Sacubitril/valsartan has an underestimated impact on the right ventricle in patients with sleep-disordered breathing, especially central sleep apnoea syndrome.

Heart failure Nocturnal ventilatory polygraphy Right ventricular function Sacubitril/valsartan Sleep apnoea syndrome

Journal

Archives of cardiovascular diseases
ISSN: 1875-2128
Titre abrégé: Arch Cardiovasc Dis
Pays: Netherlands
ID NLM: 101465655

Informations de publication

Date de publication:
18 May 2024
Historique:
received: 07 12 2023
revised: 07 04 2024
accepted: 10 04 2024
medline: 30 5 2024
pubmed: 30 5 2024
entrez: 29 5 2024
Statut: aheadofprint

Résumé

Sacubitril/valsartan has been demonstrated to significantly improve left ventricular performance and remodelling in patients with heart failure. However, its effects on the right ventricle in patients with chronic heart failure and sleep-disordered breathing (SDB) have not been studied. To investigate the impact of sacubitril/valsartan treatment on right ventricular function in patients with SDB. This was a subanalysis of an observational prospective multicentre study involving 101 patients. At inclusion, patients were evaluated by echocardiography and nocturnal ventilatory polygraphy, which allowed patients to be divided into three groups: "central-SDB"; "obstructive-SDB"; and "no-SDB". After 3 months of sacubitril/valsartan therapy, a positive impact on right ventricular function was observed. In the general population, tricuspid annular plane systolic excursion increased by +1.32±4.74mm (P=0.024) and systolic pulmonary artery pressure decreased by -3.1±10.91mmHg (P=0.048). The central-SDB group experienced the greatest echocardiographic improvement, with a significant increase in tricuspid annular plane systolic excursion of +2.1±4.9mm (P=0.045) and a significant reduction in systolic pulmonary artery pressure of -8.4±9.7mmHg (P=0.001). Sacubitril/valsartan improved right ventricular function in patients with heart failure and SDB after only 3 months of treatment. The greatest improvement in right ventricular function was observed in the central-SDB group.

Sections du résumé

BACKGROUND BACKGROUND
Sacubitril/valsartan has been demonstrated to significantly improve left ventricular performance and remodelling in patients with heart failure. However, its effects on the right ventricle in patients with chronic heart failure and sleep-disordered breathing (SDB) have not been studied.
AIM OBJECTIVE
To investigate the impact of sacubitril/valsartan treatment on right ventricular function in patients with SDB.
METHODS METHODS
This was a subanalysis of an observational prospective multicentre study involving 101 patients. At inclusion, patients were evaluated by echocardiography and nocturnal ventilatory polygraphy, which allowed patients to be divided into three groups: "central-SDB"; "obstructive-SDB"; and "no-SDB".
RESULTS RESULTS
After 3 months of sacubitril/valsartan therapy, a positive impact on right ventricular function was observed. In the general population, tricuspid annular plane systolic excursion increased by +1.32±4.74mm (P=0.024) and systolic pulmonary artery pressure decreased by -3.1±10.91mmHg (P=0.048). The central-SDB group experienced the greatest echocardiographic improvement, with a significant increase in tricuspid annular plane systolic excursion of +2.1±4.9mm (P=0.045) and a significant reduction in systolic pulmonary artery pressure of -8.4±9.7mmHg (P=0.001).
CONCLUSIONS CONCLUSIONS
Sacubitril/valsartan improved right ventricular function in patients with heart failure and SDB after only 3 months of treatment. The greatest improvement in right ventricular function was observed in the central-SDB group.

Identifiants

pubmed: 38811278
pii: S1875-2136(24)00187-6
doi: 10.1016/j.acvd.2024.04.003
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 The Authors. Published by Elsevier Masson SAS.. All rights reserved.

Auteurs

Laura Sofia Cardelli (LS)

Cardiology Department, Versilia Hospital, 55041 Camaiore, Italy.

Mariarosaria Magaldi (M)

Department of Advanced Biomedical Science, Federico II University of Naples, 80131 Napoli, Italy.

Audrey Agullo (A)

Cardiology Department, Arnaud-De-Villeneuve Hospital, 34090 Montpellier, France.

Gaetan Richard (G)

Cardiology Department, Arnaud-De-Villeneuve Hospital, 34090 Montpellier, France.

Erika Nogue (E)

Inserm, IDESP, PreMedical INRIA, CHU de Montpellier, Montpellier University, 34295 Montpellier, France.

Philippe Berdague (P)

Department of Cardiology, Béziers Hospital, 34500 Béziers, France.

Michel Galiner (M)

Cardiology Department, Toulouse-Rangueil University Hospital, 31400 Toulouse, France; MMP Department, Faculty of Health, Toulouse III Paul-Sabatier University, 31062 Toulouse, France.

Frédéric Georger (F)

Department of Cardiology, Béziers Hospital, 34500 Béziers, France.

François Picard (F)

Heart Failure Unit, Haut-Lévêque Hospital, 33604 Pessac, France.

Elvira Prunet (E)

Cardiology Department, Montpellier University, Nîmes University Hospital, 30900 Nîmes, France.

Nicolas Molinari (N)

Inserm, IDESP, PreMedical INRIA, CHU de Montpellier, Montpellier University, 34295 Montpellier, France.

Arnaud Bourdin (A)

Inserm U1046, PhyMedExp, Department of Respiratory Diseases, CNRS, UMR 9214, CHU de Montpellier, University of Montpellier, 34295 Montpellier, France.

Dany Jaffuel (D)

Inserm U1046, PhyMedExp, Department of Respiratory Diseases, CNRS, UMR 9214, CHU de Montpellier, University of Montpellier, 34295 Montpellier, France.

François Roubille (F)

Cardiology Department, Arnaud-De-Villeneuve Hospital, 34090 Montpellier, France. Electronic address: f-roubille@chu-montpellier.fr.

Classifications MeSH