Prmt6 represses the pro-adipogenic Ppar-gamma-C/ebp-alpha transcription factor loop.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
20 Mar 2024
Historique:
received: 13 12 2023
accepted: 17 03 2024
medline: 21 3 2024
pubmed: 21 3 2024
entrez: 21 3 2024
Statut: epublish

Résumé

The feed-forward loop between the transcription factors Ppar-gamma and C/ebp-alpha is critical for lineage commitment during adipocytic differentiation. Ppar-gamma interacts with epigenetic cofactors to activate C/ebp-alpha and the downstream adipocytic gene expression program. Therefore, knowledge of the epigenetic cofactors associated with Ppar-gamma, is central to understanding adipocyte differentiation in normal differentiation and disease. We found that Prmt6 is present with Ppar-gamma on the Ppar-gamma and C/ebp-alpha promoter. It contributes to the repression of C/ebp-alpha expression, in part through its ability to induce H3R2me2a. During adipocyte differentiation, Prmt6 expression is reduced and the methyltransferase leaves the promoters. As a result, the expression of Ppar-gamma and C/ebp-alpha is upregulated and the adipocytic gene expression program is established. Inhibition of Prmt6 by a small molecule enhances adipogenesis, opening up the possibility of epigenetic manipulation of differentiation. Our data provide detailed information on the molecular mechanism controlling the Ppar-gamma-C/ebp-alpha feed-forward loop. Thus, they advance our understanding of adipogenesis in normal and aberrant adipogenesis.

Identifiants

pubmed: 38509237
doi: 10.1038/s41598-024-57310-9
pii: 10.1038/s41598-024-57310-9
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6656

Informations de copyright

© 2024. The Author(s).

