Circulating Metabolic Markers Identify Patients at Risk for Tumor Recurrence: A Prospective Cohort Study in Colorectal Cancer Surgery.


Journal

Annals of surgery
ISSN: 1528-1140
Titre abrégé: Ann Surg
Pays: United States
ID NLM: 0372354

Informations de publication

Date de publication:
01 Aug 2024
Historique:
medline: 1 8 2024
pubmed: 1 8 2024
entrez: 1 8 2024
Statut: aheadofprint

Résumé

To investigate the spermidine pathway capability to predict patients at risk for tumor recurrence following colorectal cancer (CRC) surgery. Recurrence rates after CRC surgery remain about 20%, despite an optimal technique and adjuvant therapy when necessary. Identification of risk biomarkers of recurrence is an unmet need. The spermidine pathway is indispensable for cell proliferation and differentiation, and is suggested to accelerate tumor spread. Prospective cohort study of patients undergoing CRC surgery from 2015 to 2018. Plasma samples were collected before surgery and on postoperative day 4, and the spermidine pathway was assessed through mass spectrometry. Oncological outcomes were registered. 146 patients were included and 24 (16.4%) developed tumor recurrence. Higher levels of preoperative spermidine pathway components (spermidine, spermine, spermidine synthase enzyme, and spermine/arginine balance) were positively associated with recurrence. Surgery promoted a decrease in these pathway elements. The greater the decline was, the lower the risk of recurrence. Preoperative spermidine over the cut-off 0.198 µM displayed a 4.69-fold higher risk of recurrence. The spermine synthase enzyme behaved in the opposite direction. The spermidine pathway is associated with tumor recurrence following CRC surgery and, after confirmation in larger cohorts, could be translated as a risk biomarker of recurrence into clinical practice.

Sections du résumé

OBJECTIVE OBJECTIVE
To investigate the spermidine pathway capability to predict patients at risk for tumor recurrence following colorectal cancer (CRC) surgery.
SUMMARY BACKGROUND DATA BACKGROUND
Recurrence rates after CRC surgery remain about 20%, despite an optimal technique and adjuvant therapy when necessary. Identification of risk biomarkers of recurrence is an unmet need. The spermidine pathway is indispensable for cell proliferation and differentiation, and is suggested to accelerate tumor spread.
METHODS METHODS
Prospective cohort study of patients undergoing CRC surgery from 2015 to 2018. Plasma samples were collected before surgery and on postoperative day 4, and the spermidine pathway was assessed through mass spectrometry. Oncological outcomes were registered.
RESULTS RESULTS
146 patients were included and 24 (16.4%) developed tumor recurrence. Higher levels of preoperative spermidine pathway components (spermidine, spermine, spermidine synthase enzyme, and spermine/arginine balance) were positively associated with recurrence. Surgery promoted a decrease in these pathway elements. The greater the decline was, the lower the risk of recurrence. Preoperative spermidine over the cut-off 0.198 µM displayed a 4.69-fold higher risk of recurrence. The spermine synthase enzyme behaved in the opposite direction.
CONCLUSIONS CONCLUSIONS
The spermidine pathway is associated with tumor recurrence following CRC surgery and, after confirmation in larger cohorts, could be translated as a risk biomarker of recurrence into clinical practice.

Identifiants

pubmed: 39087328
doi: 10.1097/SLA.0000000000006463
pii: 00000658-990000000-01005
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Wolters Kluwer Health, Inc. All rights reserved.

Déclaration de conflit d'intérêts

Conflicts of Interest: None declared.

Auteurs

Blanca Montcusí (B)

Section of Colon and Rectal Surgery, Department of Surgery, Hospital del Mar, Barcelona, Spain.
Colorectal Neoplasms Clinical and Translational Research Group, Hospital del Mar Research Institute (IMIM), Barcelona, Spain.
Applied Metabolomics Research Group, Hospital del Mar Medical Institute (IMIM), Barcelona, Spain.
Department of Surgery, University of Barcelona (UB), Barcelona, Spain.

Francisco Madrid-Gambin (F)

Applied Metabolomics Research Group, Hospital del Mar Medical Institute (IMIM), Barcelona, Spain.
Signal and Information Processing for Sensing Systems, Institute for Bioengineering of Catalonia (IBEC), The Barcelona Institute of Science and Technology, Barcelona, Spain.

Silvia Marin (S)

Department of Biochemistry and Molecular Biomedicine, Faculty of Biology, University of Barcelona (UB), Barcelona, Spain.
Institute of Biomedicine, University of Barcelona (UB), Barcelona, Spain.
CIBER of Hepatic and Digestive Diseases (CIBEREHD), Institute of Health Carlos III (ISCIII), Madrid, Spain.

Xavier Mayol (X)

Colorectal Neoplasms Clinical and Translational Research Group, Hospital del Mar Research Institute (IMIM), Barcelona, Spain.

Marta Pascual (M)

Section of Colon and Rectal Surgery, Department of Surgery, Hospital del Mar, Barcelona, Spain.
Colorectal Neoplasms Clinical and Translational Research Group, Hospital del Mar Research Institute (IMIM), Barcelona, Spain.

Marta Cascante (M)

Department of Biochemistry and Molecular Biomedicine, Faculty of Biology, University of Barcelona (UB), Barcelona, Spain.
Institute of Biomedicine, University of Barcelona (UB), Barcelona, Spain.
CIBER of Hepatic and Digestive Diseases (CIBEREHD), Institute of Health Carlos III (ISCIII), Madrid, Spain.

Óscar J Pozo (ÓJ)

Applied Metabolomics Research Group, Hospital del Mar Medical Institute (IMIM), Barcelona, Spain.

Miguel Pera (M)

Department of Surgery, University of Barcelona (UB), Barcelona, Spain.
CIBER of Hepatic and Digestive Diseases (CIBEREHD), Institute of Health Carlos III (ISCIII), Madrid, Spain.
Gastrointestinal and Pancreatic Oncology Group, August Pi i Sunyer Biomedical Research Institute (IDIBAPS), Barcelona, Spain.
Department of General and Digestive Surgery, Institute of Digestive and Metabolic Diseases, Hospital Clínic, Barcelona, Spain.

Classifications MeSH