Specificity of serological screening tests and reference laboratory tests to diagnose gambiense human African trypanosomiasis: a prospective clinical performance study.


Journal

Infectious diseases of poverty
ISSN: 2049-9957
Titre abrégé: Infect Dis Poverty
Pays: England
ID NLM: 101606645

Informations de publication

Date de publication:
08 Jul 2024
Historique:
received: 19 04 2024
accepted: 25 06 2024
medline: 9 7 2024
pubmed: 9 7 2024
entrez: 8 7 2024
Statut: epublish

Résumé

Serological screening tests play a crucial role to diagnose gambiense human African trypanosomiasis (gHAT). Presently, they preselect individuals for microscopic confirmation, but in future "screen and treat" strategies they will identify individuals for treatment. Variability in reported specificities, the development of new rapid diagnostic tests (RDT) and the hypothesis that malaria infection may decrease RDT specificity led us to evaluate the specificity of 5 gHAT screening tests. During active screening, venous blood samples from 1095 individuals from Côte d'Ivoire and Guinea were tested consecutively with commercial (CATT, HAT Sero-K-SeT, Abbott Bioline HAT 2.0) and prototype (DCN HAT RDT, HAT Sero-K-SeT 2.0) gHAT screening tests and with a malaria RDT. Individuals with ≥ 1 positive gHAT screening test underwent microscopy and further immunological (trypanolysis with T.b. gambiense LiTat 1.3, 1.5 and 1.6; indirect ELISA/T.b. gambiense; T.b. gambiense inhibition ELISA with T.b. gambiense LiTat 1.3 and 1.5 VSG) and molecular reference laboratory tests (PCR TBRN3, 18S and TgsGP; SHERLOCK 18S Tids, 7SL Zoon, and TgsGP; Trypanozoon S One gHAT case was diagnosed. Overall test specificities (n = 1094) were: CATT 98.9% (95% CI: 98.1-99.4%); HAT Sero-K-SeT 86.7% (95% CI: 84.5-88.5%); Bioline HAT 2.0 82.1% (95% CI: 79.7-84.2%); DCN HAT RDT 78.2% (95% CI: 75.7-80.6%); and HAT Sero-K-SeT 2.0 78.4% (95% CI: 75.9-80.8%). In malaria positives, gHAT screening tests appeared less specific, but the difference was significant only in Guinea for Abbott Bioline HAT 2.0 (P = 0.03) and HAT Sero-K-Set 2.0 (P = 0.0006). The specificities of immunological and molecular laboratory tests in gHAT seropositives were 98.7-100% (n = 399) and 93.0-100% (n = 302), respectively. Among 44 reference laboratory test positives, only the confirmed gHAT patient and one screening test seropositive combined immunological and molecular reference laboratory test positivity. Although a minor effect of malaria cannot be excluded, gHAT RDT specificities are far below the 95% minimal specificity stipulated by the WHO target product profile for a simple diagnostic tool to identify individuals eligible for treatment. Unless specificity is improved, an RDT-based "screen and treat" strategy would result in massive overtreatment. In view of their inconsistent results, additional comparative evaluations of the diagnostic performance of reference laboratory tests are indicated for better identifying, among screening test positives, those at increased suspicion for gHAT. The trial was retrospectively registered under NCT05466630 in clinicaltrials.gov on July 15 2022.

Sections du résumé

BACKGROUND BACKGROUND
Serological screening tests play a crucial role to diagnose gambiense human African trypanosomiasis (gHAT). Presently, they preselect individuals for microscopic confirmation, but in future "screen and treat" strategies they will identify individuals for treatment. Variability in reported specificities, the development of new rapid diagnostic tests (RDT) and the hypothesis that malaria infection may decrease RDT specificity led us to evaluate the specificity of 5 gHAT screening tests.
METHODS METHODS
During active screening, venous blood samples from 1095 individuals from Côte d'Ivoire and Guinea were tested consecutively with commercial (CATT, HAT Sero-K-SeT, Abbott Bioline HAT 2.0) and prototype (DCN HAT RDT, HAT Sero-K-SeT 2.0) gHAT screening tests and with a malaria RDT. Individuals with ≥ 1 positive gHAT screening test underwent microscopy and further immunological (trypanolysis with T.b. gambiense LiTat 1.3, 1.5 and 1.6; indirect ELISA/T.b. gambiense; T.b. gambiense inhibition ELISA with T.b. gambiense LiTat 1.3 and 1.5 VSG) and molecular reference laboratory tests (PCR TBRN3, 18S and TgsGP; SHERLOCK 18S Tids, 7SL Zoon, and TgsGP; Trypanozoon S
RESULTS RESULTS
One gHAT case was diagnosed. Overall test specificities (n = 1094) were: CATT 98.9% (95% CI: 98.1-99.4%); HAT Sero-K-SeT 86.7% (95% CI: 84.5-88.5%); Bioline HAT 2.0 82.1% (95% CI: 79.7-84.2%); DCN HAT RDT 78.2% (95% CI: 75.7-80.6%); and HAT Sero-K-SeT 2.0 78.4% (95% CI: 75.9-80.8%). In malaria positives, gHAT screening tests appeared less specific, but the difference was significant only in Guinea for Abbott Bioline HAT 2.0 (P = 0.03) and HAT Sero-K-Set 2.0 (P = 0.0006). The specificities of immunological and molecular laboratory tests in gHAT seropositives were 98.7-100% (n = 399) and 93.0-100% (n = 302), respectively. Among 44 reference laboratory test positives, only the confirmed gHAT patient and one screening test seropositive combined immunological and molecular reference laboratory test positivity.
CONCLUSIONS CONCLUSIONS
Although a minor effect of malaria cannot be excluded, gHAT RDT specificities are far below the 95% minimal specificity stipulated by the WHO target product profile for a simple diagnostic tool to identify individuals eligible for treatment. Unless specificity is improved, an RDT-based "screen and treat" strategy would result in massive overtreatment. In view of their inconsistent results, additional comparative evaluations of the diagnostic performance of reference laboratory tests are indicated for better identifying, among screening test positives, those at increased suspicion for gHAT.
TRIAL REGISTRATION BACKGROUND
The trial was retrospectively registered under NCT05466630 in clinicaltrials.gov on July 15 2022.

