Association between polymorphisms on chromosome 17q12-q21 and rhinovirus-induced interferon responses.

17q12-q21 Bayesian methods asthma birth cohorts childhood innate immune response interferons rhinovirus

Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
15 Mar 2024
Historique:
received: 09 08 2023
revised: 23 02 2024
accepted: 01 03 2024
medline: 18 3 2024
pubmed: 18 3 2024
entrez: 17 3 2024
Statut: aheadofprint

Résumé

Single nucleotide polymorphisms (SNPs) in genes on chromosome 17q12-q21 are associated with childhood-onset asthma and rhinovirus-induced wheezing. There is little mechanistic data linking chromosome 17q12-q21 to wheezing illness. To investigate whether 17q12-q21 risk alleles may be associated with impaired interferon (IFN) responses to rhinovirus. In a population-based birth cohort of European ancestry, we stimulated peripheral blood mononuclear cells (PBMCs) with rhinovirus-A1 (RV-A1) and rhinovirus-A16 (RV-A16) and measured IFN and IFN-induced chemokine CXCL10/IP10 responses in supernatants. We investigated associations between virus-induced cytokines and six SNPs in 17q12-q21. Bayesian profile regression was applied to identify clusters of individuals with different immune response profiles and genetic variants. Five SNPs (in high linkage disequilibrium, r Polymorphisms on chromosome 17q12-q21 are associated with rhinovirus-induced IFN-β, suggesting a novel mechanism (impaired IFN-β induction) linking 17q12-q21 risk alleles with asthma/wheeze.

Sections du résumé

BACKGROUND BACKGROUND
Single nucleotide polymorphisms (SNPs) in genes on chromosome 17q12-q21 are associated with childhood-onset asthma and rhinovirus-induced wheezing. There is little mechanistic data linking chromosome 17q12-q21 to wheezing illness.
OBJECTIVE OBJECTIVE
To investigate whether 17q12-q21 risk alleles may be associated with impaired interferon (IFN) responses to rhinovirus.
METHODS METHODS
In a population-based birth cohort of European ancestry, we stimulated peripheral blood mononuclear cells (PBMCs) with rhinovirus-A1 (RV-A1) and rhinovirus-A16 (RV-A16) and measured IFN and IFN-induced chemokine CXCL10/IP10 responses in supernatants. We investigated associations between virus-induced cytokines and six SNPs in 17q12-q21. Bayesian profile regression was applied to identify clusters of individuals with different immune response profiles and genetic variants.
RESULTS RESULTS
Five SNPs (in high linkage disequilibrium, r
CONCLUSIONS CONCLUSIONS
Polymorphisms on chromosome 17q12-q21 are associated with rhinovirus-induced IFN-β, suggesting a novel mechanism (impaired IFN-β induction) linking 17q12-q21 risk alleles with asthma/wheeze.

Identifiants

pubmed: 38494094
pii: S0091-6749(24)00269-0
doi: 10.1016/j.jaci.2024.03.005
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024. Published by Elsevier Inc.

Auteurs

Eteri Regis (E)

National Heart and Lung Institute, Imperial College London, UK.

Sara Fontanella (S)

National Heart and Lung Institute, Imperial College London, UK.

John A Curtin (JA)

Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, Manchester Academic Health Sciences Centre, University of Manchester and University Hospital of South Manchester NHS Foundation Trust, Manchester, UK.

Angela Pinot de Moira (A)

National Heart and Lung Institute, Imperial College London, UK.

Michael R Edwards (MR)

National Heart and Lung Institute, Imperial College London, UK.

Clare S Murray (CS)

Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, Manchester Academic Health Sciences Centre, University of Manchester and University Hospital of South Manchester NHS Foundation Trust, Manchester, UK.

Angela Simpson (A)

Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, Manchester Academic Health Sciences Centre, University of Manchester and University Hospital of South Manchester NHS Foundation Trust, Manchester, UK.

Sebastian L Johnston (SL)

National Heart and Lung Institute, Imperial College London, UK.

Adnan Custovic (A)

National Heart and Lung Institute, Imperial College London, UK. Electronic address: a.custovic@imperial.ac.uk.

Classifications MeSH