Association of Left Atrial Stiffness With Risk of Cryptogenic Ischemic Stroke in Young Adults.

cryptogenic ischemic stroke left atrial mechanics left atrial stiffness obesity stroke in young adults

Journal

JACC. Advances
ISSN: 2772-963X
Titre abrégé: JACC Adv
Pays: United States
ID NLM: 9918419284106676

Informations de publication

Date de publication:
Apr 2024
Historique:
received: 02 10 2023
revised: 01 12 2023
accepted: 02 01 2024
medline: 28 6 2024
pubmed: 28 6 2024
entrez: 28 6 2024
Statut: epublish

Résumé

Incidence of cryptogenic ischemic stroke (CIS) in young adults is increasing. Early left atrial (LA) myopathy might be 1 of the underlying mechanisms, but this has only been scarcely explored. The purpose of this study was to assess the association between increased LA stiffness and CIS in young adults. In the multicenter SECRETO (Searching for Explanations for Cryptogenic Stroke in the Young: Revealing the Etiology, Triggers, and Outcome) study, LA function was analyzed by speckle tracking echocardiography in 150 CIS patients (aged 18-49 years) and 150 age- and sex-matched controls. Minimum and maximum LA volumes, LA reservoir and contractile strain were measured. LA stiffness was calculated by the ratio: mitral peak E-wave velocity divided by mitral annular e' velocity (E/e')/LA reservoir strain and considered increased if ≥0.22. Increased LA volumes, LA stiffness, and/or reduced LA strain indicated LA myopathy. Logistic regression was used to determine the relation between LA stiffness and CIS and the clinical variables associated with LA stiffness. Increased LA stiffness was found in 36% of patients and in 18% of controls ( LA myopathy with increased LA stiffness and impaired LA mechanics more than doubles the risk of CIS in patients under the age of 50 years. This provides new insights into the link between LA dysfunction and CIS at young ages. (Searching for Explanations for Cryptogenic Stroke in the Young: Revealing the Etiology, Triggers, and Outcome [SECRETO]; NCT01934725).

Sections du résumé

Background UNASSIGNED
Incidence of cryptogenic ischemic stroke (CIS) in young adults is increasing. Early left atrial (LA) myopathy might be 1 of the underlying mechanisms, but this has only been scarcely explored.
Objectives UNASSIGNED
The purpose of this study was to assess the association between increased LA stiffness and CIS in young adults.
Methods UNASSIGNED
In the multicenter SECRETO (Searching for Explanations for Cryptogenic Stroke in the Young: Revealing the Etiology, Triggers, and Outcome) study, LA function was analyzed by speckle tracking echocardiography in 150 CIS patients (aged 18-49 years) and 150 age- and sex-matched controls. Minimum and maximum LA volumes, LA reservoir and contractile strain were measured. LA stiffness was calculated by the ratio: mitral peak E-wave velocity divided by mitral annular e' velocity (E/e')/LA reservoir strain and considered increased if ≥0.22. Increased LA volumes, LA stiffness, and/or reduced LA strain indicated LA myopathy. Logistic regression was used to determine the relation between LA stiffness and CIS and the clinical variables associated with LA stiffness.
Results UNASSIGNED
Increased LA stiffness was found in 36% of patients and in 18% of controls (
Conclusions UNASSIGNED
LA myopathy with increased LA stiffness and impaired LA mechanics more than doubles the risk of CIS in patients under the age of 50 years. This provides new insights into the link between LA dysfunction and CIS at young ages. (Searching for Explanations for Cryptogenic Stroke in the Young: Revealing the Etiology, Triggers, and Outcome [SECRETO]; NCT01934725).

Identifiants

pubmed: 38939654
doi: 10.1016/j.jacadv.2024.100903
pii: S2772-963X(24)00082-6
pmc: PMC11198254
doi:

Banques de données

ClinicalTrials.gov
['NCT01934725']

Types de publication

Journal Article

Langues

eng

Pagination

100903

Informations de copyright

© 2024 The Authors.

