Estimated pulse wave velocity in metabolic dysfunction-associated steatotic liver disease and all-cause/cause-specific mortality.

MASLD arterial stiffness death steatotic liver disease vascular age

Journal

Journal of gastroenterology and hepatology
ISSN: 1440-1746
Titre abrégé: J Gastroenterol Hepatol
Pays: Australia
ID NLM: 8607909

Informations de publication

Date de publication:
13 May 2024
Historique:
revised: 10 04 2024
received: 20 02 2024
accepted: 30 04 2024
medline: 14 5 2024
pubmed: 14 5 2024
entrez: 13 5 2024
Statut: aheadofprint

Résumé

Several reports show a significant association between metabolic dysfunction-associated steatotic liver disease (MASLD) and arterial stiffness (estimated pulse wave velocity [ePWV]) as a surrogate marker of vascular age. We investigate whether ePWV as arterial stiffness in MASLD is associated with all-cause/cause-specific mortality. This cohort study was based on the third National Health and Nutrition Examination Survey (NHANES, 1988-1994) and NHANES 2007-2014 and linked mortality datasets through 2019. Cox regression models assessed the association between ePWV categorized by quartile and all-cause/cause-specific mortality among individuals with MASLD. During the follow-up of a median of 26.3 years (interquartile range: 19.9-27.9), higher levels of ePWV among individuals with MASLD were associated with increased all-cause mortality, which remained significant after adjusting for demographic, lifestyle, clinical, and metabolic risk factors. Furthermore, higher ePWV in MASLD was associated with higher cardiovascular mortality. There was a 44% (hazard ratio: 1.44, 95% confidence interval: 1.32-1.58) increase in all-cause mortality and a 53% (hazard ratio: 1.53, 95% confidence interval: 1.32-1.77) increase in cardiovascular mortality for every 1 m/s increase in ePWV in MASLD. However, there was no significant association between ePWV and cancer-related mortality. Sensitivity analyses using the NHANES 2007-2014 dataset showed results identical to the original analysis. Higher ePWV in MASLD was associated with a higher risk of all-cause and cardiovascular mortality beyond traditional cardiovascular risk factors. Screening for ePWV in individuals with MASLD may be an effective and beneficial approach to reducing all-cause and cardiovascular mortality.

Sections du résumé

BACKGROUND AND AIM OBJECTIVE
Several reports show a significant association between metabolic dysfunction-associated steatotic liver disease (MASLD) and arterial stiffness (estimated pulse wave velocity [ePWV]) as a surrogate marker of vascular age. We investigate whether ePWV as arterial stiffness in MASLD is associated with all-cause/cause-specific mortality.
METHODS METHODS
This cohort study was based on the third National Health and Nutrition Examination Survey (NHANES, 1988-1994) and NHANES 2007-2014 and linked mortality datasets through 2019. Cox regression models assessed the association between ePWV categorized by quartile and all-cause/cause-specific mortality among individuals with MASLD.
RESULTS RESULTS
During the follow-up of a median of 26.3 years (interquartile range: 19.9-27.9), higher levels of ePWV among individuals with MASLD were associated with increased all-cause mortality, which remained significant after adjusting for demographic, lifestyle, clinical, and metabolic risk factors. Furthermore, higher ePWV in MASLD was associated with higher cardiovascular mortality. There was a 44% (hazard ratio: 1.44, 95% confidence interval: 1.32-1.58) increase in all-cause mortality and a 53% (hazard ratio: 1.53, 95% confidence interval: 1.32-1.77) increase in cardiovascular mortality for every 1 m/s increase in ePWV in MASLD. However, there was no significant association between ePWV and cancer-related mortality. Sensitivity analyses using the NHANES 2007-2014 dataset showed results identical to the original analysis.
CONCLUSION CONCLUSIONS
Higher ePWV in MASLD was associated with a higher risk of all-cause and cardiovascular mortality beyond traditional cardiovascular risk factors. Screening for ePWV in individuals with MASLD may be an effective and beneficial approach to reducing all-cause and cardiovascular mortality.

Identifiants

pubmed: 38740513
doi: 10.1111/jgh.16608
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024 Journal of Gastroenterology and Hepatology Foundation and John Wiley & Sons Australia, Ltd.

