Milrinone vs Dobutamine for the Management of Cardiogenic Shock: Implications of Renal Function and Injury.

acute kidney injury cardiogenic shock inotropes renal dysfunction

Journal

JACC. Advances
ISSN: 2772-963X
Titre abrégé: JACC Adv
Pays: United States
ID NLM: 9918419284106676

Informations de publication

Date de publication:
Jul 2023
Historique:
received: 28 06 2022
revised: 22 03 2023
accepted: 06 04 2023
medline: 28 6 2024
pubmed: 28 6 2024
entrez: 28 6 2024
Statut: epublish

Résumé

Cardiogenic shock is associated with poor clinical outcomes. There is a paucity of prospective data examining the efficacy and safety of inotropic therapy in patients with cardiogenic shock and renal dysfunction. This study sought to examine the treatment effect of milrinone compared to dobutamine in relation to renal function. In this post hoc analysis of the DOREMI (Milrinone as Compared with Dobutamine in the Treatment of Cardiogenic Shock) trial, we examined clinical outcomes with milrinone compared to dobutamine after stratification based on baseline estimated glomerular filtration rate (eGFR) 60 ml/min/1.73 m Baseline eGFR <60 ml/min/1.73 m In patients requiring inotropic support for cardiogenic shock, baseline renal dysfunction and AKI are common. A modulating effect of AKI on the relative efficacy of milrinone compared to dobutamine was observed, characterized by attenuation of a potential clinical benefit with milrinone compared to dobutamine in patients who develop AKI.

Sections du résumé

Background UNASSIGNED
Cardiogenic shock is associated with poor clinical outcomes. There is a paucity of prospective data examining the efficacy and safety of inotropic therapy in patients with cardiogenic shock and renal dysfunction.
Objectives UNASSIGNED
This study sought to examine the treatment effect of milrinone compared to dobutamine in relation to renal function.
Methods UNASSIGNED
In this post hoc analysis of the DOREMI (Milrinone as Compared with Dobutamine in the Treatment of Cardiogenic Shock) trial, we examined clinical outcomes with milrinone compared to dobutamine after stratification based on baseline estimated glomerular filtration rate (eGFR) 60 ml/min/1.73 m
Results UNASSIGNED
Baseline eGFR <60 ml/min/1.73 m
Conclusions UNASSIGNED
In patients requiring inotropic support for cardiogenic shock, baseline renal dysfunction and AKI are common. A modulating effect of AKI on the relative efficacy of milrinone compared to dobutamine was observed, characterized by attenuation of a potential clinical benefit with milrinone compared to dobutamine in patients who develop AKI.

Identifiants

pubmed: 38938997
doi: 10.1016/j.jacadv.2023.100393
pii: S2772-963X(23)00235-1
pmc: PMC11198346
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100393

Informations de copyright

© 2023 The Authors.

Déclaration de conflit d'intérêts

Funded by the Innovation Fund of the Alternative Funding Plan for the Academic Health Sciences Centres of Ontario. The authors have reported that they have no relationships relevant to the contents of this paper to disclose.PERSPECTIVESCOMPETENCY IN MEDICAL KNOWLEDGE: Renal dysfunction is a prevalent comorbidity and a common sequelae of end-organ hypoperfusion in patients with cardiogenic shock. Significant renal dysfunction is both an indicator and a mediator of a worse prognosis in patients with cardiogenic shock and an important clinical consideration when choosing pharmacotherapies and timing interventions requiring iodinated-contrast agents. At present, there is a paucity of prospective data examining the relative efficacy and safety of milrinone in patients with cardiogenic shock and renal dysfunction. TRANSLATIONAL OUTLOOK: Future studies should further explore the association between milrinone and outcomes in patients with cardiogenic shock and renal dysfunction.

Auteurs

Pietro Di Santo (P)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.
School of Epidemiology and Public Health, University of Ottawa, Ottawa, Ontario, Canada.

Kooroush Dehghan (K)

Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Brennan Mao (B)

Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Richard G Jung (RG)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.
Department of Cellular and Molecular Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Daniel Fadare (D)

Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

John Paydar (J)

Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Simon Parlow (S)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Pouya Motazedian (P)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Graeme Prosperi-Porta (G)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Omar Abdel-Razek (O)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Joanne Joseph (J)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Cheng Yee Goh (CY)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Kevin Chung (K)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Andrew Mulloy (A)

Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

F Daniel Ramirez (FD)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Trevor Simard (T)

Department of Cardiovascular Diseases, Mayo Clinic School of Medicine, Rochester, Minnesota, USA.

Benjamin Hibbert (B)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.
Department of Cellular and Molecular Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Rebecca Mathew (R)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Juan J Russo (JJ)

CAPITAL Research Group, Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Classifications MeSH