Déclaration de conflit d'intérêts

The SECRETO study has been funded by the 10.13039/501100002341Academy of Finland under grant numbers 318075 and 322656, Helsinki and Uusimaa Hospital District (TYH2018318), and 10.13039/501100006306Sigrid Juselius Foundation. Dr Cramariuc has received clinical researcher funds and open project support from the Regional Health Authorities in Western Norway (project numbers F-12557 and F-12615). Dr Sarkanen has received grant number 322663 from the Academy of Finland. The authors have reported that they have no relationships relevant to the contents of this paper to disclose.PERSPECTIVESCOMPETENCY IN MEDICAL KNOWLEDGE: Young adults with increased LA stiffness have significantly higher risk of CIS independent of LA size and in the absence of atrial fibrillation. TRANSLATIONAL OUTLOOK: Patients with premature LA disease may benefit from early intervention, like antithrombotic therapy. This should be further investigated in clinical studies addressing preventive and therapeutic interventions in young patients at high risk of ischemic stroke.

Auteurs

Rasmus Bach Sindre (RB)

Department of Clinical Science, University of Bergen, Bergen, Norway.

Eva Gerdts (E)

Department of Clinical Science, Center for Research on Cardiac Disease in Women, University of Bergen, Bergen, Norway.

Jukka Putaala (J)

Department of Neurology, Helsinki University Hospital and University of Helsinki, Finland.

Lisa M D Grymyr (LMD)

Department of Clinical Science, University of Bergen, Bergen, Norway.
Department of Heart Disease, Haukeland University Hospital, Bergen, Norway.

Helga Midtbø (H)

Department of Clinical Science, Center for Research on Cardiac Disease in Women, University of Bergen, Bergen, Norway.
Department of Heart Disease, Haukeland University Hospital, Bergen, Norway.

Ana G Almeida (AG)

Cardiology, Heart and Vessels Department, Faculty of Medicine of Lisbon University, University Hospital Santa Maria, Lisbon, Portugal.

Odd Bech-Hanssen (O)

Department of Clinical Physiology, Sahlgrenska University Hospital and Institute of Medicine, Sahlgrenska Academy at the University of Gothenburg, Gothenburg, Sweden.

Raila Busch (R)

Department of Internal Medicine B (Cardiology), University Medicine Greifswald, Greifswald, Germany.

Rune K Eilertsen (RK)

Department of Clinical Science, University of Bergen, Bergen, Norway.

Ana Catarina Fonseca (AC)

Department of Neurosciences (Neurology), Hospital de Santa Maria, University of Lisbon, Lisbon, Portugal.

Marja Hedman (M)

Heart Center and Clinical Imaging Center, Kuopio University Hospital and University of Eastern Finland, Kuopio, Finland.

Juha Huhtakangas (J)

Department of Neurology, Oulu University Hospital, Oulu, Finland.
Research Unit of Clinical Neuroscience, University of Oulu, Oulu, Finland.

Pekka Jäkälä (P)

Department of Neurology, Kuopio University Hospital and University of Eastern Finland, Kuopio, Finland.

Riikka Lautamäki (R)

Heart Centre, Turku University Hospital and University of Turku, Turku, Finland.

Mika Lehto (M)

Department of Internal Medicine, Jorvi Hospital, HUS Helsinki University Hospital and University of Helsinki, Helsinki, Finland.

Nicolas Martinez-Majander (N)

Department of Neurology, Helsinki University Hospital and University of Helsinki, Helsinki, Finland.

Petra Redfors (P)

Department of Clinical Neuroscience, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Department of Neurology, Sahlgrenska University Hospital, Gothenburg, Sweden.

Tomi Sarkanen (T)

Department of Neurology, Tampere University Hospital and Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland.

Bettina von Sarnowski (B)

Department of Neurology, University Medicine Greifswald, Greifswald, Germany.

Juha Sinisalo (J)

Department of Cardiology, Heart and Lung Center, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Marko Virtanen (M)

Heart Hospital, Tampere University Hospital, Tampere, Finland.

Ulrike Waje-Andreassen (U)

Department of Neurology, Haukeland University Hospital, Bergen, Norway.

Nilufer Yesilot (N)

Istanbul Faculty of Medicine, Department of Neurology, Istanbul University, Istanbul, Turkiye.

Pauli Ylikotila (P)

Neurocenter, Turku University Hospital and Clinical Neurosciences, University of Turku, Turku, Finland.

Dana Cramariuc (D)

Department of Clinical Science, University of Bergen, Bergen, Norway.
Department of Heart Disease, Haukeland University Hospital, Bergen, Norway.

Classifications MeSH