Plasma taurine level is linked to symptom burden and clinical outcomes in post-COVID condition.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2024
Historique:
received: 26 01 2024
accepted: 14 05 2024
medline: 5 6 2024
pubmed: 5 6 2024
entrez: 5 6 2024
Statut: epublish

Résumé

A subset of individuals (10-20%) experience post-COVID condition (PCC) subsequent to initial SARS-CoV-2 infection, which lacks effective treatment. PCC carries a substantial global burden associated with negative economic and health impacts. This study aims to evaluate the association between plasma taurine levels with self-reported symptoms and adverse clinical outcomes in patients with PCC. We analyzed the plasma proteome and metabolome of 117 individuals during their acute COVID-19 hospitalization and at the convalescence phase six-month post infection. Findings were compared with 28 age and sex-matched healthy controls. Plasma taurine levels were negatively associated with PCC symptoms and correlated with markers of inflammation, tryptophan metabolism, and gut dysbiosis. Stratifying patients based on the trajectories of plasma taurine levels during six-month follow-up revealed a significant association with adverse clinical events. Increase in taurine levels during the transition to convalescence were associated with a reduction in adverse events independent of comorbidities and acute COVID-19 severity. In a multivariate analysis, increased plasma taurine level between acute and convalescence phase was associated with marked protection from adverse clinical events with a hazard ratio of 0.13 (95% CI: 0.05-0.35; p<0.001). Taurine emerges as a promising predictive biomarker and potential therapeutic target in PCC. Taurine supplementation has already demonstrated clinical benefits in various diseases and warrants exploration in large-scale clinical trials for alleviating PCC.

Sections du résumé

BACKGROUND BACKGROUND
A subset of individuals (10-20%) experience post-COVID condition (PCC) subsequent to initial SARS-CoV-2 infection, which lacks effective treatment. PCC carries a substantial global burden associated with negative economic and health impacts. This study aims to evaluate the association between plasma taurine levels with self-reported symptoms and adverse clinical outcomes in patients with PCC.
METHODS AND FINDINGS RESULTS
We analyzed the plasma proteome and metabolome of 117 individuals during their acute COVID-19 hospitalization and at the convalescence phase six-month post infection. Findings were compared with 28 age and sex-matched healthy controls. Plasma taurine levels were negatively associated with PCC symptoms and correlated with markers of inflammation, tryptophan metabolism, and gut dysbiosis. Stratifying patients based on the trajectories of plasma taurine levels during six-month follow-up revealed a significant association with adverse clinical events. Increase in taurine levels during the transition to convalescence were associated with a reduction in adverse events independent of comorbidities and acute COVID-19 severity. In a multivariate analysis, increased plasma taurine level between acute and convalescence phase was associated with marked protection from adverse clinical events with a hazard ratio of 0.13 (95% CI: 0.05-0.35; p<0.001).
CONCLUSIONS CONCLUSIONS
Taurine emerges as a promising predictive biomarker and potential therapeutic target in PCC. Taurine supplementation has already demonstrated clinical benefits in various diseases and warrants exploration in large-scale clinical trials for alleviating PCC.

Identifiants

pubmed: 38837993
doi: 10.1371/journal.pone.0304522
pii: PONE-D-24-02915
doi:

Substances chimiques

Taurine 1EQV5MLY3D
Biomarkers 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0304522

Informations de copyright

Copyright: © 2024 Khoramjoo et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

Auteurs

Mobin Khoramjoo (M)

Department of Physiology, University of Alberta, Edmonton, Alberta, Canada.
Mazankowski Alberta Heart Institute, University of Alberta, Edmonton, Alberta, Canada.

Kaiming Wang (K)

Mazankowski Alberta Heart Institute, University of Alberta, Edmonton, Alberta, Canada.
Division of Cardiology, Department of Medicine, University of Alberta, Edmonton, Alberta, Canada.

Karthik Srinivasan (K)

Department of Chemical and Materials Engineering, University of Alberta, Edmonton, Alberta, Canada.

Mahmoud Gheblawi (M)

Department of Physiology, University of Alberta, Edmonton, Alberta, Canada.
Mazankowski Alberta Heart Institute, University of Alberta, Edmonton, Alberta, Canada.

Rupasri Mandal (R)

The Metabolomics Innovation Center, University of Alberta, Edmonton, Alberta, Canada.

Simon Rousseau (S)

Department of Medicine, McGill University & The Research Institute of the McGill University Health Centre, Montreal, Canada.

David Wishart (D)

The Metabolomics Innovation Center, University of Alberta, Edmonton, Alberta, Canada.

Vinay Prasad (V)

Department of Chemical and Materials Engineering, University of Alberta, Edmonton, Alberta, Canada.

Lawrence Richer (L)

Department of Pediatrics, Faculty of Medicine & Dentistry, University of Alberta, Edmonton, Alberta, Canada.

Angela M Cheung (AM)

Department of Medicine, University Health Network, Toronto, Ontario, Canada.

Gavin Y Oudit (GY)

Mazankowski Alberta Heart Institute, University of Alberta, Edmonton, Alberta, Canada.
Division of Cardiology, Department of Medicine, University of Alberta, Edmonton, Alberta, Canada.

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Classifications MeSH