Adalimumab as a cause of kidney injury in patients with Crohn's disease.

Adalimumab Crohn’s disease Drug reaction Kidney injury

Journal

Pediatric nephrology (Berlin, Germany)
ISSN: 1432-198X
Titre abrégé: Pediatr Nephrol
Pays: Germany
ID NLM: 8708728

Informations de publication

Date de publication:
08 Mar 2024
Historique:
received: 01 01 2024
accepted: 27 02 2024
revised: 26 02 2024
medline: 8 3 2024
pubmed: 8 3 2024
entrez: 8 3 2024
Statut: aheadofprint

Résumé

Adalimumab (ADM) is a recombinant human monoclonal antibody (anti-TNFα) used to treat inflammatory bowel diseases. It can cause kidney injury (KI). We describe two pediatric patients with Crohn's disease (CD) in whom ADM therapy was associated with kidney injury (KI). The drug was discontinued in both cases. For the first patient, changes were irreversible despite intensive glucocorticosteroid (GCS) therapy. For the second patient, discontinuation of ADM led to an improvement in kidney function. Due to the risk of KI in patients undergoing ADM therapy, careful assessment of kidney function and early specialist referral are required. Timely withdrawal of ADM can significantly reduce kidney damage, but in some cases, the kidney damage can be irreversible.

Identifiants

pubmed: 38456916
doi: 10.1007/s00467-024-06338-0
pii: 10.1007/s00467-024-06338-0
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024. The Author(s), under exclusive licence to International Pediatric Nephrology Association.

Références

Sandys V, Moloney B, Lane L, Qazi J, Doyle B, Barry M, Leavey S, Conlon P (2018) Granulomatous interstitial nephritis secondary to adalimumab therapy. Clin Kidney J 11:219–221. https://doi.org/10.1093/ckj/sfx104
doi: 10.1093/ckj/sfx104 pubmed: 29644062
Graziano F, Busè M, Cassata N, Lentini VL, Citrano M (2022) IgA nephropathy in a child: Crohn’s disease-associated or adalimumab induced? Curr Med Res Opin 38:139–143. https://doi.org/10.1080/03007995.2021.2015155
doi: 10.1080/03007995.2021.2015155 pubmed: 34866503
Chen TJ, Yang YF, Huang PH, Lin HH, Huang CC (2010) Permanent renal loss following tumor necrosis factor α antagonists for arthritis. Rheumatol Int 30:1077–1079. https://doi.org/10.1007/s00296-009-1016-2
doi: 10.1007/s00296-009-1016-2 pubmed: 19536543
Usui J, Salvatore SP, Yamagata K, Seshan SV (2023) Clinicopathologic spectrum of renal lesions following anti-TNF-α inhibitor therapy: a single center experience. Kidney360 4:363–373. https://doi.org/10.34067/KID.0000000000000063
doi: 10.34067/KID.0000000000000063
Tamura H (2023) IgA nephropathy associated with Crohn’s disease. World J Methodol 13:67–78. https://doi.org/10.5662/wjm.v13.i3.67
doi: 10.5662/wjm.v13.i3.67 pubmed: 37456980 pmcid: 10348078

Auteurs

Krzysztof Skoczyński (K)

Department of Nephrology, Kidney Transplantation and Hypertension, The Children`s Memorial Health Institute, Warsaw, Poland.
Faculty of Medicine, Collegium Medicum, Cardinal Stefan Wyszyński University, Warsaw, Poland.

Jan Koziej (J)

Department of Nephrology, Kidney Transplantation and Hypertension, The Children`s Memorial Health Institute, Warsaw, Poland.
Faculty of Medicine, Collegium Medicum, Cardinal Stefan Wyszyński University, Warsaw, Poland.

Sylwia Szymańska (S)

Department of Pathology, The Children`s Memorial Health Institute, Warsaw, Poland.

Łukasz Obrycki (Ł)

Department of Nephrology, Kidney Transplantation and Hypertension, The Children`s Memorial Health Institute, Warsaw, Poland.

Ryszard Grenda (R)

Department of Nephrology, Kidney Transplantation and Hypertension, The Children`s Memorial Health Institute, Warsaw, Poland.

Mieczysław Litwin (M)

Department of Nephrology, Kidney Transplantation and Hypertension, The Children`s Memorial Health Institute, Warsaw, Poland. m.litwin@ipczd.pl.
Chair of Pediatric Nephrology, The Center of Continuing Medical Education, Warsaw, Poland. m.litwin@ipczd.pl.

Classifications MeSH