Effects of high- versus low-intensity lipid-lowering treatment in patients undergoing serial coronary computed tomography angiography: results of the multi-center LOCATE study.

Agatston score Atherosclerosis Coronary artery disease Low/high intensity lipid-lowering treatment Multi-center Non-calcified plaque Serial plaque quantification

Journal

Clinical research in cardiology : official journal of the German Cardiac Society
ISSN: 1861-0692
Titre abrégé: Clin Res Cardiol
Pays: Germany
ID NLM: 101264123

Informations de publication

Date de publication:
30 Jul 2024
Historique:
received: 06 06 2024
accepted: 18 07 2024
medline: 31 7 2024
pubmed: 31 7 2024
entrez: 30 7 2024
Statut: aheadofprint

Résumé

To evaluate the effects of lipid-lowering medications of different intensities on total, calcified, and non-calcified plaque volumes in patients undergoing serial cardiac computed tomography angiography (CCTA). Individuals with chronic coronary syndromes from 11 centers were included in a retrospective registry. Total, calcified, and non-calcified plaque volumes were quantified and the relative difference in plaque volumes between baseline and follow-up CCTA was calculated. The intensity of lipid-lowering treatment was designated as low, moderate, or high, based on current recommendations. Of 216 patients (mean age 63.1 ± 9.7 years), undergoing serial CCTA (median timespan = 824.5 [IQR = 463.0-1323.0] days), 89 (41.2%) received no or low-intensity lipid-lowering medications, and 80 (37.0%) and 47 (21.8%) moderate- and high-intensity lipid-lowering agents, respectively. Progression of total and non-calcified plaque was attenuated in patients on moderate-/high- versus those on no/low-intensity treatment and arrested in patients treated with high-intensity statins or PCSK9 inhibitors (p < 0.001). Halted increase of non-calcified plaque was associated with LDL-cholesterol reduction (p < 0.001), whereas calcified plaque mass and Agatston score increased irrespective of the lipid-lowering treatment (p = NS). The intensity of lipid-lowering therapy robustly predicted attenuation of non-calcified plaque progression as a function of the time duration between the two CCTA scans, and this was independent of age and cardiovascular risk factors (HR = 3.83, 95% CI = 1.81-8.05, p < 0.001). The LOCATE multi-center observational study shows that progression of non-calcified plaques, which have been previously described as precursors of acute coronary syndromes, can be attenuated with moderate-intensity, and arrested with high-intensity lipid-lowering therapy. DRKS00031954.

Identifiants

pubmed: 39080016
doi: 10.1007/s00392-024-02502-6
pii: 10.1007/s00392-024-02502-6
doi:

Banques de données

DRKS
['DRKS00031954']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Investigateurs

Sorin Giusca (S)
Moritz Schütz (M)
Lukas D Weberling (LD)
Ramona Schmitt (R)
Stefan O Schoenberg (SO)
Mustafa Kuru (M)
Steffen Klömpken (S)
Szilveszter Balint (S)
Pal Maurovitch-Horvat (P)
Johannes Görich (J)
Mustafa Emami (M)
Philipp A Kaufmann (PA)
Patrick Doeblin (P)
Natalia Solowjowa (N)
Karl Jakob Weiss (KJ)
Stefan Baumann (S)
Ksenia Stach (K)

Informations de copyright

© 2024. Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Loris Weichsel (L)

Department of Cardiology & Vascular Medicine & Pneumology, GRN Hospital Weinheim, Roentgenstrasse 1, 69469, Weinheim, Germany.
Cardiac Imaging Center Weinheim, Hector Foundations, Weinheim, Germany.

Florian André (F)

University Hospital Heidelberg, Cardiology, Angiology and Pneumology, Heidelberg, Germany.
DZHK (German Centre for Cardiovascular Research), Partner Site Heidelberg/Mannheim, 69120, Heidelberg, Germany.
MVZ-DRZ Radiology Center, Heidelberg, Germany.

Matthias Renker (M)

Department of Cardiology, Campus Kerckhoff of the Justus Liebig University Giessen, Bad Nauheim, Germany.
German Centre for Cardiovascular Research (DZHK), Partner Site Rhein Main, Bad Nauheim, Germany.

Philipp Breitbart (P)

Department of Cardiology and Angiology, Medical Center, Faculty of Medicine, University of Freiburg, Bad Krozingen, Germany.

Daniel Overhoff (D)

Department of Radiology and Nuclear Medicine, University Medical Center Mannheim (UMM), Heidelberg University, Heidelberg, Germany.

Meinrad Beer (M)

Department for Diagnostic and Interventional Radiology, University Hospital Ulm, Ulm, Germany.

Alexander Giesen (A)

Department of Cardiology & Vascular Medicine & Pneumology, GRN Hospital Weinheim, Roentgenstrasse 1, 69469, Weinheim, Germany.
Cardiac Imaging Center Weinheim, Hector Foundations, Weinheim, Germany.

Borbála Vattay (B)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Sebastian Buss (S)

MVZ-DRZ Radiology Center, Heidelberg, Germany.

Mohamed Marwan (M)

Department of Cardiology, Friedrich-Alexander Universität Erlangen-Nürnberg, Erlangen, Germany.

Christopher L Schlett (CL)

Department of Diagnostic and Interventional Radiology, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Andreas A Giannopoulos (AA)

Department of Nuclear Medicine, Cardiac Imaging, University Hospital Zurich, Zurich, Switzerland.

Sebastian Kelle (S)

German Heart Center Berlin, Berlin, Germany.
DZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Berlin, Germany.

Norbert Frey (N)

University Hospital Heidelberg, Cardiology, Angiology and Pneumology, Heidelberg, Germany.
DZHK (German Centre for Cardiovascular Research), Partner Site Heidelberg/Mannheim, 69120, Heidelberg, Germany.

Grigorios Korosoglou (G)

Department of Cardiology & Vascular Medicine & Pneumology, GRN Hospital Weinheim, Roentgenstrasse 1, 69469, Weinheim, Germany. gkorosoglou@hotmail.com.
Cardiac Imaging Center Weinheim, Hector Foundations, Weinheim, Germany. gkorosoglou@hotmail.com.

Classifications MeSH