Role of Phenylethanolamine-N-methyltransferase on Nicotine-Induced Vasodilation in Rat Cerebral Arteries.
Axo–axonal interaction
basilar artery
epinephrine
norepinephrine
phenylethanolamine‐N‐methyltransferase
Journal
Microcirculation (New York, N.Y. : 1994)
ISSN: 1549-8719
Titre abrégé: Microcirculation
Pays: United States
ID NLM: 9434935
Informations de publication
Date de publication:
04 Jun 2024
04 Jun 2024
Historique:
revised:
28
07
2023
received:
02
02
2022
accepted:
22
04
2024
medline:
5
6
2024
pubmed:
5
6
2024
entrez:
5
6
2024
Statut:
aheadofprint
Résumé
The sympathetic-parasympathetic (or axo-axonal) interaction mechanism mediated that neurogenic relaxation, which was dependent on norepinephrine (NE) releases from sympathetic nerve terminal and acts on β Blood vessel myography technique was used to measure relaxation and contraction responses of isolated basilar arterial rings of rats. Nicotine-induced relaxation was sensitive to propranolol, guanethidine (an adrenergic neuronal blocker), and N These results suggest that NE may be methylated by PNMT to form epinephrine and cause the release of NO and vasodilation. These results provide further evidence supporting the physiological significance of the axo-axonal interaction mechanism in regulating brainstem vascular tone.
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
e12858Subventions
Organisme : Taipei Tzu Chi Hospital
Organisme : Buddhist Tzu Chi Medical Foundation
ID : TCRD-TPE-108-21
Organisme : Buddhist Tzu Chi Medical Foundation
ID : TCRD-TPE-109-RT-3
Organisme : Buddhist Tzu Chi Medical Foundation
ID : TCRD-TPE-110-05
Organisme : Buddhist Tzu Chi Medical Foundation
ID : TCRD-TPE-110-12
Informations de copyright
© 2024 John Wiley & Sons Ltd.
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