Neuropathogenesis of SARS-CoV-2 in human neuronal, microglial and glial cells.


Journal

Archives of microbiology
ISSN: 1432-072X
Titre abrégé: Arch Microbiol
Pays: Germany
ID NLM: 0410427

Informations de publication

Date de publication:
08 Jul 2024
Historique:
received: 12 04 2024
accepted: 28 06 2024
medline: 8 7 2024
pubmed: 8 7 2024
entrez: 8 7 2024
Statut: epublish

Résumé

Neurological complications, both acute and chronic, are reported commonly in COVID-19 affected individuals. In this context, the understanding of pathogenesis of SARS-CoV-2 in specific cells of central nervous system (CNS) origin is relevant. The present study explores infection biology of a clinical isolate of SARS-CoV-2 in human cell lines of neural origin such as the glioblastoma (U87-MG), neuroblastoma (SHSY5Y) and microglia (C20). Despite showing clear evidence of infection by immunofluorescence with an anti-spike protein antibody, all the three neural cell lines were observed to be highly restrictive to the replication of the infecting virus. While the U87-MG glioblastoma cells demonstrated no cytopathic effects and a low viral titre with no signs of replication, the SHSY5Y neuroblastoma cells exhibited cytopathic effects with bleb formation but no evidence of viable virus. The C20 microglial cells showed neither signs of cytopathic effects nor viable virus. Ultrastructural studies demonstrated intracellular virions in infected neural cells. The presence of lipid droplets in infected SHSY5Y cells suggested an impact on host cell metabolism. The decrease in viral RNA levels over time in all the neural cell lines suggested restricted viral replication. In conclusion, this study highlights the limited susceptibility of neural cells to SARS-CoV-2 infection. This reduced permissibility of neural cell lines to SARS-CoV-2 may point to their inherent lower expression of receptors that support viral entry in addition to the intracellular factors that potently inhibit viral replication. The study findings prompt further investigation into the mechanisms of SARS-CoV-2 infection of neural cells.

Identifiants

pubmed: 38976047
doi: 10.1007/s00203-024-04075-4
pii: 10.1007/s00203-024-04075-4
doi:

Substances chimiques

Spike Glycoprotein, Coronavirus 0
RNA, Viral 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

345

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Narendra Kumar (N)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Hosur Road, Bengaluru, 560029, India.

Rashmi Santhoshkumar (R)

Electron Microscopy- Common Research Facility, Department of Neuropathology, NIMHANS, Bengaluru, 560029, India.

Ragini Agrawal (R)

Center for Infectious Disease Research (CIDR), Indian Institute of Science (IISc), CV Raman Avenue, Bangalore, 560012, India.

Amit Singh (A)

Center for Infectious Disease Research (CIDR), Indian Institute of Science (IISc), CV Raman Avenue, Bangalore, 560012, India.

Vijayalakshmi Kalyan (V)

Department of Neurophysiology, NIMHANS, Bengaluru, 560029, India.

Anita Desai (A)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Hosur Road, Bengaluru, 560029, India.

Vasanthapuram Ravi (V)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Hosur Road, Bengaluru, 560029, India.

Manjunatha M Venkataswamy (MM)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Hosur Road, Bengaluru, 560029, India. manjuvswamy@gmail.com.

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