Real-World experience with efgartigimod in patients with myasthenia gravis.

Efgartigimod Generalized myasthenia gravis MG-ADL Prednisone

Journal

Journal of neurology
ISSN: 1432-1459
Titre abrégé: J Neurol
Pays: Germany
ID NLM: 0423161

Informations de publication

Date de publication:
25 Mar 2024
Historique:
received: 29 01 2024
accepted: 29 02 2024
revised: 28 02 2024
medline: 26 3 2024
pubmed: 26 3 2024
entrez: 26 3 2024
Statut: aheadofprint

Résumé

Recommendations for the treatment of myasthenia gravis (MG) have been difficult to develop because of limited evidence from large randomized controlled trials. New drugs and treatment approaches have recently been shown to be effective in phase 3 studies in seropositive generalized (g) MG. One such drug is efgartigimod, a human-Fc-fragment of IgG1, with a high affinity for the endosomal FcRn. We conducted a multicenter study to evaluate the real-world clinical and safety effects of efgartigimod in 22 gMG patients. We evaluated the strategies for the timing of re-treatment with it. The participants received a total of 59 efgartigimod -treatment cycles. The median number of cycles was 2 (range 1-6). Twenty patients (86.3%) improved by at least 2 MG-ADL points after the first treatment cycle. The median MG-ADL score at baseline was 6.5 (range: 3-17) and 2.5 (range: 0-9) post-treatment (p < 0.001). A consistent improvement of at least 2 points in the MG-ADL score after each cycle occurs in 18 patients. The effect duration of the treatment was usually between 4 and 12 weeks. Two major clinical patterns of treatment response were found. Treatment with efgartigimod was also associated with significant reductions of prednisone doses Overall, the treatment was safe and associated with only minor adverse events. The single fatality was apparently due tosevere respiratory failure. We found that efgartigimod is clinically effective, may be used as a steroid sparing agent and is generally safe for gMG patients. We recommend a personalized preventive treatment approach until clinical stabilization, followed by discontinuation and periodic evaluations.

Identifiants

pubmed: 38528163
doi: 10.1007/s00415-024-12293-5
pii: 10.1007/s00415-024-12293-5
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany.

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Auteurs

Lior Fuchs (L)

Faculty of Medicine & Health Sciences, Tel Aviv University, Tel Aviv-Yafo, Israel.

Shahar Shelly (S)

Department of Neurology, Rambam Medical Center, Haifa, Israel.
Rappaport Faculty of Medicine, Technion-Israel Institute of Technology, Haifa, Israel.

Ifat Vigiser (I)

Faculty of Medicine & Health Sciences, Tel Aviv University, Tel Aviv-Yafo, Israel.
Neuroimmunology and MS Unit, Neurology Institute, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Hadar Kolb (H)

Neuroimmunology and MS Unit, Neurology Institute, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Keren Regev (K)

Neuroimmunology and MS Unit, Neurology Institute, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Yoel Schwartzmann (Y)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

Adi Vaknin-Dembinsky (A)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.
Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

Amir Dori (A)

Faculty of Medicine & Health Sciences, Tel Aviv University, Tel Aviv-Yafo, Israel.
Department of Neurology, Sheba Medical Center, Ramat-Gan, Israel.

Arnon Karni (A)

Faculty of Medicine & Health Sciences, Tel Aviv University, Tel Aviv-Yafo, Israel. arnonk@tlvmc.gov.il.
Neuroimmunology and MS Unit, Neurology Institute, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel. arnonk@tlvmc.gov.il.
Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel. arnonk@tlvmc.gov.il.
The Neurology Institute, Tel Aviv Sourasky Medical Center, 6 Weizmann Street, 6423906, Tel Aviv, Israel. arnonk@tlvmc.gov.il.

Classifications MeSH