Références

LeBlanc, S. E., Wu, Q., Lamba, P., Sif, S. & Imbalzano, A. N. Promoter-enhancer looping at the PPARgamma2 locus during adipogenic differentiation requires the Prmt5 methyltransferase. Nucl. Acids Res. 44, 5133–5147. https://doi.org/10.1093/nar/gkw129 (2016).
doi: 10.1093/nar/gkw129 pubmed: 26935580 pmcid: 4914087
LeBlanc, S. E. et al. Protein arginine methyltransferase 5 (Prmt5) promotes gene expression of peroxisome proliferator-activated receptor gamma2 (PPARgamma2) and its target genes during adipogenesis. Mol. Endocrinol. 26, 583–597. https://doi.org/10.1210/me.2011-1162 (2012).
doi: 10.1210/me.2011-1162 pubmed: 22361822 pmcid: 3327358
Hwang, J. W., So, Y. S., Bae, G. U., Kim, S. N. & Kim, Y. K. Protein arginine methyltransferase 6 suppresses adipogenic differentiation by repressing peroxisome proliferatoractivated receptor gamma activity. Int. J. Mol. Med. 43, 2462–2470. https://doi.org/10.3892/ijmm.2019.4147 (2019).
doi: 10.3892/ijmm.2019.4147 pubmed: 30942395
Hu, Y. J. et al. Transcriptional and post-transcriptional control of adipocyte differentiation by Jumonji domain-containing protein 6. Nucl. Acids Res. 43, 7790–7804. https://doi.org/10.1093/nar/gkv645 (2015).
doi: 10.1093/nar/gkv645 pubmed: 26117538 pmcid: 4652747
Picard, F. et al. Sirt1 promotes fat mobilization in white adipocytes by repressing PPAR-gamma. Nature 429, 771–776. https://doi.org/10.1038/nature02583 (2004).
doi: 10.1038/nature02583 pubmed: 15175761 pmcid: 2820247
Yadav, N. et al. CARM1 promotes adipocyte differentiation by coactivating PPARgamma. EMBO Rep. 9, 193–198. https://doi.org/10.1038/sj.embor.7401151 (2008).
doi: 10.1038/sj.embor.7401151 pubmed: 18188184 pmcid: 2246418
Gelman, L. et al. p300 interacts with the N- and C-terminal part of PPARgamma2 in a ligand-independent and -dependent manner, respectively. J. Biol. Chem. 274, 7681–7688. https://doi.org/10.1074/jbc.274.12.7681 (1999).
doi: 10.1074/jbc.274.12.7681 pubmed: 10075656
Kuvardina, O. N. et al. RUNX1 represses the erythroid gene expression program during megakaryocytic differentiation. Blood 125, 3570–3579. https://doi.org/10.1182/blood-2014-11-610519 (2015).
doi: 10.1182/blood-2014-11-610519 pubmed: 25911237 pmcid: 4463808
Herglotz, J. et al. Histone arginine methylation keeps RUNX1 target genes in an intermediate state. Oncogene 32, 2565–2575. https://doi.org/10.1038/onc.2012.274 (2013).
doi: 10.1038/onc.2012.274 pubmed: 22777353
Herkt, S. C. et al. Protein arginine methyltransferase 6 controls erythroid gene expression and differentiation of human CD34(+) progenitor cells. Haematologica 103, 18–29. https://doi.org/10.3324/haematol.2017.174516 (2018).
doi: 10.3324/haematol.2017.174516 pubmed: 29025910 pmcid: 5777187
Guccione, E. et al. Methylation of histone H3R2 by PRMT6 and H3K4 by an MLL complex are mutually exclusive. Nature 449, 933–937. https://doi.org/10.1038/nature06166 (2007).
doi: 10.1038/nature06166 pubmed: 17898714
Kirmizis, A. et al. Arginine methylation at histone H3R2 controls deposition of H3K4 trimethylation. Nature 449, 928–932. https://doi.org/10.1038/nature06160 (2007).
doi: 10.1038/nature06160 pubmed: 17898715 pmcid: 3350864
Hyllus, D. et al. PRMT6-mediated methylation of R2 in histone H3 antagonizes H3 K4 trimethylation. Genes Dev. 21, 3369–3380. https://doi.org/10.1101/gad.447007 (2007).
doi: 10.1101/gad.447007 pubmed: 18079182 pmcid: 2113036
Iberg, A. N. et al. Arginine methylation of the histone H3 tail impedes effector binding. J. Biol. Chem. 283, 3006–3010. https://doi.org/10.1074/jbc.C700192200 (2008).
doi: 10.1074/jbc.C700192200 pubmed: 18077460
Harrison, M. J., Tang, Y. H. & Dowhan, D. H. Protein arginine methyltransferase 6 regulates multiple aspects of gene expression. Nucl. Acids Res. 38, 2201–2216. https://doi.org/10.1093/nar/gkp1203 (2010).
doi: 10.1093/nar/gkp1203 pubmed: 20047962 pmcid: 2853117
Casadio, F. et al. H3R42me2a is a histone modification with positive transcriptional effects. Proc. Natl. Acad. Sci. USA 110, 14894–14899. https://doi.org/10.1073/pnas.1312925110 (2013).
