The structural basis of the activation and inhibition of DSR2 NADase by phage proteins.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
23 Jul 2024
Historique:
received: 29 01 2024
accepted: 09 07 2024
medline: 23 7 2024
pubmed: 23 7 2024
entrez: 22 7 2024
Statut: epublish

Résumé

DSR2, a Sir2 domain-containing protein, protects bacteria from phage infection by hydrolyzing NAD

Identifiants

pubmed: 39039073
doi: 10.1038/s41467-024-50410-0
pii: 10.1038/s41467-024-50410-0
doi:

Substances chimiques

NAD 0U46U6E8UK
NAD+ Nucleosidase EC 3.2.2.5
Viral Tail Proteins 0
Bacterial Proteins 0
Viral Proteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6185

Subventions

Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 32370742
Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 81901168
Organisme : Natural Science Foundation of Hunan Province (Hunan Provincial Natural Science Foundation)
ID : 2022JJ20056
Organisme : Natural Science Foundation of Hunan Province (Hunan Provincial Natural Science Foundation)
ID : 2023JJ40437
Organisme : Natural Science Foundation of Hunan Province (Hunan Provincial Natural Science Foundation)
ID : 2021JJ20075
Organisme : Science and Technology Bureau, Changsha (Changsha Municipal Science and Technology Bureau)
ID : kq2202088

Informations de copyright

© 2024. The Author(s).

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Auteurs

Ruiwen Wang (R)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.

Qi Xu (Q)

Zhejiang Key Laboratory of Structural Biology, School of Life Sciences, Westlake University, Hangzhou, Zhejiang, China.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, Zhejiang, China.

Zhuoxi Wu (Z)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.

Jialu Li (J)

Zhejiang Key Laboratory of Structural Biology, School of Life Sciences, Westlake University, Hangzhou, Zhejiang, China.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, Zhejiang, China.

Hao Guo (H)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.

Tianzhui Liao (T)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.

Yuan Shi (Y)

Zhejiang Key Laboratory of Structural Biology, School of Life Sciences, Westlake University, Hangzhou, Zhejiang, China.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, Zhejiang, China.

Ling Yuan (L)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.

Haishan Gao (H)

Zhejiang Key Laboratory of Structural Biology, School of Life Sciences, Westlake University, Hangzhou, Zhejiang, China.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, Zhejiang, China.

Rong Yang (R)

State Key Laboratory of Developmental Biology of Freshwater Fish, Engineering Research Center of Polyploid Fish Reproduction and Breeding of the State Education Ministry, College of Life Sciences, Hunan Normal University, Changsha, Hunan, China. rongyang@hunnu.edu.cn.

Zhubing Shi (Z)

Zhejiang Key Laboratory of Structural Biology, School of Life Sciences, Westlake University, Hangzhou, Zhejiang, China. shizhubing@westlake.edu.cn.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, Zhejiang, China. shizhubing@westlake.edu.cn.

Faxiang Li (F)

MOE Key Laboratory of Rare Pediatric Diseases, Center for Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China. chinalfx@163.com.

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