCKD) study. These analyses include 5039 Caucasian patients from the prospective GCKD cohort study followed for 6.5 years. Main inclusion criteria were an eGFR of 30-60 mL/min/1.73m Mean apoA-IV concentrations of the entire cohort were 28.9 ± 9.8 mg/dL (median 27.6 mg/dL). 615 patients had a history of cancer before the enrolment into the study. ApoA-IV concentrations above the median were associated with a lower odds for a history of cancer (OR = 0.79, p = 0.02 when adjusted age, sex, smoking, diabetes, BMI, albuminuria, statin intake, and eGFR Our data indicate an association of high apoA-IV concentrations with reduced frequencies of a history of cancer as well as incident fatal and non-fatal cancer events in a large cohort of patients with CKD.

Sections du résumé

BACKGROUND BACKGROUND
Chronic kidney disease (CKD) is highly connected to inflammation and oxidative stress. Both favour the development of cancer in CKD patients. Serum apolipoprotein A-IV (apoA-IV) concentrations are influenced by kidney function and are an early marker of kidney impairment. Besides others, it has antioxidant and anti-inflammatory properties. Proteomic studies and small case-control studies identified low apoA-IV as a biomarker for various forms of cancer; however, prospective studies are lacking. We therefore investigated whether serum apoA-IV is associated with cancer in the German Chronic Kidney Disease (GCKD) study.
METHODS METHODS
These analyses include 5039 Caucasian patients from the prospective GCKD cohort study followed for 6.5 years. Main inclusion criteria were an eGFR of 30-60 mL/min/1.73m
RESULTS RESULTS
Mean apoA-IV concentrations of the entire cohort were 28.9 ± 9.8 mg/dL (median 27.6 mg/dL). 615 patients had a history of cancer before the enrolment into the study. ApoA-IV concentrations above the median were associated with a lower odds for a history of cancer (OR = 0.79, p = 0.02 when adjusted age, sex, smoking, diabetes, BMI, albuminuria, statin intake, and eGFR
CONCLUSIONS CONCLUSIONS
Our data indicate an association of high apoA-IV concentrations with reduced frequencies of a history of cancer as well as incident fatal and non-fatal cancer events in a large cohort of patients with CKD.

Identifiants

pubmed: 38454416
doi: 10.1186/s12885-024-12053-8
pii: 10.1186/s12885-024-12053-8
pmc: PMC10921727
doi:

Substances chimiques

apolipoprotein A-IV 0
Apolipoproteins A 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

320

Subventions

Organisme : German Ministry of Education and Research
ID : FKZ 01ER 0804, 01ER 0818, 01ER 0819, 01ER 0820, and 01ER 0821
Organisme : Austrian Science Fund
ID : W-1253 DK HOROS

Investigateurs

Markus P Schneider (MP)
Mario Schiffer (M)
Hans-Ulrich Prokosch (HU)
Barbara Bärthlein (B)
Andreas Beck (A)
André Reis (A)
Arif B Ekici (AB)
Susanne Becker (S)
Ulrike Alberth-Schmidt (U)
Anke Weigel (A)
Sabine Marschall (S)
Eugenia Schefler (E)
Gerd Walz (G)
Anna Köttgen (A)
Ulla T Schultheiß (UT)
Simone Meder (S)
Erna Mitsch (E)
Ursula Reinhard (U)
Jürgen Floege (J)
Turgay Saritas (T)
Alice Gross (A)
Elke Schaeffner (E)
Seema Baid-Agrawal (S)
Kerstin Theisen (K)
Hermann Haller (H)
Martin Zeier (M)
Claudia Sommerer (C)
Mehtap Aykac (M)
Gunter Wolf (G)
Martin Busch (M)
Andy Steiner (A)
Thomas Sitter (T)
Vera Krane (V)
Antje Börner-Klein (A)
Britta Bauer (B)
Peter Oefner (P)
Wolfram Gronwald (W)
Matthias Schmid (M)
Jennifer Nadal (J)

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© 2024. The Author(s).

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Auteurs

Barbara Kollerits (B)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Simon Gruber (S)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Inga Steinbrenner (I)

Institute of Genetic Epidemiology, Faculty of Medicine and Medical Center - University of Freiburg, Freiburg, Germany.

Johannes P Schwaiger (JP)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Hansi Weissensteiner (H)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Sebastian Schönherr (S)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Lukas Forer (L)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria.

Fruzsina Kotsis (F)

Institute of Genetic Epidemiology, Faculty of Medicine and Medical Center - University of Freiburg, Freiburg, Germany.
Department of Medicine IV - Nephrology and Primary Care, Faculty of Medicine and Medical Center - University of Freiburg, Freiburg, Germany.

Ulla T Schultheiss (UT)

Institute of Genetic Epidemiology, Faculty of Medicine and Medical Center - University of Freiburg, Freiburg, Germany.
Department of Medicine IV - Nephrology and Primary Care, Faculty of Medicine and Medical Center - University of Freiburg, Freiburg, Germany.

Heike Meiselbach (H)

Department of Nephrology and Hypertension, University Hospital Erlangen, Friedrich-Alexander-Universität Erlangen-Nürnberg, Erlangen, Germany.
German Chronic Kidney Disease Study, Erlangen, Germany.

Christoph Wanner (C)

Division of Nephrology, Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Kai-Uwe Eckardt (KU)

Department of Nephrology and Hypertension, University Hospital Erlangen, Friedrich-Alexander-Universität Erlangen-Nürnberg, Erlangen, Germany.
German Chronic Kidney Disease Study, Erlangen, Germany.
Department of Nephrology and Medical Intensive Care, Charité - Universitätsmedizin Berlin, Berlin, Germany.

Florian Kronenberg (F)

Institute of Genetic Epidemiology, Medical University of Innsbruck, Schöpfstraße 41, Innsbruck, 6020, Austria. Florian.Kronenberg@i-med.ac.at.

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