Antioxidant and anti-inflammatory function of Eupatorium adenophora Spreng leaves (EASL) on human intestinal Caco-2 cells treated with tert-butyl hydroperoxide.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
07 May 2024
Historique:
received: 13 02 2024
accepted: 30 04 2024
medline: 8 5 2024
pubmed: 8 5 2024
entrez: 7 5 2024
Statut: epublish

Résumé

Chronic non-communicable diseases (CNCDs) pose a significant public health challenge. Addressing this issue, there has been a notable breakthrough in the prevention and mitigation of NCDs through the use of antioxidants and anti-inflammatory agents. In this study, we aim to explore the effectiveness of Eupatorium adenophora Spreng leaves (EASL) as an antioxidant and anti-inflammatory agent, and its potential applications. To construct a cellular model of oxidative damage and inflammation, Caco-2 cells were treated with tert-butyl hydroperoxide (t-BHP). The biocompatibility of EASL-AE with Caco-2 cells was assessed using the MTT assay, while compatibility was further verified by measuring LDH release and the protective effect against oxidative damage was also assessed using the MTT assay. Additionally, we measured intracellular oxidative stress indicators such as ROS and 8-OHdG, as well as inflammatory pathway signalling protein NFκB and inflammatory factors TNF-α and IL-1β using ELISA, to evaluate the antioxidant and anti-inflammatory capacity of EASL-AE. The scavenging capacity of EASL-AE against free radicals was determined through the DPPH Assay and ABTS Assay. Furthermore, we measured the total phenolic, total flavonoid, and total polysaccharide contents using common chemical methods. The chemical composition of EASL-AE was analyzed using the LC-MS/MS technique. Our findings demonstrate that EASL-AE is biocompatible with Caco-2 cells and non-toxic at experimental levels. Moreover, EASL-AE exhibits a significant protective effect on Caco-2 cells subjected to oxidative damage. The antioxidant effect of EASL-AE involves the scavenging of intracellular ROS, while its anti-inflammatory effect is achieved by down-regulation of the NFκB pathway. Which in turn reduces the release of inflammatory factors TNF-α and IL-1β. Through LC-MS/MS analysis, we identified 222 compounds in EASL-AE, among which gentianic acid, procaine and L-tyrosine were the compounds with high antioxidant capacity and may be the effective constituent for EASL-AE with antioxidant activity. These results suggest that EASL-AE is a natural and high-quality antioxidant and anti-inflammatory biomaterial that warrants further investigation. It holds great potential for applications in healthcare and other related fields.

Identifiants

pubmed: 38714697
doi: 10.1038/s41598-024-61012-7
pii: 10.1038/s41598-024-61012-7
doi:

Substances chimiques

tert-Butylhydroperoxide 955VYL842B
Antioxidants 0
Anti-Inflammatory Agents 0
Plant Extracts 0
Reactive Oxygen Species 0
NF-kappa B 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

10509

Subventions

Organisme : Dali Science and Technology Special Project
ID : 202301A020023

Informations de copyright

© 2024. The Author(s).

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Auteurs

Li Zheng-Qiang (L)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

Ni Jun (N)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

Zhu Xin-Yu (Z)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

Zhang Chao-Zhi (Z)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

An Rui (A)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

Yang Xu (Y)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.

She Rong (S)

Institute of Natural Antioxidants and Anti-Inflammation, Dali University, Dali, 671003, Yunnan, China. sher@eastern-himalaya.cn.
Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China. sher@eastern-himalaya.cn.

Yang Xiao-Yan (Y)

Institute of Eastern-Himalaya Biodiversity Research, Dali University, Dali, 671003, Yunnan, China.
The Provincial Innovation Team of Biodiversity Conservation and Utility of the Three Parallel Rivers Region From Dali University, Dali, 671003, Yunnan, China.

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Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
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Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
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Humans Yoga Low Back Pain Female Male

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