Clinical outcomes of multidrug-resistant tracheobronchial tuberculosis receiving anti-tuberculosis regimens containing bedaquiline or delamanid.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
29 Jul 2024
Historique:
received: 31 10 2023
accepted: 24 07 2024
medline: 29 7 2024
pubmed: 29 7 2024
entrez: 28 7 2024
Statut: epublish

Résumé

The treatment of multidrug-resistant tracheobronchial tuberculosis poses challenges, and research investigating the efficacy of bedaquiline or delamanid as treatment for this condition is limited. This retrospective cohort study was conducted from 2017 to 2021. The study extracted data of patients with multidrug-resistant tracheobronchial tuberculosis from medical records and followed up on prognoses. Participants were divided into three groups: the bedaquiline, delamanid, and control group. Clinical outcomes and the risk factors associated with early culture conversion were analyzed. This study included 101 patients, with 32, 25, and 44 patients in the bedaquiline, delamanid, and control groups respectively. The differences in the treatment success rates among the three groups did not show statistical significance. Both the bedaquiline and delamanid groups had significantly higher culture conversion rates compared to the control after 2 or 6 months of treatment, with significantly shorter median times to culture conversion (bedaquiline group: 2 weeks, delamanid group: 2 weeks, control group: 12 weeks, P < 0.001). Treatment with bedaquiline or delamanid were identified as independent predictors of culture conversion at 2 months (bedaquiline group: aOR = 13.417, 95% CI 4.067-44.260, delamanid group: aOR = 9.333, 95% CI 2.498-34.878) or 6 months (bedaquiline group: aOR = 13.333, 95% CI 3.379-52.610, delamanid group: aOR = 5.000, 95% CI 1.357-18.426) of treatment through multivariable logistic regression analyses. The delamanid group showed better improvement in lumen stenosis compared to bedaquiline. Regimens containing bedaquiline or delamanid may accelerate the culture conversion during the early treatment phase in multidrug-resistant tracheobronchial tuberculosis, and delamanid appears to have the potential to effectively improve airway stenosis.

Identifiants

pubmed: 39069547
doi: 10.1038/s41598-024-68550-0
pii: 10.1038/s41598-024-68550-0
doi:

Substances chimiques

bedaquiline 78846I289Y
Nitroimidazoles 0
Antitubercular Agents 0
Oxazoles 0
OPC-67683 0
Diarylquinolines 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

17347

Subventions

Organisme : the Medical Research Project of Chengdu Health Commission
ID : 2023038
Organisme : the Medical Research Project of Chengdu Health Commission
ID : 2022262
Organisme : Sichuan Medical Association (Hengrui) Research Fund Special Research Project
ID : 2021HR75
Organisme : the Technology Innovation Research and Development project of Chengdu Science and Technology Bureau
ID : 2022-YF05-02139-SN
Organisme : the Technology Innovation Research and Development project of Chengdu Science and Technology Bureau
ID : 2022-YF05-02148-SN

Informations de copyright

© 2024. The Author(s).

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Auteurs

Qing Chen (Q)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Tao Huang (T)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Liping Zou (L)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Xianzhen Tang (X)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Zhengyu Shi (Z)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Xinwei Wang (X)

Department of Imaging, Public Health Clinical Center of Chengdu, Chengdu, Sichuan, China.

Huaige Wu (H)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Jiying Sun (J)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Xiaoli Lu (X)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Li Liang (L)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China.

Liangshuang Jiang (L)

Department of Thoracic Surgery, Public Health Clinical Center of Chengdu, Chengdu, Sichuan, China.

Dafeng Liu (D)

Department of Internal Medicine, Public Health Clinical Center of Chengdu, Chengdu, Sichuan, China.

Shenjie Tang (S)

Beijing Tuberculosis and Thoracic Tumor Research Institute, Beijing Chest Hospital, Capital Medical University, Beijing, 101149, China. tangsj1106@vip.sina.com.

Guihui Wu (G)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China. wghwgh2584@sina.com.

Wei He (W)

Department of Tuberculosis, Public Health Clinical Center of Chengdu, Jingjusi 18 Street, Jingjiang District, Chengdu, Sichuan, China. gufuxiao_hw@163.com.

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