Identification of non-adherence to adjuvant letrozole using a population pharmacokinetics approach in hormone receptor-positive breast cancer patients.

CYP2A6 CYP3A4/5 adherence letrozole non-linear mixed-effects modelling pharmacokinetics

Journal

European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
ISSN: 1879-0720
Titre abrégé: Eur J Pharm Sci
Pays: Netherlands
ID NLM: 9317982

Informations de publication

Date de publication:
22 May 2024
Historique:
received: 27 11 2023
revised: 05 04 2024
accepted: 21 05 2024
medline: 25 5 2024
pubmed: 25 5 2024
entrez: 24 5 2024
Statut: aheadofprint

Résumé

Letrozole, an aromatase inhibitor metabolised via CYP2A6 and CYP3A4/5 enzymes, is used as adjuvant therapy for women with hormone receptor (HR)-positive early breast cancer. The objective of this study was to quantify the impact of CYP2A6 genotype on letrozole pharmacokinetics (PK), to identify non-adherent patients using a population approach and explore the possibility of a relationship between non-adherence and early relapse. Breast cancer patients enrolled in the prospective PHACS study (ClinicalTrials.gov NCT01127295) and treated with adjuvant letrozole 2.5 mg/day were included. Trough letrozole concentrations (C 617 patients contributing 2534 plasma concentrations were included and led to a one-compartment PK model with linear absorption and elimination. Model-based methods identified 28% of patients as non-adherent based on high fluctuations of their C These findings will help future investigations focusing on the exposure-efficacy relationship for letrozole in adjuvant setting.

Sections du résumé

BACKGROUND BACKGROUND
Letrozole, an aromatase inhibitor metabolised via CYP2A6 and CYP3A4/5 enzymes, is used as adjuvant therapy for women with hormone receptor (HR)-positive early breast cancer. The objective of this study was to quantify the impact of CYP2A6 genotype on letrozole pharmacokinetics (PK), to identify non-adherent patients using a population approach and explore the possibility of a relationship between non-adherence and early relapse.
METHODS METHODS
Breast cancer patients enrolled in the prospective PHACS study (ClinicalTrials.gov NCT01127295) and treated with adjuvant letrozole 2.5 mg/day were included. Trough letrozole concentrations (C
RESULTS RESULTS
617 patients contributing 2534 plasma concentrations were included and led to a one-compartment PK model with linear absorption and elimination. Model-based methods identified 28% of patients as non-adherent based on high fluctuations of their C
CONCLUSIONS CONCLUSIONS
These findings will help future investigations focusing on the exposure-efficacy relationship for letrozole in adjuvant setting.

Identifiants

pubmed: 38788907
pii: S0928-0987(24)00121-0
doi: 10.1016/j.ejps.2024.106809
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT01127295']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

106809

Informations de copyright

Copyright © 2024. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare no competing interests to declare that are relevant to the content of this article.

Auteurs

Alicja Puszkiel (A)

Cancer Research Center of Toulouse (CRCT), Inserm U1037, Université Paul Sabatier, Toulouse, France.

Florence Dalenc (F)

Institut Claudius Regaud, Institut Universitaire du Cancer de Toulouse - Oncopole, Toulouse, France.

Naïma Tafzi (N)

Department of Pharmacology, Toxicology and Pharmacovigilance, CHU Limoges, Limoges, France.

Pierre Marquet (P)

Department of Pharmacology, Toxicology and Pharmacovigilance, CHU Limoges, Limoges, France.

Marc Debled (M)

Department of Medical Oncology, Institut Bergonié, Bordeaux, France.

William Jacot (W)

Institut du Cancer de Montpellier, ICM, Université de Montpellier, IRCM, Inserm U1194, Montpellier, France.

Laurence Venat-Bouvet (L)

Department of Medical Oncology, CHU Limoges, Limoges, France.

Catherine Ferrer (C)

Department of Medical Oncology, CHU Nîmes-Carémeau, Nîmes, France.

Nadia Levasseur (N)

Department of Medical Oncology, CH Cahors, Cahors, France.

Rodolphe Paulon (R)

Department of Medical Oncology, Centre Hospitalier Intercommunal Castres-Mazamet, Castres, France.

Jérôme Dauba (J)

Department of Medical Oncology, CH Mont de Marsan, Mont-de-Marsan, France.

Alexandre Evrard (A)

Institut du Cancer de Montpellier, ICM, Université de Montpellier, IRCM, Inserm U1194, Montpellier, France; Laboratoire de Biochimie et Biologie Moléculaire, CHU Nîmes-Carémeau, Nîmes, France.

Vincent Mauriès (V)

Institut Claudius Regaud, Institut Universitaire du Cancer de Toulouse - Oncopole, Toulouse, France.

Thomas Filleron (T)

Institut Claudius Regaud, Institut Universitaire du Cancer de Toulouse - Oncopole, Toulouse, France.

Etienne Chatelut (E)

Cancer Research Center of Toulouse (CRCT), Inserm U1037, Université Paul Sabatier, Toulouse, France; Institut Claudius Regaud, Institut Universitaire du Cancer de Toulouse - Oncopole, Toulouse, France.

Fabienne Thomas (F)

Cancer Research Center of Toulouse (CRCT), Inserm U1037, Université Paul Sabatier, Toulouse, France; Institut Claudius Regaud, Institut Universitaire du Cancer de Toulouse - Oncopole, Toulouse, France.

Melanie White-Koning (M)

Cancer Research Center of Toulouse (CRCT), Inserm U1037, Université Paul Sabatier, Toulouse, France. Electronic address: melanie.white-koning@univ-tlse3.fr.

Classifications MeSH