Prospective Evaluation of FDG-PET/CT for On-treatment Assessment of Response to Neoadjuvant or Induction Chemotherapy in Invasive Bladder Cancer.

Bladder cancer Positron emission tomography (PET) computed tomography (CT) fluorodeoxyglucose F18 imaging lymph-node metastasis urothelial carcinoma

Journal

Bladder cancer (Amsterdam, Netherlands)
ISSN: 2352-3735
Titre abrégé: Bladder Cancer
Pays: Netherlands
ID NLM: 101668567

Informations de publication

Date de publication:
2023
Historique:
received: 31 03 2022
accepted: 01 12 2022
medline: 31 3 2023
pubmed: 31 3 2023
entrez: 12 7 2024
Statut: epublish

Résumé

Neoadjuvant/induction chemotherapy (NAIC) improves survival in patients with muscle-invasive bladder carcinoma (MIBC). On-treatment response assessment may aid in decisions to continue or cease NAIC. We investigated whether We prospectively included 83 patients treated for MIBC (i.e. high-risk cT2-4N0M0 or cT1-4N+M0-1a) between 2014 and 2018. Response to NAIC was assessed after 2-3 cycles with FDG-PET/CT (Peter-Mac and EORTC criteria) and CECT (RECIST1.1 criteria). We assessed prediction of complete pathological response (pCR; ypT0N0), complete pathological down-staging (pCD;≤ypT1N0), any down-staging from baseline (ypTN < cTN) and progression (inoperable tumor/ypN+/M+). The reference standard was histopathological assessment or clinical follow-up. Sensitivity, specificity, and accuracy were calculated. Pathological response rates were 21% for pCR, 29% for pCD, and 10% progressed. All patients underwent FDG-PET/CT and 61 patients also underwent CECT (73%). Accuracy of FDG-PET/CT for prediction of pCR, pCD, and progression were 73%, 48%, and 73%, respectively. Accuracy of CECT for prediction of pCR, pCD, and progression were 78%, 65%, and 67%, respectively. Specificity of CECT was significantly higher than FDG-PET/CT for prediction of pCD and any down-staging ( Routine FDG-PET/CT has insufficient predictive power to aid in response assessment compared to CECT.

Sections du résumé

BACKGROUND BACKGROUND
Neoadjuvant/induction chemotherapy (NAIC) improves survival in patients with muscle-invasive bladder carcinoma (MIBC). On-treatment response assessment may aid in decisions to continue or cease NAIC.
OBJECTIVE OBJECTIVE
We investigated whether
METHODS METHODS
We prospectively included 83 patients treated for MIBC (i.e. high-risk cT2-4N0M0 or cT1-4N+M0-1a) between 2014 and 2018. Response to NAIC was assessed after 2-3 cycles with FDG-PET/CT (Peter-Mac and EORTC criteria) and CECT (RECIST1.1 criteria). We assessed prediction of complete pathological response (pCR; ypT0N0), complete pathological down-staging (pCD;≤ypT1N0), any down-staging from baseline (ypTN < cTN) and progression (inoperable tumor/ypN+/M+). The reference standard was histopathological assessment or clinical follow-up. Sensitivity, specificity, and accuracy were calculated.
RESULTS RESULTS
Pathological response rates were 21% for pCR, 29% for pCD, and 10% progressed. All patients underwent FDG-PET/CT and 61 patients also underwent CECT (73%). Accuracy of FDG-PET/CT for prediction of pCR, pCD, and progression were 73%, 48%, and 73%, respectively. Accuracy of CECT for prediction of pCR, pCD, and progression were 78%, 65%, and 67%, respectively. Specificity of CECT was significantly higher than FDG-PET/CT for prediction of pCD and any down-staging (
CONCLUSIONS CONCLUSIONS
Routine FDG-PET/CT has insufficient predictive power to aid in response assessment compared to CECT.

Identifiants

pubmed: 38994487
doi: 10.3233/BLC-220036
pii: BLC220036
pmc: PMC11181845
doi:

Types de publication

Journal Article

Langues

eng

Pagination

49-57

Informations de copyright

© 2023 – The authors. Published by IOS Press.

Déclaration de conflit d'intérêts

SMHE: none CSV: none EEFvdP: none MLD: none AB: none MSvdH: No conflicts of interest regarding the data presented in this manuscript. Other disclosures are research support from Bristol-Myers Squibb, AstraZeneca, 4SC and Roche and consultancy fees from Bristol-Myers Squibb, Merck, Merck Sharp & Dohme, Roche, AstraZeneca, Seattle Genetics, Pfizer and Janssen (all paid to the Netherlands Cancer Institute). LSM: none KH: none EV: none BWGvR: is an Editorial Board member of this journal, but was not involved in the peer-review process nor had access to any information regarding its peer-review. No conflicts of interest regarding the data presented in this manuscript. Other disclosures are Advisory Board meetings of QED Therapeutics and Ferring.

Auteurs

Sarah M H Einerhand (SMH)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.

Charlotte S Voskuilen (CS)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.

Elies E Fransen van de Putte (EEF)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.

Maarten L Donswijk (ML)

Department of Nuclear Medicine, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands.

Annemarie Bruining (A)

Department of Radiology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands.

Michiel S van der Heijden (MS)

Department of Medical Oncology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands.

Laura S Mertens (LS)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.

Kees Hendricksen (K)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.

Erik Vegt (E)

Department of Nuclear Medicine, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands.

Bas W G van Rhijn (BWG)

Department of Urology, Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital, Amsterdam, TheNetherlands.
Department of Urology, Caritas St. Josef Medical Center, University of Regensburg, Regensburg, Germany.

Classifications MeSH