The characterization of functional conduction block in patients with multiple types of atrial tachycardia- A discussion on the mechanism of multiple atrial tachycardia.

Atrial tachycardias Catheter ablation Functional conduction block Reentry Scar

Journal

Journal of interventional cardiac electrophysiology : an international journal of arrhythmias and pacing
ISSN: 1572-8595
Titre abrégé: J Interv Card Electrophysiol
Pays: Netherlands
ID NLM: 9708966

Informations de publication

Date de publication:
21 May 2024
Historique:
received: 05 12 2023
accepted: 24 04 2024
medline: 21 5 2024
pubmed: 21 5 2024
entrez: 20 5 2024
Statut: aheadofprint

Résumé

High-resolution mapping offers superior accuracy in delineating conduction features; however, certain characteristics are still linked to elevated recurrence rates of atrial tachycardia (AT), suggesting the influence of additional mechanisms. This study systematically assessed the substrate of functional conduction block (FCB) regions in relation to the mechanisms of multiple ATs. In this study, the Carto system facilitated the mapping of ATs in 13 patients undergoing ablation, each presenting with more than two AT variants. FCB regions were marked and further analyzed. A total of 33 sustained ATs were mapped across the patient cohort. FCB regions showed convertibility in 7 of 13 patients (54%). Three kinds of presentations can be summarized by the FCB region: Firstly, the FCB region could act as the main obstacle sustaining the localized reentrant pathway, for which rounding obviously has a direct correlation with the mechanism of the AT (27%). Secondly, the FCB regions could act as obstacle lines to reorganize the propagation of the reentry in localized AT and macroreentrant AT (55%). Lastly, the FCB region could act as a bystander and may not be related to the mechanism of the ATs (18%). The potentials in FCB regions mostly performed low voltages or fragmented potentials (FPs) in the ATs which they did not perform the conduction block (90%). In multiple ATs, FCB regions may not be uncommon. The participation of FCB regions in the mechanism of ATs showed three different kinds of performance. The dynamic nature of this substrate may provide insight into the reasons for the high recurrence of related ATs.

Sections du résumé

BACKGROUND BACKGROUND
High-resolution mapping offers superior accuracy in delineating conduction features; however, certain characteristics are still linked to elevated recurrence rates of atrial tachycardia (AT), suggesting the influence of additional mechanisms. This study systematically assessed the substrate of functional conduction block (FCB) regions in relation to the mechanisms of multiple ATs.
METHODS METHODS
In this study, the Carto system facilitated the mapping of ATs in 13 patients undergoing ablation, each presenting with more than two AT variants. FCB regions were marked and further analyzed.
RESULTS RESULTS
A total of 33 sustained ATs were mapped across the patient cohort. FCB regions showed convertibility in 7 of 13 patients (54%). Three kinds of presentations can be summarized by the FCB region: Firstly, the FCB region could act as the main obstacle sustaining the localized reentrant pathway, for which rounding obviously has a direct correlation with the mechanism of the AT (27%). Secondly, the FCB regions could act as obstacle lines to reorganize the propagation of the reentry in localized AT and macroreentrant AT (55%). Lastly, the FCB region could act as a bystander and may not be related to the mechanism of the ATs (18%). The potentials in FCB regions mostly performed low voltages or fragmented potentials (FPs) in the ATs which they did not perform the conduction block (90%).
CONCLUSION CONCLUSIONS
In multiple ATs, FCB regions may not be uncommon. The participation of FCB regions in the mechanism of ATs showed three different kinds of performance. The dynamic nature of this substrate may provide insight into the reasons for the high recurrence of related ATs.

Identifiants

pubmed: 38769195
doi: 10.1007/s10840-024-01817-8
pii: 10.1007/s10840-024-01817-8
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Natural Science Foundation of Heilongjiang Province of China
ID : ZD2023H005

Informations de copyright

© 2024. The Author(s).

Références

Derval N, Takigawa M, Frontera A, Mahida S, Konstantinos V, Denis A, et al. Characterization of Complex Atrial Tachycardia in Patients With Previous Atrial Interventions Using High-Resolution Mapping. JACC: Clin Electrophysiol. 2020;6(7):815–26.
pubmed: 32703564
Frontera A, Mahajan R, Dallet C, Vlachos K, Kitamura T, Takigawa M, et al. Characterizing localized reentry with high-resolution mapping: evidence for multiple slow conducting isthmuses within the circuit. Heart Rhythm. 2019;16(5):679–85.
doi: 10.1016/j.hrthm.2018.11.027 pubmed: 30500614
Frontera A, Takigawa M, Martin R, Thompson N, Cheniti G, Massoullié G, et al. Electrogram signature of specific activation patterns: analysis of atrial tachycardias at high-density endocardial mapping. Heart Rhythm. 2018;15(1):28–37.
doi: 10.1016/j.hrthm.2017.08.001 pubmed: 28797676
Laţcu DG, Bun S-S, Viera F, Delassi T, El Jamili M, Al Amoura A, et al. Selection of critical isthmus in scar-related atrial tachycardia using a new automated ultrahigh resolution mapping system. Circ: Arrhythmia and Electrophysiology. 2017;10(1):e004510.
pubmed: 28039280
Ciaccio EJ, Coromilas J, Wit AL, Peters NS, Garan H. Source-Sink Mismatch Causing Functional Conduction Block in Re-Entrant Ventricular Tachycardia. JACC Clin Electrophysiol. 2018;4(1):1–16.
doi: 10.1016/j.jacep.2017.08.019 pubmed: 29600773 pmcid: 5874259
Takigawa M, Derval N, Maury P, Martin R, Denis A, Miyazaki S, et al. Comprehensive multicenter study of the common isthmus in post–atrial fibrillation ablation multiple-loop atrial tachycardia. Circ Arrhythm Electrophysiol. 2018;11(6):e006019.
doi: 10.1161/CIRCEP.117.006019 pubmed: 29769223
Miyazaki S, Hasegawa K, Kaseno K, Tada H. Protected channels can be formed by a functional line of block in human atrial tachycardia. Heart Rhythm. 2019;16(4):642–3.
doi: 10.1016/j.hrthm.2018.10.023 pubmed: 30366157
Vlachos K, Denis A, Takigawa M, Kitamura T, Martin CA, Frontera A, et al. The role of Marshall bundle epicardial connections in atrial tachycardias after atrial fibrillation ablation. Heart Rhythm. 2019;16(9):1341–7.
doi: 10.1016/j.hrthm.2019.05.019 pubmed: 31125669
Takigawa M, Derval N, Martin CA, Vlachos K, Denis A, Nakatani Y, et al. Mechanism of Recurrence of Atrial Tachycardia: Comparison Between First Versus Redo Procedures in a High-Resolution Mapping System. Circ Arrhythm Electrophysiol. 2020;13(1):e007273.
doi: 10.1161/CIRCEP.119.007273 pubmed: 31937120
Zhang J, Zheng L, Zhou D, Zhao A, Tang C, Zhang Y, et al. Insight into the mechanism of macroreentrant atrial tachycardia with cycle length alternans using ultrahigh density mapping system. Circ: Arrhythmia and Electrophysiology. 2019;12(11):e007634.
pubmed: 31698935
Zhou D, Hu W, Yang G, Chen H, Zhang B, Han J, et al. A postsurgery atrial tachycardia with alternating cycle length: The possible circuits revealed by high-resolution mapping. HeartRhythm Case Rep. 2020;6(6):297–9.
doi: 10.1016/j.hrcr.2019.05.005 pubmed: 32577380
Takigawa M, Denis A, Vlachos K, Martin CA, Jais P, Derval N. Two consecutive ATs demonstrating a centrifugal pattern; What is the mechanism? J Cardiovasc Electrophysiol. 2019;30(6):978–80.
doi: 10.1111/jce.13883 pubmed: 30801813

Auteurs

Bin Zhu (B)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

GuoHua Zhang (G)

Department of Cardiology, The Second Hospital of Harbin, Harbin, China.

SongCai Xie (S)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

Ying Luan (Y)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

Wei Cao (W)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

Jian Xu (J)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

Shuo Zhang (S)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.

JinWei Tian (J)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China.
Heilongjiang Provincial Key Laboratory of Panvascular Disease, Harbin, China.

Fan Wang (F)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China. wangfanrock@163.com.
Heilongjiang Provincial Key Laboratory of Panvascular Disease, Harbin, China. wangfanrock@163.com.

ShuFeng Li (S)

Department of Cardiology, 2, Affiliated Hospital of Harbin Medical University, Harbin, China. drlishufeng2@163.com.

Classifications MeSH