The impact of alcohol and illicit substance use on the pharmacokinetics of first-line TB drugs.


Journal

The Journal of antimicrobial chemotherapy
ISSN: 1460-2091
Titre abrégé: J Antimicrob Chemother
Pays: England
ID NLM: 7513617

Informations de publication

Date de publication:
10 Jul 2024
Historique:
received: 29 02 2024
accepted: 03 06 2024
medline: 10 7 2024
pubmed: 10 7 2024
entrez: 10 7 2024
Statut: aheadofprint

Résumé

In South Africa, an estimated 11% of the population have high alcohol use, a major risk factor for TB. Alcohol and other substance use are also associated with poor treatment response, with a potential mechanism being altered TB drug pharmacokinetics. To investigate the impact of alcohol and illicit substance use on the pharmacokinetics of first-line TB drugs in participants with pulmonary TB. We prospectively enrolled participants ≥15 years old, without HIV, and initiating drug-susceptible TB treatment in Worcester, South Africa. Alcohol use was measured via self-report and blood biomarkers. Other illicit substances were captured through a urine drug test. Plasma samples were drawn 1 month into treatment pre-dose, and 1.5, 3, 5 and 8 h post-dose. Non-linear mixed-effects modelling was used to describe the pharmacokinetics of rifampicin, isoniazid, pyrazinamide and ethambutol. Alcohol and drug use were tested as covariates. The study included 104 participants, of whom 70% were male, with a median age of 37 years (IQR 27-48). Alcohol use was high, with 42% and 28% of participants having moderate and high alcohol use, respectively. Rifampicin and isoniazid had slightly lower pharmacokinetics compared with previous reports, whereas pyrazinamide and ethambutol were consistent. No significant alcohol use effect was detected, other than 13% higher ethambutol clearance in participants with high alcohol use. Methaqualone use reduced rifampicin bioavailability by 19%. No clinically relevant effect of alcohol use was observed on the pharmacokinetics of first-line TB drugs, suggesting that poor treatment outcome is unlikely due to pharmacokinetic alterations. That methaqualone reduced rifampicin means dose adjustment may be beneficial.

Sections du résumé

BACKGROUND BACKGROUND
In South Africa, an estimated 11% of the population have high alcohol use, a major risk factor for TB. Alcohol and other substance use are also associated with poor treatment response, with a potential mechanism being altered TB drug pharmacokinetics.
OBJECTIVES OBJECTIVE
To investigate the impact of alcohol and illicit substance use on the pharmacokinetics of first-line TB drugs in participants with pulmonary TB.
METHODS METHODS
We prospectively enrolled participants ≥15 years old, without HIV, and initiating drug-susceptible TB treatment in Worcester, South Africa. Alcohol use was measured via self-report and blood biomarkers. Other illicit substances were captured through a urine drug test. Plasma samples were drawn 1 month into treatment pre-dose, and 1.5, 3, 5 and 8 h post-dose. Non-linear mixed-effects modelling was used to describe the pharmacokinetics of rifampicin, isoniazid, pyrazinamide and ethambutol. Alcohol and drug use were tested as covariates.
RESULTS RESULTS
The study included 104 participants, of whom 70% were male, with a median age of 37 years (IQR 27-48). Alcohol use was high, with 42% and 28% of participants having moderate and high alcohol use, respectively. Rifampicin and isoniazid had slightly lower pharmacokinetics compared with previous reports, whereas pyrazinamide and ethambutol were consistent. No significant alcohol use effect was detected, other than 13% higher ethambutol clearance in participants with high alcohol use. Methaqualone use reduced rifampicin bioavailability by 19%.
CONCLUSION CONCLUSIONS
No clinically relevant effect of alcohol use was observed on the pharmacokinetics of first-line TB drugs, suggesting that poor treatment outcome is unlikely due to pharmacokinetic alterations. That methaqualone reduced rifampicin means dose adjustment may be beneficial.

Identifiants

pubmed: 38985541
pii: 7710581
doi: 10.1093/jac/dkae206
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : United States National Institute of Allergy and Infectious Diseases
ID : R01AI119037
Organisme : Wellcome Trust Sir Henry Dale Fellowship
ID : 220587/Z/20/Z
Organisme : Adult Clinical Trial Group
Organisme : National Institute of Allergy and Infectious Diseases
Organisme : NIH HHS
ID : UM1 AI068634
Pays : United States

Informations de copyright

© The Author(s) 2024. Published by Oxford University Press on behalf of British Society for Antimicrobial Chemotherapy.

Auteurs

Marie Wijk (M)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Kamunkhwala Gausi (K)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Samantha Malatesta (S)

Department of Biostatistics, Boston University School of Public Health, Boston, MA, USA.

Sarah E Weber (SE)

Section of Infectious Diseases, Boston University School of Medicine and Boston Medical Centre, Boston, MA, USA.
Department of Epidemiology, Boston University School of Public Health, Boston, MA, USA.

Richard Court (R)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Bronwyn Myers (B)

Curtin enAble Institute, Curtin University, WA, Australia.
Mental Health, Alcohol, Substance Use and Tobacco Research Unit, South African Medical Research Council, Cape Town, South Africa.
Department of Psychiatry and Mental Health, University of Cape Town, Rondebosch, South Africa.

Tara Carney (T)

Mental Health, Alcohol, Substance Use and Tobacco Research Unit, South African Medical Research Council, Cape Town, South Africa.
Department of Psychiatry and Mental Health, University of Cape Town, Rondebosch, South Africa.

Charles D H Parry (CDH)

Mental Health, Alcohol, Substance Use and Tobacco Research Unit, South African Medical Research Council, Cape Town, South Africa.
Department of Psychiatry, Stellenbosch University, Cape Town, South Africa.

C Robert Horsburgh (CR)

Department of Epidemiology, Boston University School of Public Health, Boston, MA, USA.

Laura F White (LF)

Department of Biostatistics, Boston University School of Public Health, Boston, MA, USA.

Lubbe Wiesner (L)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Robin M Warren (RM)

DSI-NRF Centre of Excellence for Biomedical Tuberculosis Research, South African Medical Research Council Centre for Tuberculosis Research, Division of Molecular Biology and Human Genetics, Faculty of Medicine and Health Sciences, Stellenbosch University, Cape Town, South Africa.

Caitlin Uren (C)

DSI-NRF Centre of Excellence for Biomedical Tuberculosis Research, South African Medical Research Council Centre for Tuberculosis Research, Division of Molecular Biology and Human Genetics, Faculty of Medicine and Health Sciences, Stellenbosch University, Cape Town, South Africa.
Centre for Bioinformatics and Computational Biology, Stellenbosch University, Stellenbosch, South Africa.

Helen McIlleron (H)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Frank Kloprogge (F)

Institute for Global Health, University College London, London, UK.

Paolo Denti (P)

Department of Medicine, University of Cape Town, Cape Town, South Africa.

Karen R Jacobson (KR)

Section of Infectious Diseases, Boston University School of Medicine and Boston Medical Centre, Boston, MA, USA.

Classifications MeSH