Clinical impact of panel gene sequencing on therapy of advanced cancers of the digestive system: a retrospective, single center study.


Journal

BMC cancer
ISSN: 1471-2407
Titre abrégé: BMC Cancer
Pays: England
ID NLM: 100967800

Informations de publication

Date de publication:
25 Apr 2024
Historique:
received: 27 11 2023
accepted: 15 04 2024
medline: 26 4 2024
pubmed: 26 4 2024
entrez: 25 4 2024
Statut: epublish

Résumé

Panel gene sequencing is an established diagnostic tool for precision oncology of solid tumors, but its utility for the treatment of cancers of the digestive system in clinical routine is less well documented. We retrospectively identified patients with advanced or metastatic gastrointestinal, pancreaticobiliary or hepatic cancers who received panel gene sequencing at a tertiary university hospital from 2015 to 2022. For these cases, we determined the spectrum of genetic alterations, clinicopathological parameters and treatment courses. Assessment of actionability of genetic alterations was based on the OncoKB database, cancer-specific ESMO treatment guidelines, and recommendations of the local molecular tumor board. In total, 155 patients received panel gene sequencing using either the Oncomine Focus (62 cases), Comprehensive (91 cases) or Childhood Cancer Research Assay (2 cases). The mean age of patients was 61 years (range 24-90) and 37% were female. Most patients suffered from either colorectal cancer (53%) or cholangiocellular carcinoma (19%). 327 genetic alterations were discovered in 123 tumor samples, with an average number of 2.1 alterations per tumor. The most frequently altered genes were TP53, KRAS and PIK3CA. Actionable gene alterations were detected in 13.5-56.8% of tumors, according to ESMO guidelines or the OncoKB database, respectively. Thirteen patients were treated with targeted therapies based on identified molecular alterations, with a median progression-free survival of 8.8 months. Actionable genetic alterations are frequently detected by panel gene sequencing in patients with advanced cancers of the digestive tract, providing clinical benefit in selected cases. However, for the majority of identified actionable alterations, sufficient clinical evidence for targeted treatments is still lacking.

Sections du résumé

BACKGROUND BACKGROUND
Panel gene sequencing is an established diagnostic tool for precision oncology of solid tumors, but its utility for the treatment of cancers of the digestive system in clinical routine is less well documented.
METHODS METHODS
We retrospectively identified patients with advanced or metastatic gastrointestinal, pancreaticobiliary or hepatic cancers who received panel gene sequencing at a tertiary university hospital from 2015 to 2022. For these cases, we determined the spectrum of genetic alterations, clinicopathological parameters and treatment courses. Assessment of actionability of genetic alterations was based on the OncoKB database, cancer-specific ESMO treatment guidelines, and recommendations of the local molecular tumor board.
RESULTS RESULTS
In total, 155 patients received panel gene sequencing using either the Oncomine Focus (62 cases), Comprehensive (91 cases) or Childhood Cancer Research Assay (2 cases). The mean age of patients was 61 years (range 24-90) and 37% were female. Most patients suffered from either colorectal cancer (53%) or cholangiocellular carcinoma (19%). 327 genetic alterations were discovered in 123 tumor samples, with an average number of 2.1 alterations per tumor. The most frequently altered genes were TP53, KRAS and PIK3CA. Actionable gene alterations were detected in 13.5-56.8% of tumors, according to ESMO guidelines or the OncoKB database, respectively. Thirteen patients were treated with targeted therapies based on identified molecular alterations, with a median progression-free survival of 8.8 months.
CONCLUSIONS CONCLUSIONS
Actionable genetic alterations are frequently detected by panel gene sequencing in patients with advanced cancers of the digestive tract, providing clinical benefit in selected cases. However, for the majority of identified actionable alterations, sufficient clinical evidence for targeted treatments is still lacking.

Identifiants

pubmed: 38664720
doi: 10.1186/s12885-024-12261-2
pii: 10.1186/s12885-024-12261-2
doi:

Substances chimiques

Biomarkers, Tumor 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

526

Informations de copyright

© 2024. The Author(s).

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Auteurs

Lena Dreikhausen (L)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Anna Klupsch (A)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Isabella Wiest (I)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Qiyun Xiao (Q)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Nadine Schulte (N)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Johannes Betge (J)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Junior Clinical Cooperation Unit Translational Gastrointestinal Oncology and Preclinical Models, German Cancer Research Center, Heidelberg, Germany.
Mannheim Cancer Center, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Medical Faculty Mannheim, DKFZ-Hector Cancer Institute, Heidelberg University, Mannheim, Germany.

Tobias Boch (T)

Department of Medicine III, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Medical Faculty Mannheim, DKFZ-Hector Cancer Institute, Heidelberg University, Mannheim, Germany.

Christoph Brochhausen (C)

Institute of Pathology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Timo Gaiser (T)

Institute of Pathology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Ralf-Dieter Hofheinz (RD)

Mannheim Cancer Center, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Department of Medicine III, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

Matthias Ebert (M)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Mannheim Cancer Center, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.
Medical Faculty Mannheim, DKFZ-Hector Cancer Institute, Heidelberg University, Mannheim, Germany.

Tianzuo Zhan (T)

Department of Medicine II, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany. tianzuo.zhan@medma.uni-heidelberg.de.
Mannheim Cancer Center, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany. tianzuo.zhan@medma.uni-heidelberg.de.
Medical Faculty Mannheim, DKFZ-Hector Cancer Institute, Heidelberg University, Mannheim, Germany. tianzuo.zhan@medma.uni-heidelberg.de.

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