Enhancing chemotherapeutic efficacy: Niosome-encapsulated Dox-Cis with MUC-1 aptamer.


Journal

Cancer medicine
ISSN: 2045-7634
Titre abrégé: Cancer Med
Pays: United States
ID NLM: 101595310

Informations de publication

Date de publication:
Aug 2024
Historique:
revised: 10 07 2024
received: 10 06 2024
accepted: 24 07 2024
medline: 9 8 2024
pubmed: 9 8 2024
entrez: 9 8 2024
Statut: ppublish

Résumé

Cancer remains a formidable global health challenge, currently affecting nearly 20 million individuals worldwide. Due to the absence of universally effective treatments, ongoing research explores diverse strategies to combat this disease. Recent efforts have concentrated on developing combined drug regimens and targeted therapeutic approaches. This study aimed to investigate the anticancer efficacy of a conjugated drug system, consisting of doxorubicin and cisplatin (Dox-Cis), encapsulated within niosomes and modified with MUC-1 aptamers to enhance biocompatibility and target specific cancer cells. The chemical structure of the Dox-Cis conjugate was characterized using Fourier Transform Infrared Spectroscopy (FTIR) and Liquid Chromatography Quadrupole Time-of-Flight Mass Spectrometry (LC-Q-TOF/MS). The zeta potential and morphological parameters of the niosomal vesicles were determined through Dynamic Light Scattering (DLS) and Transmission Electron Microscopy (TEM). In vitro assessments of cell viability and apoptosis were conducted on MUC-1 positive HeLa cells and MUC-1 negative U87 cells. The findings confirmed the successful conjugation of Dox and Cis within the niosomes. The Nio/Dox-Cis/MUC-1 formulation demonstrated enhanced efficacy compared to the individual drugs and their unencapsulated combination in both cell lines. Notably, the Nio/Dox-Cis/MUC-1 formulation exhibited greater effectiveness on HeLa cells (38.503 ± 1.407) than on U87 cells (46.653 ± 1.297). The study underscores the potential of the Dox-Cis conjugate as a promising strategy for cancer treatment, particularly through platforms that facilitate targeted drug delivery to cancer cells. This targeted approach could lead to more effective and personalized cancer therapies.

Sections du résumé

BACKGROUND BACKGROUND
Cancer remains a formidable global health challenge, currently affecting nearly 20 million individuals worldwide. Due to the absence of universally effective treatments, ongoing research explores diverse strategies to combat this disease. Recent efforts have concentrated on developing combined drug regimens and targeted therapeutic approaches.
OBJECTIVE OBJECTIVE
This study aimed to investigate the anticancer efficacy of a conjugated drug system, consisting of doxorubicin and cisplatin (Dox-Cis), encapsulated within niosomes and modified with MUC-1 aptamers to enhance biocompatibility and target specific cancer cells.
METHODS METHODS
The chemical structure of the Dox-Cis conjugate was characterized using Fourier Transform Infrared Spectroscopy (FTIR) and Liquid Chromatography Quadrupole Time-of-Flight Mass Spectrometry (LC-Q-TOF/MS). The zeta potential and morphological parameters of the niosomal vesicles were determined through Dynamic Light Scattering (DLS) and Transmission Electron Microscopy (TEM). In vitro assessments of cell viability and apoptosis were conducted on MUC-1 positive HeLa cells and MUC-1 negative U87 cells.
RESULTS RESULTS
The findings confirmed the successful conjugation of Dox and Cis within the niosomes. The Nio/Dox-Cis/MUC-1 formulation demonstrated enhanced efficacy compared to the individual drugs and their unencapsulated combination in both cell lines. Notably, the Nio/Dox-Cis/MUC-1 formulation exhibited greater effectiveness on HeLa cells (38.503 ± 1.407) than on U87 cells (46.653 ± 1.297).
CONCLUSION CONCLUSIONS
The study underscores the potential of the Dox-Cis conjugate as a promising strategy for cancer treatment, particularly through platforms that facilitate targeted drug delivery to cancer cells. This targeted approach could lead to more effective and personalized cancer therapies.

Identifiants

pubmed: 39118454
doi: 10.1002/cam4.70079
doi:

Substances chimiques

Doxorubicin 80168379AG
Mucin-1 0
Liposomes 0
Cisplatin Q20Q21Q62J
Aptamers, Nucleotide 0
MUC1 protein, human 0
Antineoplastic Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e70079

Subventions

Organisme : Türkiye Bilimsel ve Teknolojik Araştırma Kurumu

Informations de copyright

© 2024 The Author(s). Cancer Medicine published by John Wiley & Sons Ltd.

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Auteurs

Firat Baris Barlas (FB)

Institute for Technical Chemistry, Leibniz University Hannover, Hannover, Germany.
Institue of Nanotechnology and Biotechnology, İstanbul University-Cerrahpaşa, İstanbul, Turkey.

Bilge Olceroglu (B)

Institue of Nanotechnology and Biotechnology, İstanbul University-Cerrahpaşa, İstanbul, Turkey.

Didem Ag Seleci (D)

Institute for Particle Technology (iPAT), Technische Universität Braunschweig, Braunschweig, Germany.

Zinar Pinar Gumus (ZP)

Central Research Test and Analysis Laboratory Application and Research Center, Ege University, Izmir, Turkey.

Pinar Siyah (P)

Department of Biochemistry, School of Pharmacy, Bahçeşehir University, Istanbul, Turkey.

Meriam Dabbek (M)

Institute for Technical Chemistry, Leibniz University Hannover, Hannover, Germany.

Georg Garnweitne (G)

Institute for Particle Technology (iPAT), Technische Universität Braunschweig, Braunschweig, Germany.

Frank Stahl (F)

Institute for Technical Chemistry, Leibniz University Hannover, Hannover, Germany.

Thomas Scheper (T)

Institute for Technical Chemistry, Leibniz University Hannover, Hannover, Germany.

Suna Timur (S)

Central Research Test and Analysis Laboratory Application and Research Center, Ege University, Izmir, Turkey.
Department of Biochemistry, Faculty of Science, Ege University, Izmir, Turkey.

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