Références

Murag S, Ahmed A, Kim D. Recent epidemiology of nonalcoholic fatty liver disease. Gut Liver. 2021; 15: 206–216.
Li AA, Ahmed A, Kim D. Extrahepatic manifestations of nonalcoholic fatty liver disease. Gut Liver. 2020; 14: 168–178.
Mattace‐Raso FU, van der Cammen TJ, Hofman A et al. Arterial stiffness and risk of coronary heart disease and stroke: the Rotterdam Study. Circulation 2006; 113: 657–663.
Jaruvongvanich V, Chenbhanich J, Sanguankeo A, Rattanawong P, Wijarnpreecha K, Upala S. Increased arterial stiffness in nonalcoholic fatty liver disease: a systematic review and meta‐analysis. Eur. J. Gastroenterol. Hepatol. 2017; 29: e28–e35.
Chung GE, Choi SY, Kim D et al. Nonalcoholic fatty liver disease as a risk factor of arterial stiffness measured by the cardioankle vascular index. Medicine (Baltimore). 2015; 94: e654.
Kim D, Kim WR, Kim HJ, Therneau TM. Association between noninvasive fibrosis markers and mortality among adults with nonalcoholic fatty liver disease in the United States. Hepatology 2013; 57: 1357–1365.
Kim D, Konyn P, Sandhu KK, Dennis BB, Cheung AC, Ahmed A. Metabolic dysfunction‐associated fatty liver disease is associated with increased all‐cause mortality in the United States. J. Hepatol. 2021; 75: 1284–1291.
Konyn P, Ahmed A, Kim D. Causes and risk profiles of mortality among individuals with nonalcoholic fatty liver disease. Clin. Mol. Hepatol. 2023; 29: S43–S57.
Kim D, Ahmed A. Nonalcoholic fatty liver disease in early life and all‐cause and cause‐specific mortality. Hepatobiliary Surg Nutr. 2022; 11: 317–319.
Lee JH, Kim D, Kim HJ et al. Hepatic steatosis index: a simple screening tool reflecting nonalcoholic fatty liver disease. Dig. Liver Dis. 2010; 42: 503–508.
Reference Values for Arterial Stiffness C. Determinants of pulse wave velocity in healthy people and in the presence of cardiovascular risk factors: ‘establishing normal and reference values’. Eur. Heart J. 2010; 31: 2338–2350.
Greve SV, Blicher MK, Kruger R et al. Estimated carotid‐femoral pulse wave velocity has similar predictive value as measured carotid‐femoral pulse wave velocity. J. Hypertens. 2016; 34: 1279–1289.
Vlachopoulos C, Terentes‐Printzios D, Laurent S et al. Association of estimated pulse wave velocity with survival: a secondary analysis of SPRINT. JAMA Netw. Open 2019; 2: e1912831.
Kim D, Vazquez‐Montesino LM, Li AA, Cholankeril G, Ahmed A. Inadequate physical activity and sedentary behavior are independent predictors of nonalcoholic fatty liver disease. Hepatology 2020; 72: 1556–1568.
Rinella ME, Lazarus JV, Ratziu V et al. A multisociety Delphi consensus statement on new fatty liver disease nomenclature. J. Hepatol. 2023; 79: 1542–1556.
van Sloten TT, Schram MT, van den Hurk K et al. Local stiffness of the carotid and femoral artery is associated with incident cardiovascular events and all‐cause mortality: the Hoorn study. J. Am. Coll. Cardiol. 2014; 63: 1739–1747.
Heffernan KS, Stoner L, London AS, Augustine JA, Lefferts WK. Estimated pulse wave velocity as a measure of vascular aging. PLoS ONE 2023; 18: e0280896.
Jae SY, Heffernan KS, Park JB et al. Association between estimated pulse wave velocity and the risk of cardiovascular outcomes in men. Eur. J. Prev. Cardiol. 2021; 28: e25–e27.
Heffernan KS, Wilmoth JM, London AS. Estimated pulse wave velocity and all‐cause mortality: findings from the Health and Retirement Study. Innov. Aging 2022; 6: igac056.
Greve SV, Laurent S, Olsen MH. Estimated pulse wave velocity calculated from age and mean arterial blood pressure. Pulse (Basel). 2017; 4: 175–179.
Adeva‐Andany MM, Adeva‐Contreras L, Fernandez‐Fernandez C, Gonzalez‐Lucan M, Funcasta‐Calderon R. Elastic tissue disruption is a major pathogenic factor to human vascular disease. Mol. Biol. Rep. 2021; 48: 4865–4878.
Meffert PJ, Baumeister SE, Lerch MM, Mayerle J, Kratzer W, Volzke H. Development, external validation, and comparative assessment of a new diagnostic score for hepatic steatosis. Am. J. Gastroenterol. 2014; 109: 1404–1414.
Hajifathalian K, Torabi Sagvand B, McCullough AJ. Effect of alcohol consumption on survival in nonalcoholic fatty liver disease: a national prospective cohort study. Hepatology 2019; 70: 511–521.

Auteurs

Donghee Kim (D)

Division of Gastroenterology and Hepatology, Stanford University School of Medicine, Stanford, California, USA.

Richie Manikat (R)

Division of Gastroenterology and Hepatology, Stanford University School of Medicine, Stanford, California, USA.

Karn Wijarnpreecha (K)

Department of Medicine, Division of Gastroenterology and Hepatology, University of Arizona College of Medicine, Phoenix, Arizona, USA.
Department of Internal Medicine, Division of Gastroenterology and Hepatology, Banner University Medical Center, Phoenix, Arizona, USA.

George Cholankeril (G)

Liver Center, Division of Abdominal Transplantation, Michael E DeBakey Department of General Surgery, Baylor College of Medicine, Houston, Texas, USA.
Section of Gastroenterology and Hepatology, Department of Medicine, Baylor College of Medicine, Houston, Texas, USA.

Aijaz Ahmed (A)

Division of Gastroenterology and Hepatology, Stanford University School of Medicine, Stanford, California, USA.

Classifications MeSH