Sex-specific developmental gene expression atlas unveils dimorphic gene networks in C. elegans.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
20 May 2024
Historique:
received: 23 07 2023
accepted: 24 04 2024
medline: 21 5 2024
pubmed: 21 5 2024
entrez: 20 5 2024
Statut: epublish

Résumé

Sex-specific traits and behaviors emerge during development by the acquisition of unique properties in the nervous system of each sex. However, the genetic events responsible for introducing these sex-specific features remain poorly understood. In this study, we create a comprehensive gene expression atlas of pure populations of hermaphrodites and males of the nematode Caenorhabditis elegans across development. We discover numerous differentially expressed genes, including neuronal gene families like transcription factors, neuropeptides, and G protein-coupled receptors. We identify INS-39, an insulin-like peptide, as a prominent male-biased gene expressed specifically in ciliated sensory neurons. We show that INS-39 serves as an early-stage male marker, facilitating the effective isolation of males in high-throughput experiments. Through complex and sex-specific regulation, ins-39 plays pleiotropic sexually dimorphic roles in various behaviors, while also playing a shared, dimorphic role in early life stress. This study offers a comparative sexual and developmental gene expression database for C. elegans. Furthermore, it highlights conserved genes that may underlie the sexually dimorphic manifestation of different human diseases.

Identifiants

pubmed: 38769103
doi: 10.1038/s41467-024-48369-z
pii: 10.1038/s41467-024-48369-z
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

4273

Subventions

Organisme : EC | EU Framework Programme for Research and Innovation H2020 | H2020 Priority Excellent Science | H2020 European Research Council (H2020 Excellent Science - European Research Council)
ID : ERC-2019-STG 850784
Organisme : Israel Science Foundation (ISF)
ID : 961/21

Informations de copyright

© 2024. The Author(s).

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Auteurs

Rizwanul Haque (R)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel.
Department of Molecular Neuroscience, Weizmann Institute of Science, Rehovot, Israel.

Sonu Peedikayil Kurien (SP)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel.
Department of Molecular Neuroscience, Weizmann Institute of Science, Rehovot, Israel.

Hagar Setty (H)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel.
Department of Molecular Neuroscience, Weizmann Institute of Science, Rehovot, Israel.

Yehuda Salzberg (Y)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel.
Department of Molecular Neuroscience, Weizmann Institute of Science, Rehovot, Israel.

Gil Stelzer (G)

Department of Life Sciences Core Facilities, Weizmann Institute of Science, Rehovot, Israel.

Einav Litvak (E)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel.

Hila Gingold (H)

Department of Neurobiology, Wise Faculty of Life Sciences & Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel.

Oded Rechavi (O)

Department of Neurobiology, Wise Faculty of Life Sciences & Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel.

Meital Oren-Suissa (M)

Department of Brain Sciences, Weizmann Institute of Science, Rehovot, Israel. meital.oren@weizmann.ac.il.
Department of Molecular Neuroscience, Weizmann Institute of Science, Rehovot, Israel. meital.oren@weizmann.ac.il.

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