The role of donor type and pre-transplant immunosuppression on outcomes of hematopoietic stem cell transplantation in children and young adults with severe aplastic anemia.


Journal

Pediatric transplantation
ISSN: 1399-3046
Titre abrégé: Pediatr Transplant
Pays: Denmark
ID NLM: 9802574

Informations de publication

Date de publication:
Jun 2024
Historique:
revised: 08 03 2024
received: 04 12 2023
accepted: 29 04 2024
medline: 20 5 2024
pubmed: 20 5 2024
entrez: 20 5 2024
Statut: ppublish

Résumé

The goal of this study was to assess the effect of donor type and pre-transplant immunotherapy (IST) on outcomes of hematopoietic stem cell transplantation (HSCT) for children and young adults with severe aplastic anemia (SAA). This retrospective, multi-center study included 52 SAA patients, treated in 5 pediatric transplant programs in Florida, who received HSCT between 2010 and 2020 as the first- or second-line treatment. The median age at HSCT for all 52 patients was 15 years (range 1-25). The 3-year overall survival (OS) by donor type were as follows: 95% [95% CI 85.4-99] for matched related donors (MRD) (N = 24), 84% [95% CI 63.5-99] for haploidentical (N = 13), and 71% [95% CI 36-99] for matched unrelated donors (MUD) (N = 7). The 3-year OS was 81% [95% CI 69.7-99] for all patients, 90.5% [95% CI 79.5-99] for non-IST patients (N = 27), and 70% [95% CI 51-99] for IST patients (N = 24) (log-rank p = .04). Survival of haploidentical HSCT (haplo-HSCT) recipients with post-transplant cyclophosphamide (PTCy) (N = 13) was excellent for both groups: 100% for non-IST patients (N = 3) and 80% for IST patients (N = 10). The 3-year OS for patients with previous IST by donor type in groups where >5 patients were available was 78.8% [95% CI 52.3-99] for haplo-HSCT (N = 10) and 66.7% [95% CI 28.7-99] for MUD (N = 6). Although it appears that patients receiving HSCT ≥6 months after the start of IST had worse survival, the number of patients in each category was small and log-rank was not significant(p = .65). Patients receiving MUD and haplo-HSCT with PTCy had similar outcomes, suggesting that haplo-HSCT with PTCy could be included in randomized trials of upfront IST versus alternative donor HSCT.

Sections du résumé

BACKGROUND BACKGROUND
The goal of this study was to assess the effect of donor type and pre-transplant immunotherapy (IST) on outcomes of hematopoietic stem cell transplantation (HSCT) for children and young adults with severe aplastic anemia (SAA).
METHODS METHODS
This retrospective, multi-center study included 52 SAA patients, treated in 5 pediatric transplant programs in Florida, who received HSCT between 2010 and 2020 as the first- or second-line treatment.
RESULTS RESULTS
The median age at HSCT for all 52 patients was 15 years (range 1-25). The 3-year overall survival (OS) by donor type were as follows: 95% [95% CI 85.4-99] for matched related donors (MRD) (N = 24), 84% [95% CI 63.5-99] for haploidentical (N = 13), and 71% [95% CI 36-99] for matched unrelated donors (MUD) (N = 7). The 3-year OS was 81% [95% CI 69.7-99] for all patients, 90.5% [95% CI 79.5-99] for non-IST patients (N = 27), and 70% [95% CI 51-99] for IST patients (N = 24) (log-rank p = .04). Survival of haploidentical HSCT (haplo-HSCT) recipients with post-transplant cyclophosphamide (PTCy) (N = 13) was excellent for both groups: 100% for non-IST patients (N = 3) and 80% for IST patients (N = 10). The 3-year OS for patients with previous IST by donor type in groups where >5 patients were available was 78.8% [95% CI 52.3-99] for haplo-HSCT (N = 10) and 66.7% [95% CI 28.7-99] for MUD (N = 6). Although it appears that patients receiving HSCT ≥6 months after the start of IST had worse survival, the number of patients in each category was small and log-rank was not significant(p = .65).
CONCLUSIONS CONCLUSIONS
Patients receiving MUD and haplo-HSCT with PTCy had similar outcomes, suggesting that haplo-HSCT with PTCy could be included in randomized trials of upfront IST versus alternative donor HSCT.

Identifiants

pubmed: 38766976
doi: 10.1111/petr.14784
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

e14784

Informations de copyright

© 2024 Wiley Periodicals LLC.

Références

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Auteurs

Reema Kashif (R)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Biljana Horn (B)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Jordan Milner (J)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Michael Joyce (M)

Nemours Children's Health & Wolfson Children's Hospital, Jacksonville, Florida, USA.

Mansi Dalal (M)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Jin-Ju Lee (JJ)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Kevin McNerney (K)

Center for Cell & Gene Therapy for Non-Malignant Conditions, John Hopkins All Children's Hospital, St. Petersburg, Florida, USA.

Jessica Cline (J)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

John Fort (J)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Paul Castillo (P)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Jorge Galvez (J)

Pediatric Blood & Marrow Transplantation Program, Nicklaus Children's Hospital, Miami, Florida, USA.

Warren Alperstein (W)

School of Medicine, University of Miami, Miami, Florida, USA.

John Ligon (J)

Department of Pediatrics, Division of Pediatric Hematology-Oncology & BMT, University of Florida, Gainesville, Florida, USA.

Edward Ziga (E)

School of Medicine, University of Miami, Miami, Florida, USA.

David Crawford (D)

School of Medicine, University of Miami, Miami, Florida, USA.

Deepak Chellapandian (D)

Center for Cell & Gene Therapy for Non-Malignant Conditions, John Hopkins All Children's Hospital, St. Petersburg, Florida, USA.

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