doi: 10.1073/pnas.1312925110 pubmed: 23980157 pmcid: 3773778
Di Lorenzo, A., Yang, Y., Macaluso, M. & Bedford, M. T. A gain-of-function mouse model identifies PRMT6 as a NF-kappaB coactivator. Nucl.ic Acids Res. 42, 8297–8309. https://doi.org/10.1093/nar/gku530 (2014).
doi: 10.1093/nar/gku530
Sun, Y., Chung, H. H., Woo, A. R. & Lin, V. C. Protein arginine methyltransferase 6 enhances ligand-dependent and -independent activity of estrogen receptor alpha via distinct mechanisms. Biochim. Biophys. Acta 2067–2078, 2014. https://doi.org/10.1016/j.bbamcr.2014.04.008 (1843).
doi: 10.1016/j.bbamcr.2014.04.008
Scaramuzzino, C. et al. Protein arginine methyltransferase 6 enhances polyglutamine-expanded androgen receptor function and toxicity in spinal and bulbar muscular atrophy. Neuron 85, 88–100. https://doi.org/10.1016/j.neuron.2014.12.031 (2015).
doi: 10.1016/j.neuron.2014.12.031 pubmed: 25569348 pmcid: 4305189
Lefterova, M. I. et al. PPARgamma and C/EBP factors orchestrate adipocyte biology via adjacent binding on a genome-wide scale. Genes Dev. 22, 2941–2952. https://doi.org/10.1101/gad.1709008 (2008).
doi: 10.1101/gad.1709008 pubmed: 18981473 pmcid: 2577797
Nielsen, R. et al. Genome-wide profiling of PPARgamma:RXR and RNA polymerase II occupancy reveals temporal activation of distinct metabolic pathways and changes in RXR dimer composition during adipogenesis. Genes Dev. 22, 2953–2967. https://doi.org/10.1101/gad.501108 (2008).
doi: 10.1101/gad.501108 pubmed: 18981474 pmcid: 2577787
Madsen, M. S., Siersbaek, R., Boergesen, M., Nielsen, R. & Mandrup, S. Peroxisome proliferator-activated receptor gamma and C/EBPalpha synergistically activate key metabolic adipocyte genes by assisted loading. Mol. Cell. Biol. 34, 939–954. https://doi.org/10.1128/MCB.01344-13 (2014).
doi: 10.1128/MCB.01344-13 pubmed: 24379442 pmcid: 3958030
Rauch, A. & Mandrup, S. Transcriptional networks controlling stromal cell differentiation. Nat. Rev. Mol. Cell. Biol. 22, 465–482. https://doi.org/10.1038/s41580-021-00357-7 (2021).
doi: 10.1038/s41580-021-00357-7 pubmed: 33837369
Tontonoz, P. & Spiegelman, B. M. Fat and beyond: The diverse biology of PPARgamma. Annu. Rev. Biochem. 77, 289–312. https://doi.org/10.1146/annurev.biochem.77.061307.091829 (2008).
doi: 10.1146/annurev.biochem.77.061307.091829 pubmed: 18518822
Abdallah, B. M., Alzahrani, A. M., Abdel-Moneim, A. M., Ditzel, N. & Kassem, M. A simple and reliable protocol for long-term culture of murine bone marrow stromal (mesenchymal) stem cells that retained their in vitro and in vivo stemness in long-term culture. Biol. Proced. Online 21, 3. https://doi.org/10.1186/s12575-019-0091-3 (2019).
doi: 10.1186/s12575-019-0091-3 pubmed: 30733647 pmcid: 6357407
Shen, Y. et al. A first-in-class, highly selective and cell-active allosteric inhibitor of protein arginine methyltransferase 6. J. Med. Chem. 64, 3697–3706. https://doi.org/10.1021/acs.jmedchem.0c02160 (2021).
doi: 10.1021/acs.jmedchem.0c02160 pubmed: 33591753 pmcid: 8035306
Schneider, L. et al. PRMT6 activates cyclin D1 expression in conjunction with the transcription factor LEF1. Oncogenesis 10, 42. https://doi.org/10.1038/s41389-021-00332-z (2021).
doi: 10.1038/s41389-021-00332-z pubmed: 34001852 pmcid: 8129428
Cheng, Y. et al. Decoding m(6)A RNA methylome identifies PRMT6-regulated lipid transport promoting AML stem cell maintenance. Cell. Stem Cell. 30, 69–85. https://doi.org/10.1016/j.stem.2022.12.003 (2023).
doi: 10.1016/j.stem.2022.12.003 pubmed: 36574771
Chen, W. et al. SCF-FBXO24 regulates cell proliferation by mediating ubiquitination and degradation of PRMT6. Biochem. Biophys. Res. Commun. 530, 75–81. https://doi.org/10.1016/j.bbrc.2020.06.007 (2020).
doi: 10.1016/j.bbrc.2020.06.007 pubmed: 32828318
Li, T. et al. F-box protein FBXW17-mediated proteasomal degradation of protein methyltransferase PRMT6 exaggerates CSE-induced lung epithelial inflammation and apoptosis. Front. Cell. Dev. Biol. 9, 599020. https://doi.org/10.3389/fcell.2021.599020 (2021).
doi: 10.3389/fcell.2021.599020 pubmed: 33959602 pmcid: 8095709
