Treatment of Epidermolysis Bullosa and Future Directions: A Review.

Blistering Gene therapy Repurposed drugs Skin adhesion Wound healing

Journal

Dermatology and therapy
ISSN: 2193-8210
Titre abrégé: Dermatol Ther (Heidelb)
Pays: Switzerland
ID NLM: 101590450

Informations de publication

Date de publication:
02 Aug 2024
Historique:
received: 20 05 2024
accepted: 01 07 2024
medline: 2 8 2024
pubmed: 2 8 2024
entrez: 1 8 2024
Statut: aheadofprint

Résumé

Epidermolysis bullosa (EB) comprises rare genetic disorders characterized by skin and mucosal membrane blistering induced by mechanical trauma. Molecularly, pathogenic variants affect genes encoding proteins crucial for epidermal-dermal adhesion and stability. Management of severe EB is multidisciplinary, focusing on wound healing support, ensuring that patients thrive, and complication treatment. Despite extensive research over 30 years, novel therapeutic approaches face challenges. Gene therapy and protein therapy struggle with efficacy, while regenerative cell-based therapies show limited effects. Drug repurposing to target various pathogenic mechanisms has gained attention, as has in vivo gene therapy with drugs for dystrophic and junctional EB that were recently approved by the US Food and Drug Administration (FDA) and European Medicines Agency (EMA). However, their high cost limits global accessibility. This review examines therapeutic advancements made over the past 5 years, exploiting a systematic literature review and clinical trial data.

Identifiants

pubmed: 39090514
doi: 10.1007/s13555-024-01227-8
pii: 10.1007/s13555-024-01227-8
doi:

Types de publication

Journal Article Review

Langues

eng

Informations de copyright

© 2024. The Author(s).

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Auteurs

Sorina Danescu (S)

Department of Dermatology, University of Medicine Iuliu Hatieganu Cluj-Napoca, Cluj-Napoca, Romania.

Mircea Negrutiu (M)

Department of Dermatology, University of Medicine Iuliu Hatieganu Cluj-Napoca, Cluj-Napoca, Romania.

Cristina Has (C)

Department of Dermatology, Medical Center University of Freiburg, Freiburg im Breisgau, Germany. cristina.has@uniklinik-freiburg.de.

Classifications MeSH