Altered COVID-19 immunity in children with asthma by atopic status.

Asthma RSV SARS-CoV-2 children cockroach immunity

Journal

The journal of allergy and clinical immunology. Global
ISSN: 2772-8293
Titre abrégé: J Allergy Clin Immunol Glob
Pays: United States
ID NLM: 9918453488706676

Informations de publication

Date de publication:
May 2024
Historique:
received: 22 08 2023
revised: 13 12 2023
accepted: 03 01 2024
medline: 9 4 2024
pubmed: 9 4 2024
entrez: 9 4 2024
Statut: epublish

Résumé

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection causes a spectrum of clinical outcomes that may be complicated by severe asthma. Antiviral immunity is often compromised in patients with asthma; however, whether this is true for SARS-CoV-2 immunity and children is unknown. We aimed to evaluate SARS-CoV-2 immunity in children with asthma on the basis of infection or vaccination history and compared to respiratory syncytial viral or allergen (eg, cockroach, dust mite)-specific immunity. Fifty-three children from an urban asthma study were evaluated for medical history, lung function, and virus- or allergen-specific immunity using antibody or T-cell assays. Polyclonal antibody responses to spike were observed in most children from infection and/or vaccination history. Children with atopic asthma or high allergen-specific IgE, particularly to dust mites, exhibited reduced seroconversion, antibody magnitude, and SARS-CoV-2 virus neutralization after SARS-CoV-2 infection or vaccination. T Mechanisms underpinning atopic and nonatopic asthma may complicate the development of memory to SARS-CoV-2 infection or vaccination and lead to a higher risk of repeated infection in these children.

Sections du résumé

Background UNASSIGNED
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection causes a spectrum of clinical outcomes that may be complicated by severe asthma. Antiviral immunity is often compromised in patients with asthma; however, whether this is true for SARS-CoV-2 immunity and children is unknown.
Objective UNASSIGNED
We aimed to evaluate SARS-CoV-2 immunity in children with asthma on the basis of infection or vaccination history and compared to respiratory syncytial viral or allergen (eg, cockroach, dust mite)-specific immunity.
Methods UNASSIGNED
Fifty-three children from an urban asthma study were evaluated for medical history, lung function, and virus- or allergen-specific immunity using antibody or T-cell assays.
Results UNASSIGNED
Polyclonal antibody responses to spike were observed in most children from infection and/or vaccination history. Children with atopic asthma or high allergen-specific IgE, particularly to dust mites, exhibited reduced seroconversion, antibody magnitude, and SARS-CoV-2 virus neutralization after SARS-CoV-2 infection or vaccination. T
Conclusion UNASSIGNED
Mechanisms underpinning atopic and nonatopic asthma may complicate the development of memory to SARS-CoV-2 infection or vaccination and lead to a higher risk of repeated infection in these children.

Identifiants

pubmed: 38590754
doi: 10.1016/j.jacig.2024.100236
pii: S2772-8293(24)00032-8
pmc: PMC11000189
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100236

Informations de copyright

© 2024 The Authors.

Auteurs

Sherry Tong (S)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Jordan C Scott (JC)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Enwono Eyoh (E)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Derek W Werthmann (DW)

Department of Epidemiology, Tulane University School of Public Health and Tropical Medicine, New Orleans, La.

Addison E Stone (AE)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Amelie E Murrell (AE)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Gilberto Sabino-Santos (G)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Ivy V Trinh (IV)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Sruti Chandra (S)

Department of Pediatrics, Tulane University School of Medicine, New Orleans, La.

Debra H Elliott (DH)

Department of Pediatrics, Tulane University School of Medicine, New Orleans, La.

Ashley R Smira (AR)

Department of Pediatrics, Tulane University School of Medicine, New Orleans, La.

Jalene V Velazquez (JV)

Paul G. Allen School of Global Health, Washington State University, Pullman, Wash.

John Schieffelin (J)

Department of Pediatrics, Tulane University School of Medicine, New Orleans, La.

Bo Ning (B)

Center for Cellular and Molecular Diagnostics, Tulane University School of Medicine, New Orleans, La.
Department of Biochemistry & Molecular Biology, Tulane University School of Medicine, New Orleans, La.

Tony Hu (T)

Center for Cellular and Molecular Diagnostics, Tulane University School of Medicine, New Orleans, La.
Department of Biochemistry & Molecular Biology, Tulane University School of Medicine, New Orleans, La.

Jay K Kolls (JK)

Department of Medicine, Tulane University School of Medicine, New Orleans, La.

Samuel J Landry (SJ)

Department of Biochemistry & Molecular Biology, Tulane University School of Medicine, New Orleans, La.

Kevin J Zwezdaryk (KJ)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

James E Robinson (JE)

Department of Pediatrics, Tulane University School of Medicine, New Orleans, La.

Bronwyn M Gunn (BM)

Paul G. Allen School of Global Health, Washington State University, Pullman, Wash.

Felicia A Rabito (FA)

Department of Epidemiology, Tulane University School of Public Health and Tropical Medicine, New Orleans, La.

Elizabeth B Norton (EB)

Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, La.

Classifications MeSH