Temporal and spatial heterogeneity of HER2 status in metastatic colorectal cancer.


Journal

Diagnostic pathology
ISSN: 1746-1596
Titre abrégé: Diagn Pathol
Pays: England
ID NLM: 101251558

Informations de publication

Date de publication:
22 Jun 2024
Historique:
received: 10 04 2024
accepted: 04 06 2024
medline: 23 6 2024
pubmed: 23 6 2024
entrez: 22 6 2024
Statut: epublish

Résumé

HER2-targeted therapies have recently emerged as an option in the management of metastatic colorectal cancer (mCRC) overexpressing HER2. However, data regarding HER2 status in primary CRC and its corresponding liver metastases are limited, potentially influencing clinical decisions. Therefore, the aim of this study was to compare the HER2 status in primary CRC and paired liver metastases. Patients with mCRC who were operated from their primary colorectal cancer and their corresponding synchronous or metachronous liver metastases, in the digestive surgery department of Besançon University Hospital, between April 1999 and October 2021, were included. Tissue microarrays were constructed from matched primary CRC and liver metastastic tissue samples. HER2 status was assessed by immunohistochemistry and in situ hybridization according to Valtorta's criteria. A series of 108 paired primary CRC and liver metastases, including a series of multiple liver metastases originating from the same patients (n = 24), were assessed. Among the primary CRC, 89 (82.4%), 17 (15.8%) and 2 (1.8%) cases were scored 0, 1 + and 2 + respectively. In liver metastases, 99 (91.7%), 7 (6.5%) and 2 (1.8%) were scored 0, 1 + and 2, respectively. Overall, there was a 19% discrepancy rate in HER2 status between primary CRC and metastases, which increased to 21% in cases with multiple synchronous or metachronous liver metastases in a given patient. No significant difference was found between metachronous and synchronous metastases regarding the HER2 status (p = 0.237). Our study highlights the temporal and spatial heterogeneity of HER2 status between primary CRC and corresponding liver metastases. These findings raise the question of a sequential evaluation of the HER2 status during disease progression, to provide the most suitable treatment strategy.

Sections du résumé

BACKGROUND BACKGROUND
HER2-targeted therapies have recently emerged as an option in the management of metastatic colorectal cancer (mCRC) overexpressing HER2. However, data regarding HER2 status in primary CRC and its corresponding liver metastases are limited, potentially influencing clinical decisions. Therefore, the aim of this study was to compare the HER2 status in primary CRC and paired liver metastases.
METHODS METHODS
Patients with mCRC who were operated from their primary colorectal cancer and their corresponding synchronous or metachronous liver metastases, in the digestive surgery department of Besançon University Hospital, between April 1999 and October 2021, were included. Tissue microarrays were constructed from matched primary CRC and liver metastastic tissue samples. HER2 status was assessed by immunohistochemistry and in situ hybridization according to Valtorta's criteria.
RESULTS RESULTS
A series of 108 paired primary CRC and liver metastases, including a series of multiple liver metastases originating from the same patients (n = 24), were assessed. Among the primary CRC, 89 (82.4%), 17 (15.8%) and 2 (1.8%) cases were scored 0, 1 + and 2 + respectively. In liver metastases, 99 (91.7%), 7 (6.5%) and 2 (1.8%) were scored 0, 1 + and 2, respectively. Overall, there was a 19% discrepancy rate in HER2 status between primary CRC and metastases, which increased to 21% in cases with multiple synchronous or metachronous liver metastases in a given patient. No significant difference was found between metachronous and synchronous metastases regarding the HER2 status (p = 0.237).
CONCLUSIONS CONCLUSIONS
Our study highlights the temporal and spatial heterogeneity of HER2 status between primary CRC and corresponding liver metastases. These findings raise the question of a sequential evaluation of the HER2 status during disease progression, to provide the most suitable treatment strategy.

Identifiants

pubmed: 38909265
doi: 10.1186/s13000-024-01508-y
pii: 10.1186/s13000-024-01508-y
doi:

Substances chimiques

Receptor, ErbB-2 EC 2.7.10.1
ERBB2 protein, human EC 2.7.10.1
Biomarkers, Tumor 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

88

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Flavia D'Angelo (F)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Franck Monnien (F)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Alexis Overs (A)

Department of Oncobiology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Irvin Pem (I)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Fanny Dor (F)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Marine Abad (M)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Sophie Felix (S)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Zohair Selmani (Z)

Department of Oncobiology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Zaher Lakkis (Z)

Department of Digestive Surgery, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Christophe Borg (C)

Department of Oncology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Alexandre Doussot (A)

Department of Digestive Surgery, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

Fréderic Bibeau (F)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France. fbibeau@chu-besancon.fr.

Chloé Molimard (C)

Department of Pathology, University Hospital of Besançon, 3 Boulevard Alexandre Fleming, Besancon, 25000, France.

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