Total pulmonary arterial reconstruction in a patient with arterial tortuosity syndrome affecting the pulmonary artery: a case report and review of the literature.


Journal

Journal of cardiothoracic surgery
ISSN: 1749-8090
Titre abrégé: J Cardiothorac Surg
Pays: England
ID NLM: 101265113

Informations de publication

Date de publication:
10 Jul 2024
Historique:
received: 09 04 2024
accepted: 15 06 2024
medline: 11 7 2024
pubmed: 11 7 2024
entrez: 10 7 2024
Statut: epublish

Résumé

Arterial tortuosity syndrome is a rare Autosomal recessive disease that leads to a loss of function of the connective tissues of the body, this happens due to a mutation in the solute carrier family 2 member 10 (SLC2A10) gene. ATS is more likely to occur in Large and medium-sized arteries including the aorta and pulmonary arteries. This syndrome causes the arteries to be elongated and tortuous, This tortuosity disturbs the blood circulation resulting in stenosis and lack of blood flow to organs and this chronic turbulent flow increases the risk of aneurysm development, dissection and ischemic events. A 2 years old Arabian female child was diagnosed with ATS affecting the pulmonary arteries as a newborn, underwent a pulmonary arterial surgical reconstruction at the age of 2 years old due to the development of pulmonary artery stenosis with left pulmonary artery having a peak gradient of 73 mmHg with a peak velocity of 4.3 m/s and the right pulmonary artery having a peak gradient of 46 mmHg with a peak velocity of 3.4 m/s causing right ventricular hypertension. After surgical repair the left pulmonary artery has a peak pressure gradient of 20 mmHg, with the right pulmonary artery having a peak pressure gradient of 20 mmHg. ATS is a rare genetic condition that affects the great arteries especially the pulmonary arteries causing stenotic and tortuous vessels that may be central branches or distal peripheral branches that leads to severe right ventricular dysfunction and hypertension. We believe that surgical treatment provides the optimum outcomes when compared to transcather approaches especially when the peripheral arteries are involved. Some challenges and hiccups might occur, especially lung reperfusion injury that needs to be diagnosed and treated accordingly.

Sections du résumé

BACKGROUND BACKGROUND
Arterial tortuosity syndrome is a rare Autosomal recessive disease that leads to a loss of function of the connective tissues of the body, this happens due to a mutation in the solute carrier family 2 member 10 (SLC2A10) gene. ATS is more likely to occur in Large and medium-sized arteries including the aorta and pulmonary arteries. This syndrome causes the arteries to be elongated and tortuous, This tortuosity disturbs the blood circulation resulting in stenosis and lack of blood flow to organs and this chronic turbulent flow increases the risk of aneurysm development, dissection and ischemic events.
CASE PRESENTATION METHODS
A 2 years old Arabian female child was diagnosed with ATS affecting the pulmonary arteries as a newborn, underwent a pulmonary arterial surgical reconstruction at the age of 2 years old due to the development of pulmonary artery stenosis with left pulmonary artery having a peak gradient of 73 mmHg with a peak velocity of 4.3 m/s and the right pulmonary artery having a peak gradient of 46 mmHg with a peak velocity of 3.4 m/s causing right ventricular hypertension. After surgical repair the left pulmonary artery has a peak pressure gradient of 20 mmHg, with the right pulmonary artery having a peak pressure gradient of 20 mmHg.
CONCLUSION CONCLUSIONS
ATS is a rare genetic condition that affects the great arteries especially the pulmonary arteries causing stenotic and tortuous vessels that may be central branches or distal peripheral branches that leads to severe right ventricular dysfunction and hypertension. We believe that surgical treatment provides the optimum outcomes when compared to transcather approaches especially when the peripheral arteries are involved. Some challenges and hiccups might occur, especially lung reperfusion injury that needs to be diagnosed and treated accordingly.

Identifiants

pubmed: 38987788
doi: 10.1186/s13019-024-02905-6
pii: 10.1186/s13019-024-02905-6
doi:

Types de publication

Case Reports Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

432

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Fahad M Alshair (FM)

Division of Cardiac Surgery, Department of Surgery, King Abdulaziz University Hospital, P.O. Box: 80215, Jeddah, 21589, Saudi Arabia. Fahadalshair@gmail.com.

Amal S Alsulami (AS)

Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.

Mohammad S Shihata (MS)

Cardiothoracic Surgery Department, King Faisal Specialist Hospital and Research Centre, Jeddah, Saudi Arabia.

Osman O Alradi (OO)

Division of Cardiac Surgery, Department of Surgery, King Abdulaziz University Hospital, P.O. Box: 80215, Jeddah, 21589, Saudi Arabia.
Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.

Ragab S Debis (RS)

Division of Cardiac Surgery, Department of Surgery, King Abdulaziz University Hospital, P.O. Box: 80215, Jeddah, 21589, Saudi Arabia.
Cardiothoracic Surgery Department, Al‑Azhar University, Cairo, Egypt.

Abdullah H Baghaffar (AH)

Division of Cardiac Surgery, Department of Surgery, King Abdulaziz University Hospital, P.O. Box: 80215, Jeddah, 21589, Saudi Arabia.
Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.

Mazin A Fatani (MA)

Division of Cardiac Surgery, Department of Surgery, King Abdulaziz University Hospital, P.O. Box: 80215, Jeddah, 21589, Saudi Arabia.
Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.

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