Dissociating the metabolic and tumor-suppressive activity of p53.

cancer inflammation insulin resistance obesity oncogenesis p53

Journal

Trends in endocrinology and metabolism: TEM
ISSN: 1879-3061
Titre abrégé: Trends Endocrinol Metab
Pays: United States
ID NLM: 9001516

Informations de publication

Date de publication:
01 Aug 2024
Historique:
received: 30 06 2024
revised: 24 07 2024
accepted: 24 07 2024
medline: 3 8 2024
pubmed: 3 8 2024
entrez: 2 8 2024
Statut: aheadofprint

Résumé

The tumor suppressor p53 regulates metabolic homeostasis. Recently, Tsaousidou et al. reported that selective activation of p53 via downregulation of Tudor interacting repair regulator (TIRR) confers protection against cancer despite obesity and insulin resistance, providing new insights into the role of p53 at the intersection of oncogenesis and systemic metabolism.

Identifiants

pubmed: 39095229
pii: S1043-2760(24)00199-1
doi: 10.1016/j.tem.2024.07.018
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests The authors have no conflicts of interests to declare.

Auteurs

Yoshitaka Sakurai (Y)

Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.

Naoto Kubota (N)

Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan; Department of Metabolic Medicine, Faculty of Life Science, Kumamoto University, Kumamoto, Japan.

Takashi Kadowaki (T)

Toranomon Hospital, Tokyo, Japan. Electronic address: t-kadowaki@toranomon.kkr.or.jp.

Classifications MeSH