Disrupted Endoplasmic Reticulum Ca

Ca2+ handling ER stress chaperone diabetes inositol trisphosphate receptor insulin islet ryanodine receptor unfolded protein response β-cell

Journal

Biology
ISSN: 2079-7737
Titre abrégé: Biology (Basel)
Pays: Switzerland
ID NLM: 101587988

Informations de publication

Date de publication:
25 May 2024
Historique:
received: 03 04 2024
revised: 17 05 2024
accepted: 17 05 2024
medline: 27 6 2024
pubmed: 27 6 2024
entrez: 27 6 2024
Statut: epublish

Résumé

The β-cell workload increases in the setting of insulin resistance and reduced β-cell mass, which occurs in type 2 and type 1 diabetes, respectively. The prolonged elevation of insulin production and secretion during the pathogenesis of diabetes results in β-cell ER stress. The depletion of β-cell Ca

Identifiants

pubmed: 38927260
pii: biology13060379
doi: 10.3390/biology13060379
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

Subventions

Organisme : NIH HHS
ID : 1R01DK129340-23S1
Pays : United States
Organisme : NIH HHS
ID : 1R01DK129340-23A1
Pays : United States
Organisme : NIH HHS
ID : 1R01DK136768-23A1
Pays : United States

Auteurs

Jordyn R Dobson (JR)

Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, TN 37232, USA.

David A Jacobson (DA)

Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, TN 37232, USA.

Classifications MeSH