The genetics and epidemiology of N- and O-immunoglobulin A glycomics.


Journal

Genome medicine
ISSN: 1756-994X
Titre abrégé: Genome Med
Pays: England
ID NLM: 101475844

Informations de publication

Date de publication:
09 Aug 2024
Historique:
received: 13 01 2024
accepted: 26 07 2024
medline: 10 8 2024
pubmed: 10 8 2024
entrez: 9 8 2024
Statut: epublish

Résumé

Immunoglobulin (Ig) glycosylation modulates the immune response and plays a critical role in ageing and diseases. Studies have mainly focused on IgG glycosylation, and little is known about the genetics and epidemiology of IgA glycosylation. We generated, using a novel liquid chromatography-mass spectrometry method, the first large-scale IgA glycomics dataset in serum from 2423 twins, encompassing 71 N- and O-glycan species. We showed that, despite the lack of a direct genetic template, glycosylation is highly heritable, and that glycopeptide structures are sex-specific, and undergo substantial changes with ageing. We observe extensive correlations between the IgA and IgG glycomes, and, exploiting the twin design, show that they are predominantly influenced by shared genetic factors. A genome-wide association study identified eight loci associated with both the IgA and IgG glycomes (ST6GAL1, ELL2, B4GALT1, ABCF2, TMEM121, SLC38A10, SMARCB1, and MGAT3) and two novel loci specifically modulating IgA O-glycosylation (C1GALT1 and ST3GAL1). Validation of our findings in an independent cohort of 320 individuals from Qatar showed that the underlying genetic architecture is conserved across ancestries. Our study delineates the genetic landscape of IgA glycosylation and provides novel potential functional links with the aetiology of complex immune diseases, including genetic factors involved in IgA nephropathy risk.

Sections du résumé

BACKGROUND BACKGROUND
Immunoglobulin (Ig) glycosylation modulates the immune response and plays a critical role in ageing and diseases. Studies have mainly focused on IgG glycosylation, and little is known about the genetics and epidemiology of IgA glycosylation.
METHODS METHODS
We generated, using a novel liquid chromatography-mass spectrometry method, the first large-scale IgA glycomics dataset in serum from 2423 twins, encompassing 71 N- and O-glycan species.
RESULTS RESULTS
We showed that, despite the lack of a direct genetic template, glycosylation is highly heritable, and that glycopeptide structures are sex-specific, and undergo substantial changes with ageing. We observe extensive correlations between the IgA and IgG glycomes, and, exploiting the twin design, show that they are predominantly influenced by shared genetic factors. A genome-wide association study identified eight loci associated with both the IgA and IgG glycomes (ST6GAL1, ELL2, B4GALT1, ABCF2, TMEM121, SLC38A10, SMARCB1, and MGAT3) and two novel loci specifically modulating IgA O-glycosylation (C1GALT1 and ST3GAL1). Validation of our findings in an independent cohort of 320 individuals from Qatar showed that the underlying genetic architecture is conserved across ancestries.
CONCLUSIONS CONCLUSIONS
Our study delineates the genetic landscape of IgA glycosylation and provides novel potential functional links with the aetiology of complex immune diseases, including genetic factors involved in IgA nephropathy risk.

Identifiants

pubmed: 39123268
doi: 10.1186/s13073-024-01369-6
pii: 10.1186/s13073-024-01369-6
doi:

Substances chimiques

Immunoglobulin A 0
Polysaccharides 0
Immunoglobulin G 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

96

Subventions

Organisme : Medical Research Council
ID : MR/K01353X/1-2
Pays : United Kingdom

Informations de copyright

© 2024. The Author(s).

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Auteurs

Alessia Visconti (A)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.
Center for Biostatistics, Epidemiology and Public Health, Department of Clinical and Biological Sciences, University of Turin, Turin, Italy.

Niccolò Rossi (N)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Albert Bondt (A)

Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.

Agnes Hipgrave Ederveen (AH)

Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.

Gaurav Thareja (G)

Department of Biophysics and Physiology, Weill Cornell Medicine-Qatar, Doha, Qatar.

Carolien A M Koeleman (CAM)

Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.

Nisha Stephan (N)

Department of Biophysics and Physiology, Weill Cornell Medicine-Qatar, Doha, Qatar.

Anna Halama (A)

Department of Biophysics and Physiology, Weill Cornell Medicine-Qatar, Doha, Qatar.

Hannah J Lomax-Browne (HJ)

Centre for Inflammatory Disease, Department of Immunology and Inflammation, Imperial College London, London, UK.

Matthew C Pickering (MC)

Centre for Inflammatory Disease, Department of Immunology and Inflammation, Imperial College London, London, UK.

Xu-Jie Zhou (XJ)

Renal Division, Peking University First Hospital, Beijing, China.
Peking University Institute of Nephrology, Beijing, China.
Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, China.
Key Laboratory of Chronic Kidney Disease Prevention and Treatment, Ministry of Education, Peking University, Beijing, China.

Manfred Wuhrer (M)

Center for Proteomics and Metabolomics, Leiden University Medical Center, Leiden, The Netherlands.

Karsten Suhre (K)

Department of Biophysics and Physiology, Weill Cornell Medicine-Qatar, Doha, Qatar.

Mario Falchi (M)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK. mario.falchi@kcl.ac.uk.

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