The integrin receptor beta


Journal

Respiratory research
ISSN: 1465-993X
Titre abrégé: Respir Res
Pays: England
ID NLM: 101090633

Informations de publication

Date de publication:
12 Jul 2024
Historique:
received: 27 12 2023
accepted: 29 06 2024
medline: 13 7 2024
pubmed: 13 7 2024
entrez: 12 7 2024
Statut: epublish

Résumé

Fibroblast differentiation to a myofibroblast phenotype is a feature of airway remodeling in asthma. Lung fibroblasts express the integrin receptor α To investigate the role of the β7 integrin receptor subunit and α C57BL/6 wild type (WT) and β7 integrin null mice (β Eosinophil numbers were similar in WT vs β7-/- mice. Prolonged OVA exposure in β7-/- mice was associated with reduced AHR, lung collagen content, peribronchial smooth muscle, lung tissue TGFβ and IL13 expression as compared to WT. Similar findings were observed in WT mice treated with α The β7 integrin subunit and the α4β7 integrin complex modulate AHR and airway remodeling in a murine model of allergen exposure. This effect is, at least in part, explained by inhibition of fibroblast activation and is independent of eosinophilic inflammation.

Sections du résumé

BACKGROUND BACKGROUND
Fibroblast differentiation to a myofibroblast phenotype is a feature of airway remodeling in asthma. Lung fibroblasts express the integrin receptor α
OBJECTIVES OBJECTIVE
To investigate the role of the β7 integrin receptor subunit and α
METHODS METHODS
C57BL/6 wild type (WT) and β7 integrin null mice (β
RESULTS RESULTS
Eosinophil numbers were similar in WT vs β7-/- mice. Prolonged OVA exposure in β7-/- mice was associated with reduced AHR, lung collagen content, peribronchial smooth muscle, lung tissue TGFβ and IL13 expression as compared to WT. Similar findings were observed in WT mice treated with α
CONCLUSIONS CONCLUSIONS
The β7 integrin subunit and the α4β7 integrin complex modulate AHR and airway remodeling in a murine model of allergen exposure. This effect is, at least in part, explained by inhibition of fibroblast activation and is independent of eosinophilic inflammation.

Identifiants

pubmed: 38997751
doi: 10.1186/s12931-024-02899-8
pii: 10.1186/s12931-024-02899-8
doi:

Substances chimiques

Ovalbumin 9006-59-1
Integrin beta Chains 0
Allergens 0
integrin beta7 0
Transforming Growth Factor beta 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

273

Subventions

Organisme : Ministry of Health, State of Israel
ID : 3-0000-11030

Informations de copyright

© 2024. The Author(s).

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Auteurs

Miri Assayag (M)

Department of Pulmonary Medicine, Hadassah Medical Center and Faculty of Medicine, Hebrew University of Jerusalem, Ein Kerem, Jerusalem, Israel.

Tahrir Obedeyah (T)

Department of Pulmonary Medicine, Hadassah Medical Center and Faculty of Medicine, Hebrew University of Jerusalem, Ein Kerem, Jerusalem, Israel.

Avraham Abutbul (A)

Department of Pulmonary Medicine, Hadassah Medical Center and Faculty of Medicine, Hebrew University of Jerusalem, Ein Kerem, Jerusalem, Israel.

Neville Berkman (N)

Department of Pulmonary Medicine, Hadassah Medical Center and Faculty of Medicine, Hebrew University of Jerusalem, Ein Kerem, Jerusalem, Israel. Neville@hadassah.org.il.

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