Effects of Modulating BMP9, BMPR2, and AQP1 on BMP Signaling in Human Pulmonary Microvascular Endothelial Cells.
Humans
Aquaporin 1
/ metabolism
Growth Differentiation Factor 2
/ metabolism
Signal Transduction
Endothelial Cells
/ metabolism
Bone Morphogenetic Protein Receptors, Type II
/ metabolism
Transforming Growth Factor beta1
/ metabolism
Lung
/ metabolism
Microvessels
/ metabolism
Cells, Cultured
Gene Silencing
Pulmonary Arterial Hypertension
/ metabolism
Bone Morphogenetic Proteins
AQP1
BMP9
BMPR2
PAH
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
24 Jul 2024
24 Jul 2024
Historique:
received:
27
06
2024
revised:
19
07
2024
accepted:
22
07
2024
medline:
10
8
2024
pubmed:
10
8
2024
entrez:
10
8
2024
Statut:
epublish
Résumé
Pulmonary arterial hypertension (PAH) is a chronic disease characterized by a progressive increase in mean pulmonary arterial pressure. Mutations in the
Identifiants
pubmed: 39125626
pii: ijms25158043
doi: 10.3390/ijms25158043
pii:
doi:
Substances chimiques
Aquaporin 1
146410-94-8
Growth Differentiation Factor 2
0
Bone Morphogenetic Protein Receptors, Type II
EC 2.7.11.30
Transforming Growth Factor beta1
0
GDF2 protein, human
0
BMPR2 protein, human
EC 2.7.11.30
AQP1 protein, human
0
BMP10 protein, human
0
TGFB1 protein, human
0
Bone Morphogenetic Proteins
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM