Scutellaria Root extract-induced hepatocytotoxicity can be controlled by regulating its baicalin content.

Scutellaria baicalensis Baicalin Hepatocytotoxicity Scutellaria Root

Journal

Journal of natural medicines
ISSN: 1861-0293
Titre abrégé: J Nat Med
Pays: Japan
ID NLM: 101518405

Informations de publication

Date de publication:
24 May 2024
Historique:
received: 29 09 2023
accepted: 23 03 2024
medline: 24 5 2024
pubmed: 24 5 2024
entrez: 24 5 2024
Statut: aheadofprint

Résumé

Scuellaria Root (SR, root of Scutellaria baicalensis), which has potent anti-inflammatory effects, is a component of useful Kampo formulae. Albeit a low frequency, SR induces serious interstitial pneumonia and liver dysfunction. In this study, to control the adverse effects of SR, we investigated the causal constituent responsible for its hepatocytotoxicity and aimed to develop a method to control it. As a result, we revealed that the hepatocytotoxicity of SR was correlated with its baicalin content, a major constituent in SR. It was confirmed by preparing a baicalin-free SR extract, which exhibited reduced hepatocytotoxicity. The addition of baicalin to the baicalin-free SR extract restored the hepatocytotoxicity, indicating that the hepatocytotoxicity of SR is dependent on its baicalin content. Thus, SR extract-induced hepatocytotoxicity can be controlled by regulating its baicalin content.

Identifiants

pubmed: 38787459
doi: 10.1007/s11418-024-01814-1
pii: 10.1007/s11418-024-01814-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Japan Society for the Promotion of Science
ID : 22K06676

Informations de copyright

© 2024. The Author(s) under exclusive licence to The Japanese Society of Pharmacognosy.

Références

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Auteurs

Naohiro Oshima (N)

Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan. n-oshima@nihs.go.jp.
Division of Environmental Chemistry, National Institute of Health Sciences, 3-25-26 Tonomachi, Kawasaki-Ku, Kawasaki, Kanagawa, 210-9501, Japan. n-oshima@nihs.go.jp.

Kosuke Kusamori (K)

Laboratory of Biopharmaceutics, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Ryo Takasaki (R)

Laboratory of Biopharmaceutics, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Moe Takeda (M)

Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Yuri Katsurada (Y)

Laboratory of Biopharmaceutics, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Takumi Nose (T)

Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Kazuki Okoshi (K)

Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan.

Makiya Nishikawa (M)

Laboratory of Biopharmaceutics, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan. makiya@rs.tus.ac.jp.

Noriyasu Hada (N)

Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda City, Chiba, 278-8510, Japan. hada@rs.tus.ac.jp.

Classifications MeSH