Efficacy of Systemic Treatments in Patients With Metastatic Lung Invasive Mucinous Adenocarcinoma.

5-fluorouracil Chemotherapy Immunotherapy Non-small cell lung cancer Targeted treatment

Journal

Clinical lung cancer
ISSN: 1938-0690
Titre abrégé: Clin Lung Cancer
Pays: United States
ID NLM: 100893225

Informations de publication

Date de publication:
21 May 2024
Historique:
received: 13 02 2024
revised: 04 05 2024
accepted: 14 05 2024
medline: 14 6 2024
pubmed: 14 6 2024
entrez: 13 6 2024
Statut: aheadofprint

Résumé

Invasive mucinous adenocarcinoma (IMA) is a rare histological subtype of lung invasive adenocarcinoma with unique clinical, radiological, histopathological, and genomic characteristics. There have been limited studies on the effectiveness of systemic therapy for lung IMA, with conflicting results reported. We retrospectively investigated the medical records of patients diagnosed with lung IMA. Patients who were ≥ 18 years of age and received at least 1 course of treatment for metastatic or locally advanced inoperable disease were included in the study. Archive records of 113 patients diagnosed with IMA were screened for the study. A total of 41 patients with lung IMA were included. The targetable mutation rate was 20.6% (in 6 of 29 patients). Most patients (83.1%) had received platinum-based chemotherapy as a first-line treatment. The objective response rate (ORR) was 25.7%, and median progression-free survival (PFS) and overall survival (OS) were 8.1 months (95% CI, 5.02-11.2) and 17.5 months (95% CI, 11.7-23.3 months), respectively, in the patients who received chemotherapy. The median PFS and ORR were 20.6 (95% CI, 18.9-66.5) and 66.6%, respectively, in epidermal growth factor receptor (EGFR) mutation-positive patients (n = 3) with relevant targeted therapy. Only 1 patient used oxaliplatin and capecitabine combination (XELOX) as chemotherapy in the second-line treatment and achieved a partial response (PR) at 7.2 months. Platinum-based chemotherapies moderately enhance IMA patients' survival rates. Anti-EGFR-targeted drugs are seen as potentially effective in patients with EGFR driver mutation positive. Large, prospective studies are needed to confirm our findings.

Sections du résumé

BACKGROUND BACKGROUND
Invasive mucinous adenocarcinoma (IMA) is a rare histological subtype of lung invasive adenocarcinoma with unique clinical, radiological, histopathological, and genomic characteristics. There have been limited studies on the effectiveness of systemic therapy for lung IMA, with conflicting results reported.
METHODS METHODS
We retrospectively investigated the medical records of patients diagnosed with lung IMA. Patients who were ≥ 18 years of age and received at least 1 course of treatment for metastatic or locally advanced inoperable disease were included in the study. Archive records of 113 patients diagnosed with IMA were screened for the study.
RESULTS RESULTS
A total of 41 patients with lung IMA were included. The targetable mutation rate was 20.6% (in 6 of 29 patients). Most patients (83.1%) had received platinum-based chemotherapy as a first-line treatment. The objective response rate (ORR) was 25.7%, and median progression-free survival (PFS) and overall survival (OS) were 8.1 months (95% CI, 5.02-11.2) and 17.5 months (95% CI, 11.7-23.3 months), respectively, in the patients who received chemotherapy. The median PFS and ORR were 20.6 (95% CI, 18.9-66.5) and 66.6%, respectively, in epidermal growth factor receptor (EGFR) mutation-positive patients (n = 3) with relevant targeted therapy. Only 1 patient used oxaliplatin and capecitabine combination (XELOX) as chemotherapy in the second-line treatment and achieved a partial response (PR) at 7.2 months.
CONCLUSION CONCLUSIONS
Platinum-based chemotherapies moderately enhance IMA patients' survival rates. Anti-EGFR-targeted drugs are seen as potentially effective in patients with EGFR driver mutation positive. Large, prospective studies are needed to confirm our findings.

Identifiants

pubmed: 38871540
pii: S1525-7304(24)00083-4
doi: 10.1016/j.cllc.2024.05.004
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Published by Elsevier Inc.

Déclaration de conflit d'intérêts

Disclosure Young Kwang Chae COI Research Grant: Abbvie, BMS, Biodesix, Freenome, Predicine Honoraria/Advisory Boards: Roche/Genentech, AstraZeneca, Foundation Medicine, Neogenomics, Guardant Health, Boehringher Ingelheim, Biodesix, Immuneoncia, Lilly Oncology, Merck, Takeda, Lunit, Jazz Pharmaceutical, Tempus, BMS, Regeneron, NeoImmunTech, Esai All other authors do not have competing interests.

Auteurs

Tarık Demir (T)

Northwestern University Feinberg School of Medicine, Chicago, IL. Electronic address: tarik.demir@nm.org.

Murat Araz (M)

Necmettin Erbakan University Faculty of Medicine, Konya, Turkey.

Carolyn Moloney (C)

Northwestern University Feinberg School of Medicine, Chicago, IL.

Engin Hendem (E)

Necmettin Erbakan University Faculty of Medicine, Konya, Turkey.

Mehmet Zahid Koçak (MZ)

Necmettin Erbakan University Faculty of Medicine, Konya, Turkey.

Mustafa Erman (M)

Hacettepe University Faculty of Medicine, Ankara, Turkey.

Onur Baş (O)

Hacettepe University Faculty of Medicine, Ankara, Turkey.

Osman Köstek (O)

Marmara University School of Medicine, Istanbul, Turkey.

Nadiye Sever (N)

Marmara University School of Medicine, Istanbul, Turkey.

Serdar Karakaya (S)

Ankara Ataturk Sanatoryum Education and Training Hospital, Ankara, Turkey.

Esra Zeynelgil (E)

Ankara Ataturk Sanatoryum Education and Training Hospital, Ankara, Turkey.

Liam Il-Young Chung (LI)

Northwestern University Feinberg School of Medicine, Chicago, IL.

Young Kwang Chae (YK)

Northwestern University Feinberg School of Medicine, Chicago, IL.

Classifications MeSH