Sialyl-Tn serves as a potential therapeutic target for ovarian cancer.

Companion diagnostic Ovarian cancer Sialyl-Tn Targeted therapy

Journal

Journal of ovarian research
ISSN: 1757-2215
Titre abrégé: J Ovarian Res
Pays: England
ID NLM: 101474849

Informations de publication

Date de publication:
02 Apr 2024
Historique:
received: 24 08 2023
accepted: 21 03 2024
medline: 3 4 2024
pubmed: 3 4 2024
entrez: 2 4 2024
Statut: epublish

Résumé

Ovarian cancer remains the deadliest of the gynecologic cancers in the United States. There have been limited advances in treatment strategies that have seen marked increases in overall survival. Thus, it is essential to continue developing and validating new treatment strategies and markers to identify patients who would benefit from the new strategy. In this report, we sought to further validate applications for a novel humanized anti-Sialyl Tn antibody-drug conjugate (anti-STn-ADC) in ovarian cancer. We aimed to further test a humanized anti-STn-ADC in sialyl-Tn (STn) positive and negative ovarian cancer cell line, patient-derived organoid (PDO), and patient-derived xenograft (PDX) models. Furthermore, we sought to determine whether serum STn levels would reflect STn positivity in the tumor samples enabling us to identify patients that an anti-STn-ADC strategy would best serve. We developed a custom ELISA with high specificity and sensitivity, that was used to assess whether circulating STn levels would correlate with stage, progression-free survival, overall survival, and its value in augmenting CA-125 as a diagnostic. Lastly, we assessed whether the serum levels reflected what was observed via immunohistochemical analysis in a subset of tumor samples. Our in vitro experiments further define the specificity of the anti-STn-ADC. The ovarian cancer PDO, and PDX models provide additional support for an anti-STn-ADC-based strategy for targeting ovarian cancer. The custom serum ELISA was informative in potential triaging of patients with elevated levels of STn. However, it was not sensitive enough to add value to existing CA-125 levels for a diagnostic. While the ELISA identified non-serous ovarian tumors with low CA-125 levels, the sample numbers were too small to provide any confidence the STn ELISA would meaningfully add to CA-125 for diagnosis. Our preclinical data support the concept that an anti-STn-ADC may be a viable option for treating patients with elevated STn levels. Moreover, our STn-based ELISA could complement IHC in identifying patients with whom an anti-STn-based strategy might be more effective.

Sections du résumé

BACKGROUND BACKGROUND
Ovarian cancer remains the deadliest of the gynecologic cancers in the United States. There have been limited advances in treatment strategies that have seen marked increases in overall survival. Thus, it is essential to continue developing and validating new treatment strategies and markers to identify patients who would benefit from the new strategy. In this report, we sought to further validate applications for a novel humanized anti-Sialyl Tn antibody-drug conjugate (anti-STn-ADC) in ovarian cancer.
METHODS METHODS
We aimed to further test a humanized anti-STn-ADC in sialyl-Tn (STn) positive and negative ovarian cancer cell line, patient-derived organoid (PDO), and patient-derived xenograft (PDX) models. Furthermore, we sought to determine whether serum STn levels would reflect STn positivity in the tumor samples enabling us to identify patients that an anti-STn-ADC strategy would best serve. We developed a custom ELISA with high specificity and sensitivity, that was used to assess whether circulating STn levels would correlate with stage, progression-free survival, overall survival, and its value in augmenting CA-125 as a diagnostic. Lastly, we assessed whether the serum levels reflected what was observed via immunohistochemical analysis in a subset of tumor samples.
RESULTS RESULTS
Our in vitro experiments further define the specificity of the anti-STn-ADC. The ovarian cancer PDO, and PDX models provide additional support for an anti-STn-ADC-based strategy for targeting ovarian cancer. The custom serum ELISA was informative in potential triaging of patients with elevated levels of STn. However, it was not sensitive enough to add value to existing CA-125 levels for a diagnostic. While the ELISA identified non-serous ovarian tumors with low CA-125 levels, the sample numbers were too small to provide any confidence the STn ELISA would meaningfully add to CA-125 for diagnosis.
CONCLUSIONS CONCLUSIONS
Our preclinical data support the concept that an anti-STn-ADC may be a viable option for treating patients with elevated STn levels. Moreover, our STn-based ELISA could complement IHC in identifying patients with whom an anti-STn-based strategy might be more effective.

Identifiants

pubmed: 38566237
doi: 10.1186/s13048-024-01397-1
pii: 10.1186/s13048-024-01397-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

71

Subventions

Organisme : NCI NIH HHS
ID : HHSN261200700063C
Pays : United States
Organisme : NCI NIH HHS
ID : HHSN261200700063C
Pays : United States
Organisme : NCI NIH HHS
ID : HHSN261200700063C
Pays : United States
Organisme : NCI NIH HHS
ID : HHSN261200700063C
Pays : United States
Organisme : NCI NIH HHS
ID : HHSN261200700063C
Pays : United States
Organisme : NIH HHS
ID : NIH 1DP5OD029637
Pays : United States

Informations de copyright

© 2024. The Author(s).

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Auteurs

Linah Al-Alem (L)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.
Obstetrics, Gynecology and Reproductive Biology, Harvard Medical School, Boston, MA, 02115, USA.

Jillian M Prendergast (JM)

Siamab Therapeutics, Inc, Newton, MA, 02458, USA.

Justin Clark (J)

Siamab Therapeutics, Inc, Newton, MA, 02458, USA.

Bianca Zarrella (B)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.

Dominique T Zarrella (DT)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.

Sarah J Hill (SJ)

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA, 02215, USA.
Division of Molecular and Cellular Oncology, Dana-Farber Cancer Institute, Boston, MA, 02215, USA.
Department of Medicine, Harvard Medical School, Boston, MA, 02115, USA.

Whitfield B Growdon (WB)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.
Obstetrics, Gynecology and Reproductive Biology, Harvard Medical School, Boston, MA, 02115, USA.
Division of Gynecologic Oncology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.

Venkatesh Pooladanda (V)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA.
Obstetrics, Gynecology and Reproductive Biology, Harvard Medical School, Boston, MA, 02115, USA.

David R Spriggs (DR)

Division of Hematology-Oncology, Massachusetts General Hospital, 55 Fruit St, Boston, MA, 02114, USA.
Department of Medicine, Massachusetts General Hospital, Boston, MA, 02114, USA.

Daniel Cramer (D)

Obstetrics and Gynecology Epidemiology Center, Brigham and Women's Hospital, Boston, MA, 02115, USA.

Kevin M Elias (KM)

Division of Gynecologic Oncology, Department of Obstetrics and Gynecology, Brigham and Women's Hospital, Boston, MA, 02115, USA.

Rawan I Nazer (RI)

Siamab Therapeutics, Inc, Newton, MA, 02458, USA.

Steven J Skates (SJ)

Biostatistics Center, Massachusetts General Hospital, Boston, MA, 02114, USA.

Jeff Behrens (J)

Siamab Therapeutics, Inc, Newton, MA, 02458, USA.

Daniel T Dransfield (DT)

Siamab Therapeutics, Inc, Newton, MA, 02458, USA.

Bo R Rueda (BR)

Vincent Center for Reproductive Biology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA. brueda@mgh.harvard.edu.
Obstetrics, Gynecology and Reproductive Biology, Harvard Medical School, Boston, MA, 02115, USA. brueda@mgh.harvard.edu.
Division of Gynecologic Oncology, Department of Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, 02114, USA. brueda@mgh.harvard.edu.

Classifications MeSH