Molecular methods enhance the detection of pyoderma-related Streptococcus pyogenes and emm-type distribution in children.

Streptococcus pyogenes emm-typing PCR The Gambia epidemiology molecular diagnostics skin infection strain diversity

Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
15 Jul 2024
Historique:
received: 15 02 2024
revised: 06 07 2024
accepted: 11 07 2024
medline: 15 7 2024
pubmed: 15 7 2024
entrez: 15 7 2024
Statut: aheadofprint

Résumé

Streptococcus pyogenes-related skin infections are increasingly implicated in the development of rheumatic heart disease (RHD) in lower-resourced settings, where they are often associated with scabies. The true prevalence of S. pyogenes-related pyoderma may be underestimated by bacterial culture. A multiplex qPCR for S. pyogenes, Staphylococcus aureus and Sarcoptes scabiei was applied to 250 pyoderma swabs from a cross-sectional study of children <5 years in The Gambia. Direct PCR-based emm-typing was used to supplement previous whole genome sequencing (WGS) of cultured isolates. Pyoderma lesions with S. pyogenes increased from 51% (127/250) using culture to 80% (199/250) with qPCR. Compared to qPCR, the sensitivity of culture was 95.4% for S. pyogenes (95% CI 77.2-99.9) in samples with S. pyogenes alone (22/250, 9%), but 59.9% (95% CI 52.3-67.2) for samples with S. aureus co-infection (177/250, 71%). Direct PCR-based emm-typing was successful in 50% (46/92) of cases, identifying 27 emm-types, including six not identified by WGS (total 52 emm-types). Bacterial culture significantly underestimates the burden of S. pyogenes in pyoderma, particularly when co-infected with S. aureus. Molecular methods should be used to enhance the detection of S. pyogenes in surveillance studies and clinical trials of preventative measures in RHD-endemic settings.

Sections du résumé

BACKGROUND BACKGROUND
Streptococcus pyogenes-related skin infections are increasingly implicated in the development of rheumatic heart disease (RHD) in lower-resourced settings, where they are often associated with scabies. The true prevalence of S. pyogenes-related pyoderma may be underestimated by bacterial culture.
METHODS METHODS
A multiplex qPCR for S. pyogenes, Staphylococcus aureus and Sarcoptes scabiei was applied to 250 pyoderma swabs from a cross-sectional study of children <5 years in The Gambia. Direct PCR-based emm-typing was used to supplement previous whole genome sequencing (WGS) of cultured isolates.
RESULTS RESULTS
Pyoderma lesions with S. pyogenes increased from 51% (127/250) using culture to 80% (199/250) with qPCR. Compared to qPCR, the sensitivity of culture was 95.4% for S. pyogenes (95% CI 77.2-99.9) in samples with S. pyogenes alone (22/250, 9%), but 59.9% (95% CI 52.3-67.2) for samples with S. aureus co-infection (177/250, 71%). Direct PCR-based emm-typing was successful in 50% (46/92) of cases, identifying 27 emm-types, including six not identified by WGS (total 52 emm-types).
CONCLUSIONS CONCLUSIONS
Bacterial culture significantly underestimates the burden of S. pyogenes in pyoderma, particularly when co-infected with S. aureus. Molecular methods should be used to enhance the detection of S. pyogenes in surveillance studies and clinical trials of preventative measures in RHD-endemic settings.

Identifiants

pubmed: 39008379
pii: 7713856
doi: 10.1093/infdis/jiae359
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© The Author(s) 2024. Published by Oxford University Press on behalf of Infectious Diseases Society of America.

Auteurs

Jennifer N Hall (JN)

Division of Clinical Medicine and NIHR Sheffield Biomedical Research Centre, School of Medicine and Population Health, University of Sheffield, Sheffield, United Kingdom.
The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.
School of Biosciences, University of Sheffield, Sheffield, United Kingdom.

Edwin P Armitage (EP)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.
Department of Clinical Research, Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, London, United Kingdom.

Elina Senghore (E)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.

Saffiatou Darboe (S)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.

Momodou Barry (M)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.

Janko Camara (J)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.

Sulayman Bah (S)

Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.

Alexander J Keeley (AJ)

Division of Clinical Medicine and NIHR Sheffield Biomedical Research Centre, School of Medicine and Population Health, University of Sheffield, Sheffield, United Kingdom.
The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.
Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.
Department of Clinical Research, Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, London, United Kingdom.

James S McCarthy (JS)

Peter Doherty Institute of Infection and Immunity, University of Melbourne, Melbourne, Australia.

Pierre Smeesters (P)

Department of Paediatrics, Brussels University Hospital, Academic Children Hospital Queen Fabiola, Université libre de Bruxelles, 1020 Brussels, Belgium.
Molecular Bacteriology Laboratory, Université Libre de Bruxelles, 1070 Brussels, Belgium.

Claire E Turner (CE)

The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.
School of Biosciences, University of Sheffield, Sheffield, United Kingdom.

Thomas C Darton (TC)

Division of Clinical Medicine and NIHR Sheffield Biomedical Research Centre, School of Medicine and Population Health, University of Sheffield, Sheffield, United Kingdom.
The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.

Michael Marks (M)

Department of Clinical Research, Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, London, United Kingdom.
Hospital for Tropical Diseases, University College London Hospital, London, United Kingdom.
Division of Infection & Immunity, University College London, London, United Kingdom.

Adrienn Angyal (A)

Division of Clinical Medicine and NIHR Sheffield Biomedical Research Centre, School of Medicine and Population Health, University of Sheffield, Sheffield, United Kingdom.
The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.

Thushan I de Silva (TI)

Division of Clinical Medicine and NIHR Sheffield Biomedical Research Centre, School of Medicine and Population Health, University of Sheffield, Sheffield, United Kingdom.
The Florey Institute of Infection, University of Sheffield, Sheffield, United Kingdom.
Vaccines and Immunity Theme, Medical Research Council Unit The Gambia at The London School of Hygiene & Tropical Medicine, Banjul, PO Box 273, The Gambia.
Department of Clinical Research, Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, London, United Kingdom.

Classifications MeSH