Magnetic Resonance-Guided Focused Ultrasound for Treatment of Essential Tremor: Ventral Intermediate Nucleus Ablation Alone or Additional Posterior Subthalamic Area Lesioning?

essential tremor magnetic resonance-guided focused ultrasound posterior subthalamic area ventral intermediate nucleus

Journal

Movement disorders clinical practice
ISSN: 2330-1619
Titre abrégé: Mov Disord Clin Pract
Pays: United States
ID NLM: 101630279

Informations de publication

Date de publication:
12 Mar 2024
Historique:
revised: 18 12 2023
received: 31 07 2023
accepted: 24 01 2024
medline: 12 3 2024
pubmed: 12 3 2024
entrez: 12 3 2024
Statut: aheadofprint

Résumé

Magnetic resonance-guided focused ultrasound (MRgFUS) for treatment of essential tremor (ET) traditionally targets the ventral intermediate (Vim) nucleus. Recent strategies include a secondary lesion to the posterior subthalamic area (PSA). The aim was to compare lesion characteristics, tremor improvement, and adverse events (AE) between patients in whom satisfactory tremor suppression was achieved with lesioning of the Vim alone and patients who required additional lesioning of the PSA. Retrospective analysis of data collected from ET patients treated with MRgFUS at St Vincent's Hospital Sydney was performed. Clinical Rating Scale for Tremor (CRST), hand tremor score (HTS), and Quality of Life in Essential Tremor Questionnaire (QUEST) were collected pre- and posttreatment in addition to the prevalence of AEs. The lesion coordinates and overlap with the dentatorubrothalamic tract (DRTT) were evaluated using magnetic resonance imaging. Twenty-one patients were treated in Vim only, and 14 were treated with dual Vim-PSA lesions. Clinical data were available for 29 of the 35 patients (19 single target and 10 dual target). At follow-up (mean: 18.80 months) HTS, CRST, and QUEST in single-target patients improved by 57.97% (P < 0.001), 36.71% (P < 0.001), and 58.26% (P < 0.001), whereas dual-target patients improved by 68.34% (P < 0.001), 35.37% (P < 0.003), and 46.97% (P < 0.005), respectively. The Vim lesion of dual-target patients was further anterior relative to the posterior commissure (PC) (7.84 mm), compared with single-target patients (6.92 mm), with less DRTT involvement (14.85% vs. 23.21%). Dual-target patients exhibited a greater proportion of patients with acute motor AEs (100% vs. 58%); however, motor AE prevalence was similar in both groups at long-term follow-up (33% vs. 38%). Posterior placement of lesions targeting the Vim may confer greater tremor suppression. The addition of a PSA lesion, in patients with inadequate tremor control despite Vim lesioning, had a trend toward better long-term tremor suppression; however, this approach was associated with greater prevalence of gait disturbance in the short term.

Sections du résumé

BACKGROUND BACKGROUND
Magnetic resonance-guided focused ultrasound (MRgFUS) for treatment of essential tremor (ET) traditionally targets the ventral intermediate (Vim) nucleus. Recent strategies include a secondary lesion to the posterior subthalamic area (PSA).
OBJECTIVE OBJECTIVE
The aim was to compare lesion characteristics, tremor improvement, and adverse events (AE) between patients in whom satisfactory tremor suppression was achieved with lesioning of the Vim alone and patients who required additional lesioning of the PSA.
METHODS METHODS
Retrospective analysis of data collected from ET patients treated with MRgFUS at St Vincent's Hospital Sydney was performed. Clinical Rating Scale for Tremor (CRST), hand tremor score (HTS), and Quality of Life in Essential Tremor Questionnaire (QUEST) were collected pre- and posttreatment in addition to the prevalence of AEs. The lesion coordinates and overlap with the dentatorubrothalamic tract (DRTT) were evaluated using magnetic resonance imaging.
RESULTS RESULTS
Twenty-one patients were treated in Vim only, and 14 were treated with dual Vim-PSA lesions. Clinical data were available for 29 of the 35 patients (19 single target and 10 dual target). At follow-up (mean: 18.80 months) HTS, CRST, and QUEST in single-target patients improved by 57.97% (P < 0.001), 36.71% (P < 0.001), and 58.26% (P < 0.001), whereas dual-target patients improved by 68.34% (P < 0.001), 35.37% (P < 0.003), and 46.97% (P < 0.005), respectively. The Vim lesion of dual-target patients was further anterior relative to the posterior commissure (PC) (7.84 mm), compared with single-target patients (6.92 mm), with less DRTT involvement (14.85% vs. 23.21%). Dual-target patients exhibited a greater proportion of patients with acute motor AEs (100% vs. 58%); however, motor AE prevalence was similar in both groups at long-term follow-up (33% vs. 38%).
CONCLUSION CONCLUSIONS
Posterior placement of lesions targeting the Vim may confer greater tremor suppression. The addition of a PSA lesion, in patients with inadequate tremor control despite Vim lesioning, had a trend toward better long-term tremor suppression; however, this approach was associated with greater prevalence of gait disturbance in the short term.

Identifiants

pubmed: 38469997
doi: 10.1002/mdc3.14005
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : The Brain and Mind Centre Partnership Grant
Organisme : The GE Healthcare Medical Physics PhD Scholarship

Informations de copyright

© 2024 The Authors. Movement Disorders Clinical Practice published by Wiley Periodicals LLC on behalf of International Parkinson and Movement Disorder Society.

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Auteurs

Kain Kyle (K)

Faculty of Medicine and Health Translational Research Collective, The University of Sydney, Camperdown, New South Wales, Australia.
Brain and Mind Centre, The University of Sydney, Sydney, New South Wales, Australia.
Sydney Neuroimaging Analysis Centre, Camperdown, New South Wales, Australia.

James Peters (J)

Department of Neurology, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.
School of Medicine, University of New South Wales, Sydney, New South Wales, Australia.

Benjamin Jonker (B)

Department of Neurosurgery, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.
Royal Prince Alfred Institute of Academic Surgery, University of Sydney, Camperdown, New South Wales, Australia.

Yael Barnett (Y)

Department of Neurology, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.
Department of Medical Imaging, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.

Joel Maamary (J)

Department of Neurology, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.
School of Medicine, University of New South Wales, Sydney, New South Wales, Australia.

Michael Barnett (M)

Faculty of Medicine and Health Translational Research Collective, The University of Sydney, Camperdown, New South Wales, Australia.
Brain and Mind Centre, The University of Sydney, Sydney, New South Wales, Australia.
Sydney Neuroimaging Analysis Centre, Camperdown, New South Wales, Australia.
Department of Neurology, Royal Prince Alfred Hospital, Camperdown, New South Wales, Australia.

Jerome Maller (J)

GE Healthcare, Melbourne, Victoria, Australia.

Chenyu Wang (C)

Faculty of Medicine and Health Translational Research Collective, The University of Sydney, Camperdown, New South Wales, Australia.
Brain and Mind Centre, The University of Sydney, Sydney, New South Wales, Australia.
Sydney Neuroimaging Analysis Centre, Camperdown, New South Wales, Australia.

Stephen Tisch (S)

Department of Neurology, St Vincent's Hospital, Darlinghurst, New South Wales, Australia.
School of Medicine, University of New South Wales, Sydney, New South Wales, Australia.

Classifications MeSH