PIEZO1 mechanically regulates the antitumour cytotoxicity of T lymphocytes.


Journal

Nature biomedical engineering
ISSN: 2157-846X
Titre abrégé: Nat Biomed Eng
Pays: England
ID NLM: 101696896

Informations de publication

Date de publication:
21 Mar 2024
Historique:
received: 27 06 2023
accepted: 05 02 2024
medline: 22 3 2024
pubmed: 22 3 2024
entrez: 22 3 2024
Statut: aheadofprint

Résumé

The killing function of cytotoxic T cells can be enhanced biochemically. Here we show that blocking the mechanical sensor PIEZO1 in T cells strengthens their traction forces and augments their cytotoxicity against tumour cells. By leveraging cytotoxic T cells collected from tumour models in mice and from patients with cancers, we show that PIEZO1 upregulates the transcriptional factor GRHL3, which in turn induces the expression of the E3 ubiquitin ligase RNF114. RNF114 binds to filamentous actin, causing its downregulation and rearrangement, which depresses traction forces in the T cells. In mice with tumours, the injection of cytotoxic T cells collected from the animals and treated with a PIEZO1 antagonist promoted their infiltration into the tumour and attenuated tumour growth. As an immunomechanical regulator, PIEZO1 could be targeted to enhance the outcomes of cancer immunotherapies.

Identifiants

pubmed: 38514773
doi: 10.1038/s41551-024-01188-5
pii: 10.1038/s41551-024-01188-5
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 82341009, 92169101
Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 82073184
Organisme : Chinese Academy of Medical Sciences (CAMS)
ID : 2021-I2M-1-066
Organisme : Natural Science Foundation of Beijing Municipality (Beijing Natural Science Foundation)
ID : Z200017

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Ruiyang Pang (R)

State Key Laboratory of Common Mechanism Research for Major Diseases, Department of Pharmacology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China.

Weihao Sun (W)

Department of Mechanics and Engineering Science, College of Engineering, Peking University, Beijing, China.
Wenzhou Institute, University of Chinese Academy of Sciences, Wenzhou, China.

Yingyun Yang (Y)

Department of Gastroenterology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.

Dahan Wen (D)

State Key Laboratory of Common Mechanism Research for Major Diseases, Department of Pharmacology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China.

Feng Lin (F)

Wenzhou Institute, University of Chinese Academy of Sciences, Wenzhou, China.

Dingding Wang (D)

State Key Laboratory of Common Mechanism Research for Major Diseases, Center for Bioinformatics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China.

Kailong Li (K)

Department of Biochemistry and Biophysics, Beijing Key Laboratory of Protein Posttranslational Modifications and Cell Function, School of Basic Medical Sciences, Peking University Health Science Centre, Beijing, China.

Ning Zhang (N)

MRC Protein Phosphorylation and Ubiquitylation Unit, School of Life Sciences, University of Dundee, Dundee, UK.
Institute of Blood Transfusion, Peking Union Medical College & Chinese Academy of Medical Sciences, Chengdu, China.

Junbo Liang (J)

State Key Laboratory of Common Mechanism Research for Major Diseases, Center for Bioinformatics, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China. liangjunbo@ibms.pumc.edu.cn.

Chunyang Xiong (C)

Department of Mechanics and Engineering Science, College of Engineering, Peking University, Beijing, China. cyxiong@pku.edu.cn.
Wenzhou Institute, University of Chinese Academy of Sciences, Wenzhou, China. cyxiong@pku.edu.cn.

Yuying Liu (Y)

State Key Laboratory of Common Mechanism Research for Major Diseases, Department of Pharmacology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China. yuying@ibms.pumc.edu.cn.
Clinical Immunology Centre, CAMS, Beijing, China. yuying@ibms.pumc.edu.cn.

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