The Role of microRNA in the Regulation of Cortisol Metabolism in the Adipose Tissue in the Course of Obesity.
Humans
Hydrocortisone
/ metabolism
MicroRNAs
/ genetics
Obesity
/ metabolism
Male
Female
Middle Aged
Adult
Receptors, Glucocorticoid
/ metabolism
11-beta-Hydroxysteroid Dehydrogenase Type 1
/ metabolism
11-beta-Hydroxysteroid Dehydrogenase Type 2
/ metabolism
Adipose Tissue
/ metabolism
Intra-Abdominal Fat
/ metabolism
Gene Expression Regulation
RNA, Messenger
/ metabolism
Carbohydrate Dehydrogenases
adipose tissue
cortisol metabolism
glucocorticoid receptor alpha
metabolic inflammation
microRNA
obesity
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
07 May 2024
07 May 2024
Historique:
received:
31
03
2024
revised:
30
04
2024
accepted:
04
05
2024
medline:
25
5
2024
pubmed:
25
5
2024
entrez:
25
5
2024
Statut:
epublish
Résumé
The similarity of the clinical picture of metabolic syndrome and hypercortisolemia supports the hypothesis that obesity may be associated with impaired expression of genes related to cortisol action and metabolism in adipose tissue. The expression of genes encoding the glucocorticoid receptor alpha (
Identifiants
pubmed: 38791098
pii: ijms25105058
doi: 10.3390/ijms25105058
pii:
doi:
Substances chimiques
Hydrocortisone
WI4X0X7BPJ
MicroRNAs
0
Receptors, Glucocorticoid
0
11-beta-Hydroxysteroid Dehydrogenase Type 1
EC 1.1.1.146
11-beta-Hydroxysteroid Dehydrogenase Type 2
EC 1.1.1.146
HSD11B1 protein, human
EC 1.1.1.146
HSD11B2 protein, human
EC 1.1.1.146
galactose-6-phosphate dehydrogenase
EC 1.1.1.-
glucocorticoid receptor alpha
0
RNA, Messenger
0
Carbohydrate Dehydrogenases
EC 1.1.-
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : National Science Centre Poland
ID : 2018/31/B/NZ5/01556