Cardio-metabolic health effects of CPAP treatment for sleep apnoea during weight loss: A randomised controlled pilot trial.

Continuous positive airway pressure Obesity Obstructive sleep apnoea Weight loss

Journal

Obesity research & clinical practice
ISSN: 1871-403X
Titre abrégé: Obes Res Clin Pract
Pays: Netherlands
ID NLM: 101303911

Informations de publication

Date de publication:
01 Jul 2024
Historique:
received: 22 02 2024
revised: 06 06 2024
accepted: 24 06 2024
medline: 3 7 2024
pubmed: 3 7 2024
entrez: 2 7 2024
Statut: aheadofprint

Résumé

This study assessed whether the addition of continuous positive airway pressure (CPAP) during weight loss would enhance cardiometabolic health improvements in patients with obesity and Obstructive Sleep Apnoea (OSA). Patients with overweight or obesity, pre-diabetes and moderatesevere OSA were randomised to receive CPAP therapy with a weight loss programme (CPAP+WL) or a weight loss programme alone (WL alone). 2-hour glucose assessed by an oral glucose tolerance test. 24 hr blood pressure, body composition (DEXA) and fasting blood markers. 17 patients completed 3-month follow-up assessments (8 CPAP+WL and 9 WL alone). Overall, participants in both groups lost ∼12 kg which reduced polysomnography determined OSA severity by ∼45 %. In the CPAP+WL group, CPAP use (compliance 5.29 hrs/night) did not improve any outcome above WL alone. There was no improvement in 2-hour glucose in either group. However, in the pooled (n = 17) analysis there were overall improvements in most outcomes including insulin sensitivity (.000965 units, p = .008), sleep systolic BP (- 16.2 mmHg, p = .0003), sleep diastolic BP (-9.8 mmHg, p = 0.02), wake diastolic BP (- 4.3 mmHg, p = .03) and sleepiness (Epworth Sleepiness Score -3.2, p = .0003). In addition, there were reductions in glucose area under the curve (-230 units, p = .009), total (-0.86 mmol/L, p = 0.006) and LDL cholesterol (-0.58 mmol/L, p = 0.007), triglycerides (-0.75 mmol/L, p = 0.004), fat mass (-7.6 kg, p < .0001) and abdominal fat (-310 cm3, p < .0001). Weight loss reduced OSA and improved sleepiness and cardiometabolic health. These improvements were not further enhanced by using CPAP. Results suggest weight loss should be the primary focus of treatment for patients with OSA and obesity.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
This study assessed whether the addition of continuous positive airway pressure (CPAP) during weight loss would enhance cardiometabolic health improvements in patients with obesity and Obstructive Sleep Apnoea (OSA).
METHODS AND RESULTS RESULTS
Patients with overweight or obesity, pre-diabetes and moderatesevere OSA were randomised to receive CPAP therapy with a weight loss programme (CPAP+WL) or a weight loss programme alone (WL alone).
PRIMARY OUTCOME METHODS
2-hour glucose assessed by an oral glucose tolerance test.
SECONDARY OUTCOMES RESULTS
24 hr blood pressure, body composition (DEXA) and fasting blood markers. 17 patients completed 3-month follow-up assessments (8 CPAP+WL and 9 WL alone). Overall, participants in both groups lost ∼12 kg which reduced polysomnography determined OSA severity by ∼45 %. In the CPAP+WL group, CPAP use (compliance 5.29 hrs/night) did not improve any outcome above WL alone. There was no improvement in 2-hour glucose in either group. However, in the pooled (n = 17) analysis there were overall improvements in most outcomes including insulin sensitivity (.000965 units, p = .008), sleep systolic BP (- 16.2 mmHg, p = .0003), sleep diastolic BP (-9.8 mmHg, p = 0.02), wake diastolic BP (- 4.3 mmHg, p = .03) and sleepiness (Epworth Sleepiness Score -3.2, p = .0003). In addition, there were reductions in glucose area under the curve (-230 units, p = .009), total (-0.86 mmol/L, p = 0.006) and LDL cholesterol (-0.58 mmol/L, p = 0.007), triglycerides (-0.75 mmol/L, p = 0.004), fat mass (-7.6 kg, p < .0001) and abdominal fat (-310 cm3, p < .0001).
CONCLUSION CONCLUSIONS
Weight loss reduced OSA and improved sleepiness and cardiometabolic health. These improvements were not further enhanced by using CPAP. Results suggest weight loss should be the primary focus of treatment for patients with OSA and obesity.

Identifiants

pubmed: 38955574
pii: S1871-403X(24)00062-0
doi: 10.1016/j.orcp.2024.06.003
pii:
doi:

Types de publication

Letter

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Asia Oceania Association for the Study of Obesity. Published by Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest S. Postnova reports grants unrelated to the manuscript from NHMRC and ARC. T. Markovich reports grants unrelated to the manuscript from NHMRC and advisory board participation with Nestle Health Science and Eli Lilly. S. Twigg reports advisory board participation with Abbott Diabetes care and Nevro Inc and payments from Nevro Inc and Astra Zeneca Inc for presentations. R. Grunstein reports advisory board participation and payments from Eli Lilly for SURMOUNT OSA weight loss trial. All other authors have no conflict of interest to declare.

Auteurs

Camilla M Hoyos (CM)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Faculty of Medicine, Health and Human Sciences, Macquarie University, Australia.

Elizabeth A Machan (EA)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Faculty of Medicine, University of Sydney, Australia.

Brendon J Yee (BJ)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Department of Respiratory and Sleep Medicine, Royal Prince Alfred Hospital, Australia; Faculty of Medicine, University of Sydney, Australia.

Svetlana Postnova (S)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Faculty of Science, University of Sydney, Australia.

Nathaniel S Marshall (NS)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Faculty of Medicine, Health and Human Sciences, Macquarie University, Australia.

Tania Markovic (T)

Faculty of Medicine, University of Sydney, Australia.

Stephen M Twigg (SM)

Faculty of Medicine, University of Sydney, Australia.

Ronald R Grunstein (RR)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Department of Respiratory and Sleep Medicine, Royal Prince Alfred Hospital, Australia.

Craig L Phillips (CL)

CIRUS Centre for Sleep and Chronobiology, Woolcock Institute of Medical Research, Macquarie University, Australia; Faculty of Medicine, Health and Human Sciences, Macquarie University, Australia. Electronic address: c.phillips@mq.edu.au.

Classifications MeSH