Lee, Y. H. et al. Protein arginine methyltransferase 6 regulates embryonic stem cell identity. Stem Cells Dev. 21, 2613–2622. https://doi.org/10.1089/scd.2011.0330 (2012).
doi: 10.1089/scd.2011.0330 pubmed: 22455726 pmcid: 5729635
Viswakarma, N. et al. Coactivators in PPAR-regulated gene expression. PPAR Res. https://doi.org/10.1155/2010/250126 (2010).
doi: 10.1155/2010/250126 pubmed: 20885938 pmcid: 2946606
Zhong, Y. et al. PRMT4 facilitates white adipose tissue browning and thermogenesis by methylating PPARgamma. Diabetes 72, 1095–1111. https://doi.org/10.2337/db22-1016 (2023).
doi: 10.2337/db22-1016 pubmed: 37216643 pmcid: 10382653
Mikkelsen, T. S. et al. Comparative epigenomic analysis of murine and human adipogenesis. Cell 143, 156–169. https://doi.org/10.1016/j.cell.2010.09.006 (2010).
doi: 10.1016/j.cell.2010.09.006 pubmed: 20887899 pmcid: 2950833
Siersbaek, R. et al. Extensive chromatin remodelling and establishment of transcription factor “hotspots” during early adipogenesis. EMBO J. 30, 1459–1472. https://doi.org/10.1038/emboj.2011.65 (2011).
doi: 10.1038/emboj.2011.65 pubmed: 21427703 pmcid: 3102274
Sarusi Portuguez, A. et al. Hierarchical role for transcription factors and chromatin structure in genome organization along adipogenesis. FEBS J. 284, 3230–3244. https://doi.org/10.1111/febs.14183 (2017).
doi: 10.1111/febs.14183 pubmed: 28755519
Wang, D., Kuang, Y. L., Zhang, G. L., Xiao, K. & Liu, Y. L. Lysine-specific demethylase 1 in energy metabolism: A novel target for obesity. J. Nutr. 152, 1611–1620. https://doi.org/10.1093/jn/nxac080 (2022).
doi: 10.1093/jn/nxac080 pubmed: 35380692
Prakasam, R. et al. LSD1/PRMT6-targeting gene therapy to attenuate androgen receptor toxic gain-of-function ameliorates spinobulbar muscular atrophy phenotypes in flies and mice. Nat. Commun. https://doi.org/10.1038/s41467-023-36186-9 (2023).
doi: 10.1038/s41467-023-36186-9 pubmed: 37353549 pmcid: 10290075
Musri, M. M. et al. Histone demethylase LSD1 regulates adipogenesis. J. Biol. Chem. 285, 30034–30041. https://doi.org/10.1074/jbc.M110.151209 (2010).
doi: 10.1074/jbc.M110.151209 pubmed: 20656681 pmcid: 2943311
Zhao, X. et al. Methylation of RUNX1 by PRMT1 abrogates SIN3A binding and potentiates its transcriptional activity. Genes Dev. 22, 640–653. https://doi.org/10.1101/gad.1632608 (2008).
doi: 10.1101/gad.1632608 pubmed: 18316480 pmcid: 2259033
Hu, E., Tontonoz, P. & Spiegelman, B. M. Transdifferentiation of myoblasts by the adipogenic transcription factors PPAR gamma and C/EBP alpha. Proc. Natl. Acad. Sci. U.S.A. 92, 9856–9860. https://doi.org/10.1073/pnas.92.21.9856 (1995).
doi: 10.1073/pnas.92.21.9856 pubmed: 7568232 pmcid: 40901
Tontonoz, P., Hu, E. & Spiegelman, B. M. Stimulation of adipogenesis in fibroblasts by PPAR gamma 2, a lipid-activated transcription factor. Cell 79, 1147–1156. https://doi.org/10.1016/0092-8674(94)90006-x (1994).
doi: 10.1016/0092-8674(94)90006-x pubmed: 8001151
Rosen, E. D. et al. PPAR gamma is required for the differentiation of adipose tissue in vivo and in vitro. Mol. Cell. 4, 611–617. https://doi.org/10.1016/s1097-2765(00)80211-7 (1999).
doi: 10.1016/s1097-2765(00)80211-7 pubmed: 10549292
Wu, Z. et al. Cross-regulation of C/EBP alpha and PPAR gamma controls the transcriptional pathway of adipogenesis and insulin sensitivity. Mol. Cell. 3, 151–158. https://doi.org/10.1016/s1097-2765(00)80306-8 (1999).
doi: 10.1016/s1097-2765(00)80306-8 pubmed: 10078198

Auteurs

Mirjam Gerstner (M)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany.

Vivien Heller (V)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany.

Johannes Fechner (J)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany.

Benedikt Hermann (B)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany.

Lei Wang (L)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany.

Joern Lausen (J)

Department of Eukaryotic Genetics, Institute of Biomedical Genetics, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany. joern.lausen@ibmg.uni-stuttgart.de.

Classifications MeSH