Identifiants

pubmed: 38978124
doi: 10.1186/s40249-024-01220-5
pii: 10.1186/s40249-024-01220-5
doi:

Substances chimiques

Antibodies, Protozoan 0

Banques de données

ClinicalTrials.gov
['NCT05466630']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

53

Subventions

Organisme : Swiss Agency for Development and Cooperation
ID : 81071426
Organisme : Swiss Agency for Development and Cooperation
ID : 7F-08866.03.01
Organisme : Bill & Melinda Gates Foundation
ID : INV-001785
Pays : United States
Organisme : Bill and Melinda Gates Foundation
ID : OPP1033712
Organisme : Bill and Melinda Gates Foundation
ID : OPP1154033
Organisme : Bill and Melinda Gates Foundation
ID : INV-031353

Informations de copyright

© 2024. The Author(s).

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Auteurs

Martial Kassi N'Djetchi (MK)

Laboratory of Biodiversity and Ecosystem Management, Jean Lorougnon Guédé University, Daloa, Côte d'Ivoire.

Oumou Camara (O)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.

Mathurin Koffi (M)

Laboratory of Biodiversity and Ecosystem Management, Jean Lorougnon Guédé University, Daloa, Côte d'Ivoire.

Mamadou Camara (M)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.

Dramane Kaba (D)

Trypanosomosis Research Unit, Pierre Richet Institute, Bouaké, Côte d'Ivoire.

Jacques Kaboré (J)

International Research and Development Centre on Livestock in Subhumid Zones, Bobo-Dioulasso, Burkina Faso.

Alkali Tall (A)

National Program for Malaria Control, Conakry, Guinea.

Brice Rotureau (B)

Parasitology Unit, Institut Pasteur de Guinée, Conakry, Guinea.

Lucy Glover (L)

Trypanosome Molecular Biology Unit, Department of Parasites and Insect Vectors, Pasteur Institute, Paris Cité University, Paris, France.

Mélika Barkissa Traoré (MB)

Laboratory of Biodiversity and Ecosystem Management, Jean Lorougnon Guédé University, Daloa, Côte d'Ivoire.

Minayegninrin Koné (M)

Laboratory of Biodiversity and Ecosystem Management, Jean Lorougnon Guédé University, Daloa, Côte d'Ivoire.

Bamoro Coulibaly (B)

Trypanosomosis Research Unit, Pierre Richet Institute, Bouaké, Côte d'Ivoire.

Guy Pacome Adingra (GP)

Trypanosomosis Research Unit, Pierre Richet Institute, Bouaké, Côte d'Ivoire.

Aissata Soumah (A)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.

Mohamed Gassama (M)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.

Abdoulaye Dansy Camara (AD)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.

Charlie Franck Alfred Compaoré (CFA)

International Research and Development Centre on Livestock in Subhumid Zones, Bobo-Dioulasso, Burkina Faso.

Aïssata Camara (A)

Parasitology Unit, Institut Pasteur de Guinée, Conakry, Guinea.

Salimatou Boiro (S)

Parasitology Unit, Institut Pasteur de Guinée, Conakry, Guinea.

Elena Perez Anton (EP)

Trypanosome Molecular Biology Unit, Department of Parasites and Insect Vectors, Pasteur Institute, Paris Cité University, Paris, France.

Paul Bessell (P)

Independent Consultant, Edinburgh, UK.

Nick Van Reet (N)

Department of Biomedical Sciences, Institute of Tropical Medicine, Antwerp, Belgium.

Bruno Bucheton (B)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.
Intertryp, IRD-CIRAD-University of Montpellier, Montpellier, France.

Vincent Jamonneau (V)

Trypanosomosis Research Unit, Pierre Richet Institute, Bouaké, Côte d'Ivoire.
Intertryp, IRD-CIRAD-University of Montpellier, Montpellier, France.

Jean-Mathieu Bart (JM)

National Program for Neglected Tropical Disease Control, Patient Management, Ministry of Health, Conakry, Guinea.
Intertryp, IRD-CIRAD-University of Montpellier, Montpellier, France.

Philippe Solano (P)

Intertryp, IRD-CIRAD-University of Montpellier, Montpellier, France.

Sylvain Biéler (S)

Foundation for Innovative New Diagnostics, Geneva, Switzerland.

Veerle Lejon (V)

Intertryp, IRD-CIRAD-University of Montpellier, Montpellier, France. Veerle.lejon@ird.fr.

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