Focal adhesions contain three specialized actin nanoscale layers.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
21 Mar 2024
Historique:
received: 25 01 2024
accepted: 13 03 2024
medline: 22 3 2024
pubmed: 22 3 2024
entrez: 22 3 2024
Statut: epublish

Résumé

Focal adhesions (FAs) connect inner workings of cell to the extracellular matrix to control cell adhesion, migration and mechanosensing. Previous studies demonstrated that FAs contain three vertical layers, which connect extracellular matrix to the cytoskeleton. By using super-resolution iPALM microscopy, we identify two additional nanoscale layers within FAs, specified by actin filaments bound to tropomyosin isoforms Tpm1.6 and Tpm3.2. The Tpm1.6-actin filaments, beneath the previously identified α-actinin cross-linked actin filaments, appear critical for adhesion maturation and controlled cell motility, whereas the adjacent Tpm3.2-actin filament layer beneath seems to facilitate adhesion disassembly. Mechanistically, Tpm3.2 stabilizes ACF-7/MACF1 and KANK-family proteins at adhesions, and hence targets microtubule plus-ends to FAs to catalyse their disassembly. Tpm3.2 depletion leads to disorganized microtubule network, abnormally stable FAs, and defects in tail retraction during migration. Thus, FAs are composed of distinct actin filament layers, and each may have specific roles in coupling adhesions to the cytoskeleton, or in controlling adhesion dynamics.

Identifiants

pubmed: 38514695
doi: 10.1038/s41467-024-46868-7
pii: 10.1038/s41467-024-46868-7
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2547

Subventions

Organisme : Academy of Finland (Suomen Akatemia)
ID : 346133

Informations de copyright

© 2024. The Author(s).

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Auteurs

Reena Kumari (R)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Katharina Ven (K)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Megan Chastney (M)

Turku Bioscience Centre, University of Turku and Åbo Akademi University, FI-20520, Turku, Finland.

Shrikant B Kokate (SB)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Johan Peränen (J)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Jesse Aaron (J)

Advanced Imaging Center, HHMI Janelia Research Campus, Ashburn, VA, 20147, USA.

Konstantin Kogan (K)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Leonardo Almeida-Souza (L)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.
Faculty of Biological and Environmental Sciences, University of Helsinki, Helsinki, Finland.

Elena Kremneva (E)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland.

Renaud Poincloux (R)

Institut de Pharmacologie et de Biologie Structurale, Université de Toulouse, CNRS, UPS, Toulouse, France.

Teng-Leong Chew (TL)

Advanced Imaging Center, HHMI Janelia Research Campus, Ashburn, VA, 20147, USA.

Peter W Gunning (PW)

School of Biomedical Sciences, UNSW Sydney, Wallace Wurth Building, Sydney, NSW 2052, Australia.

Johanna Ivaska (J)

Turku Bioscience Centre, University of Turku and Åbo Akademi University, FI-20520, Turku, Finland.
Department of Life Technologies, University of Turku, FI-20520, Turku, Finland.
InFLAMES Research Flagship Center, University of Turku, Turku, Finland.
Foundation for the Finnish Cancer Institute, Tukholmankatu 8, FI-00014, Helsinki, Finland.

Pekka Lappalainen (P)

HiLIFE Institute of Biotechnology, University of Helsinki, FI-00014, Helsinki, Finland. pekka.lappalainen@helsinki.fi.
Faculty of Biological and Environmental Sciences, University of Helsinki, Helsinki, Finland. pekka.lappalainen@helsinki.fi.

Classifications MeSH