Gut microbiome signature and nasal lavage inflammatory markers in young people with asthma.

Asthma gut microbiota microbiome stool

Journal

The journal of allergy and clinical immunology. Global
ISSN: 2772-8293
Titre abrégé: J Allergy Clin Immunol Glob
Pays: United States
ID NLM: 9918453488706676

Informations de publication

Date de publication:
May 2024
Historique:
received: 05 04 2023
revised: 04 10 2023
accepted: 24 12 2023
medline: 8 4 2024
pubmed: 8 4 2024
entrez: 8 4 2024
Statut: epublish

Résumé

Asthma is a complex disease and a severe global public health problem resulting from interactions between genetic background and environmental exposures. It has been suggested that gut microbiota may be related to asthma development; however, such relationships needs further investigation. This study aimed to characterize the gut microbiota as well as the nasal lavage cytokine profile of asthmatic and nonasthmatic individuals. Stool and nasal lavage samples were collected from 29 children and adolescents with type 2 asthma and 28 children without asthma in Brazil. Amplicon sequencing of the stool bacterial V4 region of the 16S rRNA gene was performed using Illumina MiSeq. Microbiota analysis was performed by QIIME 2 and PICRUSt2. Type 2 asthma phenotype was characterized by high sputum eosinophil counts and positive skin prick tests for house dust mite, cockroach, and/or cat or dog dander. The nasal immune marker profile was assessed using a customized multiplex panel. Stool microbiota differed significantly between asthmatic and nonasthmatic participants ( The stool microbiota differed between asthmatic and nonasthmatic young people in Brazil. Asthma was associated with higher

Sections du résumé

Background UNASSIGNED
Asthma is a complex disease and a severe global public health problem resulting from interactions between genetic background and environmental exposures. It has been suggested that gut microbiota may be related to asthma development; however, such relationships needs further investigation.
Objective UNASSIGNED
This study aimed to characterize the gut microbiota as well as the nasal lavage cytokine profile of asthmatic and nonasthmatic individuals.
Methods UNASSIGNED
Stool and nasal lavage samples were collected from 29 children and adolescents with type 2 asthma and 28 children without asthma in Brazil. Amplicon sequencing of the stool bacterial V4 region of the 16S rRNA gene was performed using Illumina MiSeq. Microbiota analysis was performed by QIIME 2 and PICRUSt2. Type 2 asthma phenotype was characterized by high sputum eosinophil counts and positive skin prick tests for house dust mite, cockroach, and/or cat or dog dander. The nasal immune marker profile was assessed using a customized multiplex panel.
Results UNASSIGNED
Stool microbiota differed significantly between asthmatic and nonasthmatic participants (
Conclusion UNASSIGNED
The stool microbiota differed between asthmatic and nonasthmatic young people in Brazil. Asthma was associated with higher

Identifiants

pubmed: 38585449
doi: 10.1016/j.jacig.2024.100242
pii: S2772-8293(24)00038-9
pmc: PMC10998106
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100242

Informations de copyright

© 2024 Published by Elsevier Inc. on behalf of the American Academy of Allergy, Asthma & Immunology.

Déclaration de conflit d'intérêts

Supported by the 10.13039/501100000781European Research Council under the European Union’s Seventh Framework Programme (FP7/2007-2013), ERC grants 668954 and 101020088; the 10.13039/501100002322Coordenação de Aperfeiçoamento de Pessoal de Nível Superior–Brasil, finance code 001; the 10.13039/501100006181Fundação de Amparo à Pesquisa do Estado da Bahia (PNE0003/2014, PNX0001/2014, and BOL0281/2020); and the 10.13039/501100003593Conselho Nacional de Desenvolvimento Científico e Tecnológico (40314 and 308521/2019-6). Disclosure of potential conflict of interest: The authors declare that they have no relevant conflicts of interest.

Auteurs

Bianca Sampaio Dotto Fiuza (B)

Instituto de Ciências da Saúde, Universidade Federal da Bahia, Salvador, Brazil.

Candace Machado de Andrade (C)

Instituto de Ciências da Saúde, Universidade Federal da Bahia, Salvador, Brazil.

Pedro Milet Meirelles (PM)

Instituto de Biologia, Universidade Federal da Bahia, Salvador, Brazil.
Instituto Nacional de Ciência e Tecnologia em Estudos Interdisciplinares e Transdisciplinares em Ecologia e Evolução (IN-TREE), Salvador, Brazil.

Jorley Santos da Silva (J)

Instituto de Biologia, Universidade Federal da Bahia, Salvador, Brazil.

Milca de Jesus Silva (M)

Instituto de Ciências da Saúde, Universidade Federal da Bahia, Salvador, Brazil.

Cinthia Vila Nova Santana (C)

Fundação ProAR Salvador, Salvador, Brazil.

Gabriela Pimentel Pinheiro (G)

Fundação ProAR Salvador, Salvador, Brazil.

Harriet Mpairwe (H)

MRC/UVRI and LSHTM Uganda Research Unit, Entebbe, Uganda.

Philip Cooper (P)

Fundacion Ecuatoriana Para Investigacion en Salud, Quito, Ecuador.
Universidad Internacional del Ecuador, Quito, Ecuador.
St George's University of London, London, United Kingdom.

Collin Brooks (C)

Centre for Public Health Research, Massey University, Wellington, New Zealand.

Lucy Pembrey (L)

London School of Hygiene and Tropical Medicine, London, United Kingdom.

Steven Taylor (S)

College of Medicine and Public Health, Flinders University, Adelaide, Australia.

Jeroen Douwes (J)

Centre for Public Health Research, Massey University, Wellington, New Zealand.

Álvaro A Cruz (ÁA)

Fundação ProAR Salvador, Salvador, Brazil.
Faculdade de Medicina, Universidade Federal da Bahia, Salvador, Brazil.

Mauricio L Barreto (ML)

Centro de Integração de Dados e Conhecimentos para Saúde, Fiocruz, Salvador, Brazil.

Neil Pearce (N)

Centre for Public Health Research, Massey University, Wellington, New Zealand.
London School of Hygiene and Tropical Medicine, London, United Kingdom.

Camila A V Figueiredo (CAV)

Instituto de Ciências da Saúde, Universidade Federal da Bahia, Salvador, Brazil.

Classifications MeSH