Long-read transcript sequencing identifies differential isoform expression in the entorhinal cortex in a transgenic model of tau pathology.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
02 Aug 2024
Historique:
received: 04 10 2023
accepted: 10 07 2024
medline: 3 8 2024
pubmed: 3 8 2024
entrez: 2 8 2024
Statut: epublish

Résumé

Increasing evidence suggests that alternative splicing plays an important role in Alzheimer's disease (AD) pathology. We used long-read sequencing in combination with a novel bioinformatics tool (FICLE) to profile transcript diversity in the entorhinal cortex of female transgenic (TG) mice harboring a mutant form of human tau. Our analyses revealed hundreds of novel isoforms and identified differentially expressed transcripts - including specific isoforms of Apoe, App, Cd33, Clu, Fyn and Trem2 - associated with the development of tau pathology in TG mice. Subsequent profiling of the human cortex from AD individuals and controls revealed similar patterns of transcript diversity, including the upregulation of the dominant TREM2 isoform in AD paralleling the increased expression of the homologous transcript in TG mice. Our results highlight the importance of differential transcript usage, even in the absence of gene-level expression alterations, as a mechanism underpinning gene regulation in the development of AD neuropathology.

Identifiants

pubmed: 39095344
doi: 10.1038/s41467-024-50486-8
pii: 10.1038/s41467-024-50486-8
doi:

Substances chimiques

tau Proteins 0
Protein Isoforms 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6458

Subventions

Organisme : Alzheimer's Research UK (ARUK)
ID : 573312
Organisme : Alzheimer's Research UK (ARUK)
ID : MR/W004984/1
Organisme : Alzheimer's Society
ID : 231
Pays : United Kingdom
Organisme : Wellcome Trust (Wellcome)
ID : WT097835MF
Organisme : Wellcome Trust (Wellcome)
ID : WT101650MA

Informations de copyright

© 2024. The Author(s).

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Auteurs

Szi Kay Leung (SK)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK. s.k.leung@exeter.ac.uk.

Rosemary A Bamford (RA)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Aaron R Jeffries (AR)

Biosciences, University of Exeter, Exeter, UK.

Isabel Castanho (I)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.
Department of Pathology, Beth Israel Deaconess Medical Center, Boston, MA, USA.
Harvard Medical School, Boston, MA, USA.

Barry Chioza (B)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Christine S Flaxman (CS)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Karen Moore (K)

Biosciences, University of Exeter, Exeter, UK.

Emma L Dempster (EL)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Joshua Harvey (J)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Jonathan T Brown (JT)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Zeshan Ahmed (Z)

Eli Lilly, Cambridge, MA, USA.

Paul O'Neill (P)

Biosciences, University of Exeter, Exeter, UK.

Sarah J Richardson (SJ)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Eilis Hannon (E)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK.

Jonathan Mill (J)

Department of Clinical and Biomedical Sciences, University of Exeter, Exeter, UK. j.mill@exeter.ac.uk.

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