Epigenetic scores derived in saliva are associated with gestational age at birth.


Journal

Clinical epigenetics
ISSN: 1868-7083
Titre abrégé: Clin Epigenetics
Pays: Germany
ID NLM: 101516977

Informations de publication

Date de publication:
29 Jun 2024
Historique:
received: 21 12 2023
accepted: 22 06 2024
medline: 2 7 2024
pubmed: 2 7 2024
entrez: 2 7 2024
Statut: epublish

Résumé

Epigenetic scores (EpiScores), reflecting DNA methylation (DNAm)-based surrogates for complex traits, have been developed for multiple circulating proteins. EpiScores for pro-inflammatory proteins, such as C-reactive protein (DNAm CRP), are associated with brain health and cognition in adults and with inflammatory comorbidities of preterm birth in neonates. Social disadvantage can become embedded in child development through inflammation, and deprivation is overrepresented in preterm infants. We tested the hypotheses that preterm birth and socioeconomic status (SES) are associated with alterations in a set of EpiScores enriched for inflammation-associated proteins. In total, 104 protein EpiScores were derived from saliva samples of 332 neonates born at gestational age (GA) 22.14 to 42.14 weeks. Saliva sampling was between 36.57 and 47.14 weeks. Forty-three (41%) EpiScores were associated with low GA at birth (standardised estimates |0.14 to 0.88|, Bonferroni-adjusted p-value < 8.3 × 10 Low birth GA is substantially associated with a set of EpiScores. The set was enriched for inflammatory proteins, providing new insights into immune dysregulation in preterm infants. SES had fewer associations with EpiScores; these tended to have small effect sizes and were not statistically significant after adjusting for inflammatory comorbidities. This suggests that inflammation is unlikely to be the primary axis through which SES becomes embedded in the development of preterm infants in the neonatal period.

Sections du résumé

BACKGROUND BACKGROUND
Epigenetic scores (EpiScores), reflecting DNA methylation (DNAm)-based surrogates for complex traits, have been developed for multiple circulating proteins. EpiScores for pro-inflammatory proteins, such as C-reactive protein (DNAm CRP), are associated with brain health and cognition in adults and with inflammatory comorbidities of preterm birth in neonates. Social disadvantage can become embedded in child development through inflammation, and deprivation is overrepresented in preterm infants. We tested the hypotheses that preterm birth and socioeconomic status (SES) are associated with alterations in a set of EpiScores enriched for inflammation-associated proteins.
RESULTS RESULTS
In total, 104 protein EpiScores were derived from saliva samples of 332 neonates born at gestational age (GA) 22.14 to 42.14 weeks. Saliva sampling was between 36.57 and 47.14 weeks. Forty-three (41%) EpiScores were associated with low GA at birth (standardised estimates |0.14 to 0.88|, Bonferroni-adjusted p-value < 8.3 × 10
CONCLUSIONS CONCLUSIONS
Low birth GA is substantially associated with a set of EpiScores. The set was enriched for inflammatory proteins, providing new insights into immune dysregulation in preterm infants. SES had fewer associations with EpiScores; these tended to have small effect sizes and were not statistically significant after adjusting for inflammatory comorbidities. This suggests that inflammation is unlikely to be the primary axis through which SES becomes embedded in the development of preterm infants in the neonatal period.

Identifiants

pubmed: 38951914
doi: 10.1186/s13148-024-01701-2
pii: 10.1186/s13148-024-01701-2
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

84

Subventions

Organisme : MRC Clinician Scientist Fellowship
ID : MR/X019535/1
Organisme : Sir Henry Dale Fellowship jointly funded by the Wellcome Trust and the Royal Society
ID : 221890/Z/20/Z
Organisme : UKRI MRC Programme Grant
ID : MR/X003434/1

Informations de copyright

© 2024. The Author(s).

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Auteurs

Katie Mckinnon (K)

Centre for Reproductive Health, Institute for Regeneration and Repair, University of Edinburgh, 4-5 Little France Drive, Edinburgh, EH16 4UU, UK.

Eleanor L S Conole (ELS)

Lothian Birth Cohorts, Department of Psychology, University of Edinburgh, Edinburgh, UK.

Kadi Vaher (K)

Centre for Reproductive Health, Institute for Regeneration and Repair, University of Edinburgh, 4-5 Little France Drive, Edinburgh, EH16 4UU, UK.
Centre for Clinical Brain Sciences, University of Edinburgh, Edinburgh, UK.

Robert F Hillary (RF)

Centre for Genomic and Experimental Medicine, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh, UK.

Danni A Gadd (DA)

Centre for Genomic and Experimental Medicine, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh, UK.

Justyna Binkowska (J)

Centre for Reproductive Health, Institute for Regeneration and Repair, University of Edinburgh, 4-5 Little France Drive, Edinburgh, EH16 4UU, UK.

Gemma Sullivan (G)

Centre for Clinical Brain Sciences, University of Edinburgh, Edinburgh, UK.

Anna J Stevenson (AJ)

Centre for Genomic and Experimental Medicine, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh, UK.

Amy Corrigan (A)

Centre for Reproductive Health, Institute for Regeneration and Repair, University of Edinburgh, 4-5 Little France Drive, Edinburgh, EH16 4UU, UK.

Lee Murphy (L)

Edinburgh Clinical Research Facility, University of Edinburgh, Edinburgh, UK.

Heather C Whalley (HC)

Centre for Clinical Brain Sciences, University of Edinburgh, Edinburgh, UK.

Hilary Richardson (H)

School of Philosophy, Psychology, and Language Sciences, University of Edinburgh, Edinburgh, UK.

Riccardo E Marioni (RE)

Centre for Genomic and Experimental Medicine, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh, UK.

Simon R Cox (SR)

Lothian Birth Cohorts, Department of Psychology, University of Edinburgh, Edinburgh, UK.

James P Boardman (JP)

Centre for Reproductive Health, Institute for Regeneration and Repair, University of Edinburgh, 4-5 Little France Drive, Edinburgh, EH16 4UU, UK. James.Boardman@ed.ac.uk.
Centre for Clinical Brain Sciences, University of Edinburgh, Edinburgh, UK. James.Boardman@ed.ac.uk.

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