Redox heterogeneity in mouse embryonic stem cells individualizes cell fate decisions.

H3K4me3 epigenome Nrf2 signaling Wnt signaling pathway cell fate decision embryonic stem cells mesendoderm neuroectoderm reactive oxygen species redox stem cell heterogeneity

Journal

Developmental cell
ISSN: 1878-1551
Titre abrégé: Dev Cell
Pays: United States
ID NLM: 101120028

Informations de publication

Date de publication:
30 Jul 2024
Historique:
received: 22 12 2023
revised: 23 04 2024
accepted: 09 07 2024
medline: 7 8 2024
pubmed: 7 8 2024
entrez: 6 8 2024
Statut: aheadofprint

Résumé

Pluripotent embryonic stem cells (ESCs) can develop into any cell type in the body. Yet, the regulatory mechanisms that govern cell fate decisions during embryogenesis remain largely unknown. We now demonstrate that mouse ESCs (mESCs) display large natural variations in mitochondrial reactive oxygen species (mitoROS) levels that individualize their nuclear redox state, H3K4me3 landscape, and cell fate. While mESCs with high mitoROS levels (mitoROS

Identifiants

pubmed: 39106861
pii: S1534-5807(24)00445-3
doi: 10.1016/j.devcel.2024.07.008
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests The authors declare no competing interests.

Auteurs

Agnes Ulfig (A)

Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA. Electronic address: aku@hi.is.

Ursula Jakob (U)

Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA; Biological Chemistry Department, University of Michigan Medical School, Ann Arbor, MI, USA. Electronic address: ujakob@umich.edu.

Classifications MeSH