Dietary linoleic acid supplementation protects against obesity-induced microglial reactivity in mice.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
19 Mar 2024
Historique:
received: 15 01 2024
accepted: 13 03 2024
medline: 20 3 2024
pubmed: 20 3 2024
entrez: 20 3 2024
Statut: epublish

Résumé

We investigated whether linoleic acid (LA) supplementation could modulate emotional behavior and microglia-related neuroinflammation. For that, male mice of C57BL/6J genetic background fed either a high-fat diet (HFD) or a standard diet (STD) for 12 weeks, were treated with a vehicle or LA solution for 5 weeks before being evaluated for emotional behavior using a battery of behavioral tests. The animals were subsequently sacrificed and their brains collected and processed for immunofluorescence staining, targeting microglia-specific calcium-binding proteins (IBA-1). Neuroinflammation severity was assessed in multiple hypothalamic, cortical and subcortical brain regions. We show an anxio-depressive-like effect of sustained HFD feeding that was neither alleviated nor worsened with LA supplementation. However, increased IBA-1 expression and microgliosis in the HFD group were largely attenuated by LA supplementation. These observations demonstrate that the anti-neuroinflammatory properties of LA are not restricted to hypothalamic areas but are also evident at the cortical and subcortical levels. This study discloses that neuroinflammation plays a role in the genesis of neuropsychiatric disorders in the context of obesity, and that LA supplementation is a useful dietary strategy to alleviate the impact of obesity-related neuroinflammation.

Identifiants

pubmed: 38503857
doi: 10.1038/s41598-024-56959-6
pii: 10.1038/s41598-024-56959-6
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6644

Informations de copyright

© 2024. The Author(s).

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Auteurs

Lucas Jantzen (L)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France.

Stéphanie Dumontoy (S)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France.

Bahrie Ramadan (B)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France.

Christophe Houdayer (C)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France.

Emmanuel Haffen (E)

Université de Franche-Comté, UMR INSERM 1322 LINC, service de psychiatrie de l'adulte, CIC-1431 INSERM, CHU de Besançon, 25030, Besançon, France.

Aziz Hichami (A)

Physiologie de la Nutrition & Toxicologie (NUTox), UMR UB/Institut Agro/INSERM U1231, Lipides, Nutrition & Cancer, LABEX-LipStick, Université de Bourgogne, Dijon, France.

Naim Akhtar Khan (NA)

Physiologie de la Nutrition & Toxicologie (NUTox), UMR UB/Institut Agro/INSERM U1231, Lipides, Nutrition & Cancer, LABEX-LipStick, Université de Bourgogne, Dijon, France.

Vincent Van Waes (V)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France.

Lidia Cabeza (L)

Université de Franche-Comté, UMR INSERM 1322 LINC, 19, Rue Ambroise Paré, 25000, Besançon Cedex, France. lidia.cabeza_alvarez@univ-fcomte.fr.

Classifications MeSH